Dystrophin deficiency in Drosophila reduces lifespan and causes a dilated cardiomyopathy phenotype. - Test2 - elhamkhani - TriplyDB

Dystrophin deficiency in Drosophila reduces lifespan and causes a dilated cardiomyopathy phenotype.

Dystrophin deficiency in Drosophila reduces lifespan and causes a dilated cardiomyopathy phenotype.

http://www.wikidata.org/entity/Q30493760

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Sestrins at the crossroad between stress and aging Methods to assess Drosophila heart development, function and aging Model organisms in the fight against muscular dystrophy: lessons from drosophila and Zebrafish Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heart Complex genetic architecture of cardiac disease in a wild type inbred strain of Drosophila melanogaster A Drosophila model of high sugar diet-induced cardiomyopathy Exercise-training in young Drosophila melanogaster reduces age-related decline in mobility and cardiac performance Multiple measures of functionality exhibit progressive decline in a parallel, stochastic fashion in Drosophila Sod2 null mutants A mighty small heart: the cardiac proteome of adult Drosophila melanogaster The conserved WW-domain binding sites in Dystroglycan C-terminus are essential but partially redundant for Dystroglycan function d4eBP acts downstream of both dTOR and dFoxo to modulate cardiac functional aging in Drosophila.High-fat-diet-induced obesity and heart dysfunction are regulated by the TOR pathway in Drosophila SMAD signaling drives heart and muscle dysfunction in a Drosophila model of muscular dystrophy Chronic hypoxia impairs muscle function in the Drosophila model of Duchenne's muscular dystrophy (DMD).Genetic modifier screens reveal new components that interact with the Drosophila dystroglycan-dystrophin complex.Serial examination of an inducible and reversible dilated cardiomyopathy in individual adult Drosophila.The evolutionarily conserved RNA binding protein SMOOTH is essential for maintaining normal muscle function Affecting Rhomboid-3 function causes a dilated heart in adult Drosophila.A dual role for integrin-linked kinase and β1-integrin in modulating cardiac aging.Non-autonomous modulation of heart rhythm, contractility and morphology in adult fruit flies.Age-dependent electrical and morphological remodeling of the Drosophila heart caused by hERG/seizure mutations.Characterization, design, and function of the mitochondrial proteome: from organs to organisms Aging and CaMKII alter intracellular Ca2+ transients and heart rhythm in Drosophila melanogaster.New dystrophin/dystroglycan interactors control neuron behavior in Drosophila eye.The role of pygopus in the differentiation of intracardiac valves in Drosophila.Flightless flies: Drosophila models of neuromuscular disease.The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster Sizing up models of heart failure: Proteomics from flies to humans.Drosophila as a model to study cardiac aging Transcription factor neuromancer/TBX20 is required for cardiac function in Drosophila with implications for human heart disease.A method to measure myocardial calcium handling in adult Drosophila Mechanical and non-mechanical functions of Dystrophin can prevent cardiac abnormalities in Drosophila.Cardiomyopathy is associated with ribosomal protein gene haplo-insufficiency in Drosophila melanogaster Cardiac aging: from molecular mechanisms to significance in human health and disease Probing the polygenic basis of cardiomyopathies in Drosophila.Genetic elevation of sphingosine 1-phosphate suppresses dystrophic muscle phenotypes in Drosophila.Pentamidine rescues contractility and rhythmicity in a Drosophila model of myotonic dystrophy heart dysfunction.Deletion of Siah-interacting protein gene in Drosophila causes cardiomyopathy.Partial loss of GATA factor Pannier impairs adult heart function in Drosophila.Pygopus maintains heart function in aging Drosophila independently of canonical Wnt signaling

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P2860

Dystrophin deficiency in Drosophila reduces lifespan and causes a dilated cardiomyopathy phenotype.

http://www.wikidata.org/entity/Q30493760

description

2008 nî lūn-bûn

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2008 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2008 թվականի հունվարին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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type

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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Dystrophin deficiency in Droso ...... ated cardiomyopathy phenotype.

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P2093

David Yaffe

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Grant Hogg

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Jeffrey S Chamberlain

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Karen Ocorr

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Ouarda Taghli-Lamallem

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Rolf Bodmer

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Takeshi Akasaka

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Uri Nudel

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P2860

KCNQ potassium channel mutations cause cardiac arrhythmias in Drosophila that mimic the effects of aging.

The genome sequence of Drosophila melanogaster

Targeted gene expression as a means of altering cell fates and generating dominant phenotypes

The complete sequence of dystrophin predicts a rod-shaped cytoskeletal protein

The gene tinman is required for specification of the heart and visceral muscles in Drosophila

Drosophila, an emerging model for cardiac disease

Dissecting muscle and neuronal disorders in a Drosophila model of muscular dystrophy.

Drosophila as a model for the identification of genes causing adult human heart disease.

Myosin transducer mutations differentially affect motor function, myofibril structure, and the performance of skeletal and cardiac muscles

Modeling human neurodegenerative diseases in Drosophila: on a wing and a prayer.

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