Corticotropin-releasing factor 1 antagonists selectively reduce ethanol self-administration in ethanol-dependent rats - Test2 - elhamkhani - TriplyDB

Corticotropin-releasing factor 1 antagonists selectively reduce ethanol self-administration in ethanol-dependent rats

Corticotropin-releasing factor 1 antagonists selectively reduce ethanol self-administration in ethanol-dependent rats

http://www.wikidata.org/entity/Q30486793

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Neuropharmacology of alcohol addiction Acute withdrawal, protracted abstinence and negative affect in alcoholism: are they linked?Stress, dysregulation of drug reward pathways, and the transition to drug dependence Chronic alcohol neuroadaptation and stress contribute to susceptibility for alcohol craving and relapse Corticotropin-releasing factor-1 receptor involvement in behavioral neuroadaptation to ethanol: a urocortin1-independent mechanism Amygdala protein kinase C epsilon controls alcohol consumption A role for brain stress systems in addiction.Alcohol response and consumption in adolescent rhesus macaques: life history and genetic influences Effects of alcohol dependence and withdrawal on stress responsiveness and alcohol consumption Animal models of alcohol and drug dependence Neuropeptide modulation of central amygdala neuroplasticity is a key mediator of alcohol dependence.Urocortins: CRF's siblings and their potential role in anxiety, depression and alcohol drinking behavior.Behavioral, biological, and chemical perspectives on targeting CRF(1) receptor antagonists to treat alcoholism.Alcohol and nicotine interactions: pre-clinical models of dependence An Update on CRF Mechanisms Underlying Alcohol Use Disorders and Dependence CRF1-R activation of the dynorphin/kappa opioid system in the mouse basolateral amygdala mediates anxiety-like behavior Preclinical studies of alcohol binge drinking The central amygdala as an integrative hub for anxiety and alcohol use disorders Synaptic effects induced by alcohol.Predator odor stress alters corticotropin-releasing factor-1 receptor (CRF1R)-dependent behaviors in rats.Nicotine dependence produces hyperalgesia: role of corticotropin-releasing factor-1 receptors (CRF1Rs) in the central amygdala (CeA).Corticotropin-releasing factor (CRF) and α 2 adrenergic receptors mediate heroin withdrawal-potentiated startle in rats Noradrenergic transmission in the extended amygdala: role in increased drug-seeking and relapse during protracted drug abstinence Corticotropin-releasing factor acting on corticotropin-releasing factor receptor type 1 is critical for binge alcohol drinking in mice.Development of ethanol withdrawal-related sensitization and relapse drinking in mice selected for high- or low-ethanol preference.Role of corticotropin-releasing factor in drug addiction: potential for pharmacological intervention.Single-nucleotide polymorphisms in corticotropin releasing hormone receptor 1 gene (CRHR1) are associated with quantitative trait of event-related potential and alcohol dependence Progress in corticotropin-releasing factor-1 antagonist development.The role of CRF and CRF-related peptides in the dark side of addiction.CRF(1) receptor antagonists attenuate escalated cocaine self-administration in rats Disruption of the CRF(2) receptor pathway decreases the somatic expression of opiate withdrawal.A CRF(2) agonist administered into the central nucleus of the amygdala decreases ethanol self-administration in ethanol-dependent rats.Dependence-induced increases in ethanol self-administration in mice are blocked by the CRF1 receptor antagonist antalarmin and by CRF1 receptor knockout.The behavioral, anatomical and pharmacological parallels between social attachment, love and addiction.Hedonic Homeostatic Dysregulation as a Driver of Drug-Seeking Behavior Operant ethanol self-administration in ethanol dependent mice.Neurobiological mechanisms of addiction: focus on corticotropin-releasing factor.Neurobiological mechanisms contributing to alcohol-stress-anxiety interactions.Role of innate and drug-induced dysregulation of brain stress and arousal systems in addiction: Focus on corticotropin-releasing factor, nociceptin/orphanin FQ, and orexin/hypocretin.Corticotropin-releasing factor-1 receptor activation mediates nicotine withdrawal-induced deficit in brain reward function and stress-induced relapse

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Corticotropin-releasing factor 1 antagonists selectively reduce ethanol self-administration in ethanol-dependent rats

http://www.wikidata.org/entity/Q30486793

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2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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Corticotropin-releasing factor ...... tion in ethanol-dependent rats

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Corticotropin-releasing factor ...... tion in ethanol-dependent rats

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J.BIOPSYCH.2006.03.063

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Biological Psychiatry

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Corticotropin-releasing factor ...... tion in ethanol-dependent rats

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Cindy K Funk

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Eric P Zorrilla

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George F Koob

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Kenner C Rice

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Mei-Jing Lee

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P2860

Expression cloning of a human corticotropin-releasing-factor receptor.

Human urocortin II: mild locomotor suppressive and delayed anxiolytic-like effects of a novel corticotropin-releasing factor related peptide

Cloning and functional expression of a rat brain corticotropin releasing factor (CRF) receptor

4-(1,3-Dimethoxyprop-2-ylamino)-2,7-dimethyl-8-(2, 4-dichlorophenyl)pyrazolo[1,5-a]-1,3,5-triazine: a potent, orally bioavailable CRF(1) receptor antagonist

Corticotropin releasing factor receptor 1-deficient mice display decreased anxiety, impaired stress response, and aberrant neuroendocrine development

Corticotropin-releasing factor CRF1, but not CRF2, receptors mediate anxiogenic-like behavior

Corticotropin-releasing factor and Urocortin I modulate excitatory glutamatergic synaptic transmission

Characterization of a 41-residue ovine hypothalamic peptide that stimulates secretion of corticotropin and beta-endorphin

New perspectives in basal forebrain organization of special relevance for neuropsychiatric disorders: the striatopallidal, amygdaloid, and corticopetal components of substantia innominata.

The CRF1 receptor antagonist antalarmin reduces volitional ethanol consumption in isolation-reared fawn-hooded rats.

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78-86

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10.1016/J.BIOPSYCH.2006.03.063

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1

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6910684

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16876134

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2741496

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