Neurological and behavioral abnormalities, ventricular dilatation, altered cellular functions, inflammation, and neuronal injury in brains of mice due to common, persistent, parasitic infection. - Test2 - elhamkhani - TriplyDB

Neurological and behavioral abnormalities, ventricular dilatation, altered cellular functions, inflammation, and neuronal injury in brains of mice due to common, persistent, parasitic infection.

Neurological and behavioral abnormalities, ventricular dilatation, altered cellular functions, inflammation, and neuronal injury in brains of mice due to common, persistent, parasitic infection.

http://www.wikidata.org/entity/Q30484779

about

Mice infected with low-virulence strains of Toxoplasma gondii lose their innate aversion to cat urine, even after extensive parasite clearance Toxoplasma gondii infection and behaviour - location, location, location?Effects of Extracts from Thai Piperaceae Plants against Infection with Toxoplasma gondii Toxoplasma gondii-induced neuronal alterations Long-Term Relationships: the Complicated Interplay between the Host and the Developmental Stages of Toxoplasma gondii during Acute and Chronic Infections Autoimmune diseases, gastrointestinal disorders and the microbiome in schizophrenia: more than a gut feeling Characteristics and critical function of CD8+ T cells in the Toxoplasma-infected brain.Chronic Toxoplasma gondii in Nurr1-null heterozygous mice exacerbates elevated open field activity Inflammation and the two-hit hypothesis of schizophrenia.Neurophysiological Changes Induced by Chronic Toxoplasma gondii Infection Acquired infection with Toxoplasma gondii in adult mice results in sensorimotor deficits but normal cognitive behavior despite widespread brain pathology.Activated microglia contribute to neuronal apoptosis in Toxoplasmic encephalitis.The distribution of Toxoplasma gondii cysts in the brain of a mouse with latent toxoplasmosis: implications for the behavioral manipulation hypothesis.Are There any Relationships between Latent Toxoplasma gondii Infection, Testosterone Elevation, and Risk of Autism Spectrum Disorder?Toxoplasma gondii actively inhibits neuronal function in chronically infected mice.Seroprevalence of Toxoplasma gondii infection and associated risk factors in Huicholes in Mexico T-cell production of matrix metalloproteinases and inhibition of parasite clearance by TIMP-1 during chronic Toxoplasma infection in the brain.Toxoplasmosis and epilepsy--systematic review and meta analysis.Chronic infection of Toxoplasma gondii downregulates miR-132 expression in multiple brain regions in a sex-dependent manner Incidence of adult brain cancers is higher in countries where the protozoan parasite Toxoplasma gondii is common.Global Metabolomic Profiling of Mice Brains following Experimental Infection with the Cyst-Forming Toxoplasma gondii.Experimental Toxoplasmosis in Rats Induced Orally with Eleven Strains of Toxoplasma gondii of Seven Genotypes: Tissue Tropism, Tissue Cyst Size, Neural Lesions, Tissue Cyst Rupture without Reactivation, and Ocular Lesions.Behavioral changes in mice caused by Toxoplasma gondii invasion of brain GLT-1-Dependent Disruption of CNS Glutamate Homeostasis and Neuronal Function by the Protozoan Parasite Toxoplasma gondii.Huntingtons Disease Mice Infected with Toxoplasma gondii Demonstrate Early Kynurenine Pathway Activation, Altered CD8+ T-Cell Responses, and Premature Mortality.The microRNA miR-181c controls microglia-mediated neuronal apoptosis by suppressing tumor necrosis factor.Latent toxoplasmosis and human Shared Immune and Repair Markers During Experimental Toxoplasma Chronic Brain Infection and Schizophrenia Role of infectious agents in the carcinogenesis of brain and head and neck cancers.Chronic Toxoplasma gondii infection enhances β-amyloid phagocytosis and clearance by recruited monocytes.Behavioral Abnormalities in a Mouse Model of Chronic Toxoplasmosis Are Associated with MAG1 Antibody Levels and Cyst Burden.New paradigms for understanding and step changes in treating active and chronic, persistent apicomplexan infections.Transcriptome analysis of mouse brain infected with Toxoplasma gondii.Transplantation of Fas-deficient or wild-type neural stem/progenitor cells (NPCs) is equally efficient in treating experimental autoimmune encephalomyelitis (EAE).Evaluation of kynurenine pathway metabolism in Toxoplasma gondii-infected mice: implications for schizophrenia.Chronic murine toxoplasmosis is defined by subtle changes in neuronal connectivity.Is Toxoplasma gondii a Trigger of Bipolar Disorder?Inflammation in neurodegenerative diseases.Neuropsychiatric manifestations of latent toxoplasmosis on mothers and their offspring.Dissecting Amyloid Beta Deposition Using Distinct Strains of the Neurotropic Parasite Toxoplasma gondii as a Novel Tool.

P2860

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P2860

Neurological and behavioral abnormalities, ventricular dilatation, altered cellular functions, inflammation, and neuronal injury in brains of mice due to common, persistent, parasitic infection.

http://www.wikidata.org/entity/Q30484779

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2008 nî lūn-bûn

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2008 թվականի հոտեմբերին հրատարակված գիտական հոդված

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Catherine Reardon

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Craig W Roberts

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David J P Ferguson

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P2860

The transcriptome of the intraerythrocytic developmental cycle of Plasmodium falciparum

An animal model of age-related macular degeneration in senescent Ccl-2- or Ccr-2-deficient mice

Molecular cloning and chromosomal mapping of a bone marrow stromal cell surface gene, BST2, that may be involved in pre-B-cell growth

Trex1 prevents cell-intrinsic initiation of autoimmunity

Antibodies to Toxoplasma gondii in patients with schizophrenia: a meta-analysis

The effect of Toxoplasma gondii on animal behavior: playing cat and mouse

Early infections of Toxoplasma gondii and the later development of schizophrenia

Fatal attraction in rats infected with Toxoplasma gondii

Cyclophilin C-associated protein: a normal secreted glycoprotein that down-modulates endotoxin and proinflammatory responses in vivo

Histone hyperacetylation induces demethylation of reelin and 67-kDa glutamic acid decarboxylase promoters

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