Elevated BDNF after cocaine withdrawal facilitates LTP in medial prefrontal cortex by suppressing GABA inhibition - Test2 - elhamkhani - TriplyDB

Elevated BDNF after cocaine withdrawal facilitates LTP in medial prefrontal cortex by suppressing GABA inhibition

Elevated BDNF after cocaine withdrawal facilitates LTP in medial prefrontal cortex by suppressing GABA inhibition

http://www.wikidata.org/entity/Q30497605

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Exercise as a novel treatment for drug addiction: a neurobiological and stage-dependent hypothesis.Diffusion dynamics of synaptic molecules during inhibitory postsynaptic plasticity Fyn-dependent gene networks in acute ethanol sensitivity Chronic cocaine disrupts mesocortical learning mechanisms Addiction is Not a Natural Kind The Buzz about anabolic androgenic steroids: electrophysiological effects in excitable tissues.Reduced levels of brain-derived neurotrophic factor contribute to synaptic imbalance during the critical period of respiratory development in rats.Relapse to cocaine-seeking after abstinence is regulated by cAMP-dependent protein kinase A in the prefrontal cortex.Drug-evoked synaptic plasticity in addiction: from molecular changes to circuit remodeling GABAAα1-mediated plasticity in the orbitofrontal cortex regulates context-dependent action selection.BDNF interacts with endocannabinoids to regulate cocaine-induced synaptic plasticity in mouse midbrain dopamine neurons.Brain-derived neurotrophic factor and addiction: Pathological versus therapeutic effects on drug seeking.Current perspectives on the neurobiology of drug addiction: a focus on genetics and factors regulating gene expression.Upregulation of nerve growth factor in central amygdala increases sensitivity to opioid reward Regulation of BDNF expression by cocaine.Formation of aversive memories associated with conditioned drug withdrawal requires BDNF expression in the amygdala in acute morphine-dependent rats.The Medial Orbitofrontal Cortex Regulates Sensitivity to Outcome Value.Repeated cocaine exposure increases fast-spiking interneuron excitability in the rat medial prefrontal cortex.Prefrontal cortical BDNF: A regulatory key in cocaine- and food-reinforced behaviors.Opiate versus psychostimulant addiction: the differences do matter Developmental disruption of gamma-aminobutyric acid function in the medial prefrontal cortex by noncontingent cocaine exposure during early adolescence.Signals from the Fourth Dimension Regulate Drug Relapse Compartmentalization of GABAergic inhibition by dendritic spines.Systemic Delivery of a Brain-Penetrant TrkB Antagonist Reduces Cocaine Self-Administration and Normalizes TrkB Signaling in the Nucleus Accumbens and Prefrontal Cortex.Adaptations in AMPA receptor transmission in the nucleus accumbens contributing to incubation of cocaine craving.Opiate exposure and withdrawal dynamically regulate mRNA expression in the serotonergic dorsal raphe nucleus.Changes in microRNA expression profile in hippocampus during the acquisition and extinction of cocaine-induced conditioned place preference in rats Juvenile methylphenidate reduces prefrontal cortex plasticity via D3 receptor and BDNF in adulthood.BDNF-endocannabinoid interactions at neocortical inhibitory synapses require phospholipase C signaling Cocaine Promotes Coincidence Detection and Lowers Induction Threshold during Hebbian Associative Synaptic Potentiation in Prefrontal Cortex.Early-life cocaine interferes with BDNF-mediated behavioral plasticity.Long-term plasticity at inhibitory synapses Designing modulators of 5-hydroxytryptamine signaling to treat abuse disorders.Multiple faces of BDNF in cocaine addiction.Regulation of GABAARs by phosphorylation Glutamatergic synaptic plasticity in the mesocorticolimbic system in addiction H3 and H4 Lysine Acetylation Correlates with Developmental and Experimentally Induced Adult Experience-Dependent Plasticity in the Mouse Visual Cortex.Role for Endogenous BDNF in Endocannabinoid-Mediated Long-Term Depression at Neocortical Inhibitory Synapses(,.)Brain-derived neurotrophic factor rapidly increases AMPA receptor surface expression in rat nucleus accumbens.Brain-derived neurotrophic factor interacts with adult-born immature cells in the dentate gyrus during consolidation of overlapping memories.

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Elevated BDNF after cocaine withdrawal facilitates LTP in medial prefrontal cortex by suppressing GABA inhibition

http://www.wikidata.org/entity/Q30497605

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@ast

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@en

type

label

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@ast

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@en

prefLabel

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@ast

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@en

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J.NEURON.2010.08.012

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P1476

Elevated BDNF after cocaine wi ...... by suppressing GABA inhibition

@en

P2093

Byung Kook Lim

http://www.w3.org/2001/XMLSchema#string

Hui Lu

http://www.w3.org/2001/XMLSchema#string

Mu-Ming Poo

http://www.w3.org/2001/XMLSchema#string

Nina Khoshnevisrad

http://www.w3.org/2001/XMLSchema#string

Pei-Lin Cheng

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P2860

NEURAL MECHANISMS OF ADDICTION: The Role of Reward-Related Learning and Memory

Ventral tegmental area BDNF induces an opiate-dependent-like reward state in naive rats

Millisecond-timescale, genetically targeted optical control of neural activity

Modulation of GABA(A) receptor phosphorylation and membrane trafficking by phospholipase C-related inactive protein/protein phosphatase 1 and 2A signaling complex underlying brain-derived neurotrophic factor-dependent regulation of GABAergic inhibit

Limbic activation during cue-induced cocaine craving

Molecular basis of long-term plasticity underlying addiction

Neural systems for behavioral activation and reward.

Regulation of a protein phosphatase cascade allows convergent dopamine and glutamate signals to activate ERK in the striatum

Single cocaine exposure in vivo induces long-term potentiation in dopamine neurons.

Incentive-sensitization and addiction.

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821-833

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scholarly article

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10.1016/J.NEURON.2010.08.012

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46180810

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20826313

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2998346

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