Postmortem brain: an underutilized substrate for studying severe mental illness
about
Investigating the potential influence of cause of death and cocaine levels on the differential expression of genes associated with cocaine abuseAltered serine/threonine kinase activity in schizophreniaA molecular profile of cocaine abuse includes the differential expression of genes that regulate transcription, chromatin, and dopamine cell phenotype.The novel α7β2-nicotinic acetylcholine receptor subtype is expressed in mouse and human basal forebrain: biochemical and pharmacological characterization.GluN2B protein deficits in the left, but not the right, hippocampus in schizophrenia.Alterations of mGluR5 and its endogenous regulators Norbin, Tamalin and Preso1 in schizophrenia: towards a model of mGluR5 dysregulation.Decreased chloride channel expression in the dorsolateral prefrontal cortex in schizophrenia.Post-mortem analysis of neuroinflammatory changes in human Alzheimer's disease.Converging models of schizophrenia--Network alterations of prefrontal cortex underlying cognitive impairments.Expression quantitative trait loci (eQTLs) in microRNA genes are enriched for schizophrenia and bipolar disorder association signals.The NSW brain tissue resource centre: Banking for alcohol and major neuropsychiatric disorders research.Protein Kinase Activity Decreases with Higher Braak Stages of Alzheimer's Disease PathologyMolecular evidence of synaptic pathology in the CA1 region in schizophrenia.Neuroproteomic profiling of human body fluids.Proteome effects of antipsychotic drugs: Learning from preclinical models.Employing proteomics to unravel the molecular effects of antipsychotics and their role in schizophrenia.Extracellular matrix proteomics in schizophrenia and Alzheimer's disease.The role of glutamate transporters in the pathophysiology of neuropsychiatric disorders.Microglia and Brain Plasticity in Acute Psychosis and Schizophrenia Illness Course: A Meta-Review.Slc7a11 (xCT) protein expression is not altered in the depressed brain and system xc- deficiency does not affect depression-associated behaviour in the corticosterone mouse model.Altered fucosyltransferase expression in the superior temporal gyrus of elderly patients with schizophrenia.Defects in Bioenergetic Coupling in Schizophrenia.Synaptic loss in schizophrenia: a meta-analysis and systematic review of synaptic protein and mRNA measures.Neuron-specific deficits of bioenergetic processes in the dorsolateral prefrontal cortex in schizophrenia.Essential Role of Keap1-Nrf2 Signaling in Mood Disorders: Overview and Future Perspective
P2860
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P2860
Postmortem brain: an underutilized substrate for studying severe mental illness
description
2013 nî lūn-bûn
@nan
2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Postmortem brain: an underutilized substrate for studying severe mental illness
@ast
Postmortem brain: an underutilized substrate for studying severe mental illness
@en
type
label
Postmortem brain: an underutilized substrate for studying severe mental illness
@ast
Postmortem brain: an underutilized substrate for studying severe mental illness
@en
prefLabel
Postmortem brain: an underutilized substrate for studying severe mental illness
@ast
Postmortem brain: an underutilized substrate for studying severe mental illness
@en
P2093
P2860
P356
P1476
Postmortem brain: an underutilized substrate for studying severe mental illness
@en
P2093
James H Meador-Woodruff
John H Hammond
Robert E McCullumsmith
P2860
P2888
P356
10.1038/NPP.2013.239
P407
P577
2013-10-04T00:00:00Z
P6179
1021375395