Mitochondrial toxins in models of neurodegenerative diseases. I: In vivo brain hydroxyl radical production during systemic MPTP treatment or following microdialysis infusion of methylpyridinium or azide ions.

Mitochondrial toxins in models of neurodegenerative diseases. I: In vivo brain hydroxyl radical production during systemic MPTP treatment or following microdialysis infusion of methylpyridinium or azide ions.

Item
http://www.wikidata.org/entity/Q30471061
Cellular and biochemical actions of melatonin which protect against free radicals: role in neurodegenerative disordersNeuronal death is an active, caspase-dependent process after moderate but not severe DNA damage.Protective effect of α-lipoic acid against antimycin A cytotoxicity in MC3T3-E1 osteoblastic cellsPyrroloquinoline quinone stimulates mitochondrial biogenesis through cAMP response element-binding protein phosphorylation and increased PGC-1alpha expressionEndogenous 3,4-dihydroxyphenylalanine and dopaquinone modifications on protein tyrosine: links to mitochondrially derived oxidative stress via hydroxyl radicalMutations in cytochrome c oxidase subunit VIa cause neurodegeneration and motor dysfunction in DrosophilaMechanism of oxidative stress in neurodegeneration.Imaging of superoxide generation in the dopaminergic area of the brain in Parkinson's disease, using mito-TEMPO.Calcium and cell death signaling in neurodegeneration and aging.Glucose Levels in Culture Medium Determine Cell Death Mode in MPP(+)-treated Dopaminergic Neuronal Cells.Protective effect of harmalol and harmaline on MPTP neurotoxicity in the mouse and dopamine-induced damage of brain mitochondria and PC12 cells.Paeoniflorin isolated from Paeonia lactiflora attenuates osteoblast cytotoxicity induced by antimycin A.Overexpression of CYP2D6 attenuates the toxicity of MPP+ in actively dividing and differentiated PC12 cells.Mitochondria As Sources and Targets of Methane.Honokiol protects osteoblastic MC3T3-E1 cells against antimycin A-induced cytotoxicity.
P2860

Mitochondrial toxins in models of neurodegenerative diseases. I: In vivo brain hydroxyl radical production during systemic MPTP treatment or following microdialysis infusion of methylpyridinium or azide ions.

Item
http://www.wikidata.org/entity/Q30471061
description
1997 nî lūn-bûn
@nan
1997 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@ast
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@en
type
label
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@ast
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@en
prefLabel
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@ast
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@en
P1433
P1476
P2093
P304
P31
P356
P407
P433
P478
P577
P698
P921
P356
P1433
P1476
Mitochondrial toxins in models ...... ethylpyridinium or azide ions.
@en
P2093
P304
P31
P356
10.1016/S0006-8993(97)00429-0
P407
P433
P478
P577
P698
P921