Differences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures. - Test2 - elhamkhani - TriplyDB

Differences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures.

Differences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures.

http://www.wikidata.org/entity/Q30496411

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Novel determinants of the neuronal Cl(-) concentration Anesthesia with sevoflurane in neonatal rats: Developmental neuroendocrine abnormalities and alleviating effects of the corticosteroid and Cl(-) importer antagonists.Molecular mechanisms of antiseizure drug activity at GABAA receptors Roles of aldosterone and oxytocin in abnormalities caused by sevoflurane anesthesia in neonatal rats Transgenic mouse lines for non-invasive ratiometric monitoring of intracellular chloride.The Subiculum: A Potential Site of Ictogenesis in a Neonatal Seizure Model.Early postnatal EEG features of perinatal arterial ischaemic stroke with seizures Twenty years of fluorescence imaging of intracellular chloride Questioning the depolarizing effects of GABA during early brain development.Optogenetic activation of cajal-retzius cells reveals their glutamatergic output and a novel feedforward circuit in the developing mouse hippocampus.Structural foundations of optogenetics: Determinants of channelrhodopsin ion selectivity.Bumetanide enhances phenobarbital efficacy in a rat model of hypoxic neonatal seizures.Progressive NKCC1-dependent neuronal chloride accumulation during neonatal seizures.Cell-attached recordings of responses evoked by photorelease of GABA in the immature cortical neurons.Pharmacotherapeutic targeting of cation-chloride cotransporters in neonatal seizures Propofol-induced electroencephalographic seizures in neonatal rats: the role of corticosteroids and γ-aminobutyric acid type A receptor-mediated excitation PACAP modulation of calcium ion activity in developing granule cells of the neonatal mouse olfactory bulb.Efficacy of synaptic inhibition depends on multiple, dynamically interacting mechanisms implicated in chloride homeostasis.Traumatic alterations in GABA signaling disrupt hippocampal network activity in the developing brain.Developmental Decrease of Neuronal Chloride Concentration Is Independent of Trauma in Thalamocortical Brain Slices.Rapamycin down-regulates KCC2 expression and increases seizure susceptibility to convulsants in immature rats.Bumetanide, an Inhibitor of NKCC1 (Na-K-2Cl Cotransporter Isoform 1), Enhances Propofol-Induced Loss of Righting Reflex but Not Its Immobilizing Actions in Neonatal Rats.Flupirtine effectively prevents development of acute neonatal seizures in an animal model of global hypoxia.Role of Steroids in Hyperexcitatory Adverse and Anesthetic Effects of Sevoflurane in Neonatal Rats.Recording EEG in immature rats with a novel miniature telemetry system.Development and physiology of GABAergic feedback excitation in parvalbumin expressing interneurons of the mouse basolateral amygdala.Optogenetic Visualization of Presynaptic Tonic Inhibition of Cerebellar Parallel Fibers A GABAergic projection from the zona incerta to cortex promotes cortical neuron development.GABAergic depolarization during early cortical development and implications for anticonvulsive therapy in neonates.Excitatory action of GABA on immature neurons is not due to absence of ketone bodies metabolites or other energy substrates.Optogenetic reporters: Fluorescent protein-based genetically encoded indicators of signaling and metabolism in the brain Cortical inhibition, pH and cell excitability in epilepsy: what are optimal targets for antiepileptic interventions?Computational models of neuron-astrocyte interaction in epilepsy.Electrographic status epilepticus in children with critical illness: Epidemiology and outcome Pharmacotherapy for Seizures in Neonates with Hypoxic Ischemic Encephalopathy.Pharmacotherapy for Neonatal Seizures: Current Knowledge and Future Perspectives.Levetiracetam for the Treatment of Seizures in Neonatal Hypoxic Ischemic Encephalopathy.Chloride Dysregulation, Seizures, and Cerebral Edema: A Relationship with Therapeutic Potential.Heightened stress response and cognitive impairment after repeated neonatal sevoflurane exposures might be linked to excessive GABAAR-mediated depolarization.Response to comments on "Local impermeant anions establish the neuronal chloride concentration".

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Differences in cortical versus subcortical GABAergic signaling: a candidate mechanism of electroclinical uncoupling of neonatal seizures.

http://www.wikidata.org/entity/Q30496411

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2009 nî lūn-bûn

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2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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Differences in cortical versus ...... coupling of neonatal seizures.

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Differences in cortical versus ...... coupling of neonatal seizures.

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Differences in cortical versus ...... coupling of neonatal seizures.

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J.NEURON.2009.08.022

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Differences in cortical versus ...... coupling of neonatal seizures.

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Brian J Bacskai

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George Augustine

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Guoping Feng

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Joseph Glykys

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Kevin J Staley

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Kishore V Kuchibhotla

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Thomas Kuner

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Volodymyr I Dzhala

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P2860

The SLC4 family of HCO 3 - transporters

GABA: a pioneer transmitter that excites immature neurons and generates primitive oscillations

Absence seizures: a review of recent reports with new concepts

NKCC1 transporter facilitates seizures in the developing brain

The K+/Cl- co-transporter KCC2 renders GABA hyperpolarizing during neuronal maturation

Differential regulation of gamma-aminobutyric acid receptor channels by diazepam and phenobarbital

Disruption of KCC2 reveals an essential role of K-Cl cotransport already in early synaptic inhibition

Cl- uptake promoting depolarizing GABA actions in immature rat neocortical neurones is mediated by NKCC1

Expression and localization of Na-driven Cl-HCO(3)(-) exchanger (SLC4A8) in rodent CNS

GABA regulates synaptic integration of newly generated neurons in the adult brain.

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10.1016/J.NEURON.2009.08.022

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19755108

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