Onset coding is degraded in auditory nerve fibers from mutant mice lacking synaptic ribbons. - Test2 - elhamkhani - TriplyDB

Onset coding is degraded in auditory nerve fibers from mutant mice lacking synaptic ribbons.

Onset coding is degraded in auditory nerve fibers from mutant mice lacking synaptic ribbons.

http://www.wikidata.org/entity/Q30478316

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Cochlear neuropathy and the coding of supra-threshold sound.Selective inner hair cell loss in prematurity: a temporal bone study of infants from a neonatal intensive care unit Neural Hyperactivity of the Central Auditory System in Response to Peripheral Damage.The cochlea as an independent neuroendocrine organ: expression and possible roles of a local hypothalamic-pituitary-adrenal axis-equivalent signaling system Dual-color STED microscopy reveals a sandwich structure of Bassoon and Piccolo in active zones of adult and aged mice.New insights into cochlear sound encoding Cochlear Synaptopathy and Noise-Induced Hidden Hearing Loss.Neural spike-timing patterns vary with sound shape and periodicity in three auditory cortical fields.Transient auditory nerve demyelination as a new mechanism for hidden hearing loss.Synaptic studies inform the functional diversity of cochlear afferents Toward a Differential Diagnosis of Hidden Hearing Loss in Humans.Persistent Thalamic Sound Processing Despite Profound Cochlear Denervation Functional modeling of the human auditory brainstem response to broadband stimulation Coding Deficits in Noise-Induced Hidden Hearing Loss May Stem from Incomplete Repair of Ribbon Synapses in the Cochlea.ERK2 mediates inner hair cell survival and decreases susceptibility to noise-induced hearing loss Phase-locking precision is enhanced by multiquantal release at an auditory hair cell ribbon synapse.Specific synaptopathies diversify brain responses and hearing disorders: you lose the gain from early life.Relating structure and function of inner hair cell ribbon synapses.The auditory nerve overlapped waveform (ANOW) originates in the cochlear apex Morphological and physiological development of auditory synapses.Ouabain-induced cochlear nerve degeneration: synaptic loss and plasticity in a mouse model of auditory neuropathy.Quantitative analysis of ribbons, vesicles, and cisterns at the cat inner hair cell synapse: correlations with spontaneous rate.Developmental acquisition of a rapid calcium-regulated vesicle supply allows sustained high rates of exocytosis in auditory hair cells.Noise-induced cochlear neuropathy is selective for fibers with low spontaneous rates.Insult-induced adaptive plasticity of the auditory system A Gata3-Mafb transcriptional network directs post-synaptic differentiation in synapses specialized for hearing Making choice between competing rewards in uncertain vs. safe social environment: role of neuronal nicotinic receptors of acetylcholine.L-type CaV1.2 deletion in the cochlea but not in the brainstem reduces noise vulnerability: implication for CaV1.2-mediated control of cochlear BDNF expression.Restoration of hearing in the VGLUT3 knockout mouse using virally mediated gene therapy.Transmitter release from cochlear hair cells is phase locked to cyclic stimuli of different intensities and frequencies.The reduced cochlear output and the failure to adapt the central auditory response causes tinnitus in noise exposed rats.Exocytosis in the frog amphibian papilla.The cochlear CRF signaling systems and their mechanisms of action in modulating cochlear sensitivity and protection against trauma.Silent damage of noise on cochlear afferent innervation in guinea pigs and the impact on temporal processing.Opposing gradients of ribbon size and AMPA receptor expression underlie sensitivity differences among cochlear-nerve/hair-cell synapses The diverse roles of ribbon synapses in sensory neurotransmission.Bassoon and the synaptic ribbon organize Ca²+ channels and vesicles to add release sites and promote refilling EF-hand protein Ca2+ buffers regulate Ca2+ influx and exocytosis in sensory hair cells Specialized postsynaptic morphology enhances neurotransmitter dilution and high-frequency signaling at an auditory synapse Conical tomography of a ribbon synapse: structural evidence for vesicle fusion.

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P2860

Onset coding is degraded in auditory nerve fibers from mutant mice lacking synaptic ribbons.

http://www.wikidata.org/entity/Q30478316

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Onset coding is degraded in au ...... mice lacking synaptic ribbons.

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Onset coding is degraded in au ...... mice lacking synaptic ribbons.

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type

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Onset coding is degraded in au ...... mice lacking synaptic ribbons.

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Onset coding is degraded in au ...... mice lacking synaptic ribbons.

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prefLabel

Onset coding is degraded in au ...... mice lacking synaptic ribbons.

@ast

Onset coding is degraded in au ...... mice lacking synaptic ribbons.

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JNEUROSCI.0389-10.2010

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Journal of Neuroscience

P1476

Onset coding is degraded in au ...... mice lacking synaptic ribbons.

@en

P2093

Andreas Neef

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Bradley N Buran

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Eckart D Gundelfinger

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M Charles Liberman

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Nicola Strenzke

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P2860

Phase locking of auditory-nerve fibers to the envelopes of high-frequency sounds: implications for sound localization

Molecular dissection of the photoreceptor ribbon synapse: physical interaction of Bassoon and RIBEYE is essential for the assembly of the ribbon complex

The modulation transfer function for speech intelligibility

The Ca2+ channel subunit beta2 regulates Ca2+ channel abundance and function in inner hair cells and is required for hearing

The precedence effect

Prestin is required for electromotility of the outer hair cell and for the cochlear amplifier

Phase-locked response to low-frequency tones in single auditory nerve fibers of the squirrel monkey

Refractoriness and neural precision

Response properties of single auditory nerve fibers in the mouse

Piccolo, a presynaptic zinc finger protein structurally related to bassoon

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10.1523/JNEUROSCI.0389-10.2010

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