Effects of continuously enhanced corticotropin releasing factor expression within the bed nucleus of the stria terminalis on conditioned and unconditioned anxiety
about
Angiotensin type 1 receptor inhibition enhances the extinction of fear memory.Neuropeptide regulation of signaling and behavior in the BNST.The Deakin/Graeff hypothesis: focus on serotonergic inhibition of panic.Optogenetic strategies to investigate neural circuitry engaged by stress.Striatal-enriched protein tyrosine phosphatase-STEPs toward understanding chronic stress-induced activation of corticotrophin releasing factor neurons in the rat bed nucleus of the stria terminalis.Activation of corticotropin releasing factor-containing neurons in the rat central amygdala and bed nucleus of the stria terminalis following exposure to two different anxiogenic stressors.Serotonergic systems in the balance: CRHR1 and CRHR2 differentially control stress-induced serotonin synthesis.GABA and NMDA receptors in CRF neurons have opposing effects in fear acquisition and anxiety in central amygdala vs. bed nucleus of the stria terminalis.The CRH1 antagonist GSK561679 increases human fear but not anxiety as assessed by startle.Importance of CRF receptor-mediated mechanisms of the bed nucleus of the stria terminalis in the processing of anxiety and pain.The role of PKC signaling in CRF-induced modulation of startle.Increased anxiety in corticotropin-releasing factor type 2 receptor-null mice requires recent acute stress exposure and is associated with dysregulated serotonergic activity in limbic brain areas.Sex differences in anxiety and emotional behavior.Anxiogenic effects of CGRP within the BNST may be mediated by CRF acting at BNST CRFR1 receptors.Corticotropin-releasing factor-related peptides, serotonergic systems, and emotional behaviorStress-related serotonergic systems: implications for symptomatology of anxiety and affective disorders.Behavioral Studies and Genetic Alterations in Corticotropin-Releasing Hormone (CRH) Neurocircuitry: Insights into Human Psychiatric Disorders.Evidence for coordinated functional activity within the extended amygdala of non-human and human primates.Addiction is a Reward Deficit and Stress Surfeit Disorder.Escitalopram alters gene expression and HPA axis reactivity in rats following chronic overexpression of corticotropin-releasing factor from the central amygdala.Addiction as a stress surfeit disorder.Differential contribution of CBP:CREB binding to corticotropin-releasing hormone expression in the infant and adult hypothalamusOrganization of multisynaptic circuits within and between the medial and the central extended amygdala.Functional topography of serotonergic systems supports the Deakin/Graeff hypothesis of anxiety and affective disorders.Mechanisms in the bed nucleus of the stria terminalis involved in control of autonomic and neuroendocrine functions: a review.The CRF family of neuropeptides and their receptors - mediators of the central stress response.Knockdown of BNST GluN2B-containing NMDA receptors mimics the actions of ketamine on novelty-induced hypophagia.Targeting Corticotropin-Releasing Factor Projections from the Oval Nucleus of the Bed Nucleus of the Stria Terminalis Using Cell-Type Specific Neuronal Tracing Studies in Mouse and Rat Brain.Overexpressing Corticotropin-Releasing Factor in the Primate Amygdala Increases Anxious Temperament and Alters Its Neural CircuitAllelic variation in CRHR1 predisposes to panic disorder: evidence for biased fear processing.Corticotropin releasing factor type-1 receptor antagonism in the dorsolateral bed nucleus of the stria terminalis disrupts contextually conditioned fear, but not unconditioned fear to a predator odor.Oxytocin receptor neurotransmission in the dorsolateral bed nucleus of the stria terminalis facilitates the acquisition of cued fear in the fear-potentiated startle paradigm in rats.Early life adversity and serotonin transporter gene variation interact to affect DNA methylation of the corticotropin-releasing factor gene promoter region in the adult rat brain.Two models of inescapable stress increase tph2 mRNA expression in the anxiety-related dorsomedial part of the dorsal raphe nucleus.The Neuropeptide Tac2 Controls a Distributed Brain State Induced by Chronic Social Isolation Stress
P2860
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P2860
Effects of continuously enhanced corticotropin releasing factor expression within the bed nucleus of the stria terminalis on conditioned and unconditioned anxiety
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Effects of continuously enhanc ...... oned and unconditioned anxiety
@ast
Effects of continuously enhanc ...... oned and unconditioned anxiety
@en
type
label
Effects of continuously enhanc ...... oned and unconditioned anxiety
@ast
Effects of continuously enhanc ...... oned and unconditioned anxiety
@en
prefLabel
Effects of continuously enhanc ...... oned and unconditioned anxiety
@ast
Effects of continuously enhanc ...... oned and unconditioned anxiety
@en
P2093
P2860
P356
P1433
P1476
Effects of continuously enhanc ...... oned and unconditioned anxiety
@en
P2093
D L Walker
E I Flandreau
S M Freeman
P2860
P2888
P304
P356
10.1038/MP.2011.188
P407
P577
2012-01-31T00:00:00Z
P5875
P6179
1001637053