DNA repair modulates the vulnerability of the developing brain to alkylating agents - Test2 - elhamkhani - TriplyDB

DNA repair modulates the vulnerability of the developing brain to alkylating agents

DNA repair modulates the vulnerability of the developing brain to alkylating agents

http://www.wikidata.org/entity/Q30488029

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The Response to Oxidative DNA Damage in Neurons: Mechanisms and Disease Is neurodegenerative disease a long-latency response to early-life genotoxin exposure?Aag DNA glycosylase promotes alkylation-induced tissue damage mediated by Parp1 The cycad genotoxin MAM modulates brain cellular pathways involved in neurodegenerative disease and cancer in a DNA damage-linked manner Unraveling 50-Year-Old Clues Linking Neurodegeneration and Cancer to Cycad Toxins: Are microRNAs Common Mediators?Animal models of brain maldevelopment induced by cycad plant genotoxins Altered methylation of the DNA repair gene MGMT is associated with neural tube defects.Human Ntera2 cells as a predictive in vitro test system for developmental neurotoxicity.O6-methylguanine induces altered proteins at the level of transcription in human cells DNA repair mechanisms in dividing and non-dividing cells.Establishment and characterization of Prnp knockdown neuroblastoma cells using dual microRNA-mediated RNA interference Cigarette smoke induces DNA damage and alters base-excision repair and tau levels in the brain of neonatal mice.Base excision repair in physiology and pathology of the central nervous system Balancing repair and tolerance of DNA damage caused by alkylating agents Evidence for base excision repair processing of DNA interstrand crosslinks.Transcriptional profiling of the mouse hippocampus supports an NMDAR-mediated neurotoxic mode of action for benzo[a]pyrene.Toxicity biomarkers among US children compared to a similar cohort in France: a blinded study measuring urinary porphyrins.Multifaceted roles of alkyltransferase and related proteins in DNA repair, DNA damage, resistance to chemotherapy, and research tools.Small-molecule inhibitors of DNA damage-repair pathways: an approach to overcome tumor resistance to alkylating anticancer drugs.Comparative human and rat neurospheres reveal species differences in chemical effects on neurodevelopmental key events.Kinetic mechanism for the flipping and excision of 1,N(6)-ethenoadenine by human alkyladenine DNA glycosylase.

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

http://www.wikidata.org/entity/Q30488029

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2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009 թվականի հունվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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J.DNAREP.2008.12.002

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DNA repair modulates the vulnerability of the developing brain to alkylating agents

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A Olivas

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D Doerge

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G E Kisby

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P2860

N-methylpurine DNA glycosylase overexpression increases alkylation sensitivity by rapidly removing non-toxic 7-methylguanine adducts

Biomarkers in maternal and newborn blood indicate heightened fetal susceptibility to procarcinogenic DNA damage

Neurocognitive effects of treatment for childhood cancer

Genotoxicants target distinct molecular networks in neonatal neurons

Relationship between polycyclic aromatic hydrocarbon-DNA adducts, environmental tobacco smoke, and child development in the World Trade Center cohort

PAH-DNA adducts in cord blood and fetal and child development in a Chinese cohort

Effect of prenatal exposure to airborne polycyclic aromatic hydrocarbons on neurodevelopment in the first 3 years of life among inner-city children

Involvement of nucleotide excision repair in a recombination-independent and error-prone pathway of DNA interstrand cross-link repair.

Role of nucleotide- and base-excision repair in genotoxin-induced neuronal cell death.

Heterozygosity for the mouse Apex gene results in phenotypes associated with oxidative stress.

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