Role of mTOR in podocyte function and diabetic nephropathy in humans and mice. - Test2 - elhamkhani - TriplyDB

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

http://www.wikidata.org/entity/Q30500731

about

Role of mTOR Inhibitors in Kidney Disease Podocytes Nonimmunologic targets of immunosuppressive agents in podocytes Endoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the Ugly Role of nutrient-sensing signals in the pathogenesis of diabetic nephropathy The role of autophagy in the pathogenesis of diabetic nephropathy Cell biology and pathology of podocytes Molecular mechanisms of diabetic kidney disease Podocyte-Specific Deletion of Murine CXADR Does Not Impair Podocyte Development, Function or Stress Response Protective effects of the mTOR inhibitor everolimus on cytoskeletal injury in human podocytes are mediated by RhoA signaling Autophagy in kidney health and disease.Judicious Toggling of mTOR Activity to Combat Insulin Resistance and Cancer: Current Evidence and Perspectives Mesenchymal Stem Cells as Therapeutic Candidates for Halting the Progression of Diabetic Nephropathy 2017 update on the relationship between diabetes and colorectal cancer: epidemiology, potential molecular mechanisms and therapeutic implications Increased 4E-BP1 Expression Protects against Diet-Induced Obesity and Insulin Resistance in Male Mice.Epidermal growth factor receptor inhibition slows progression of diabetic nephropathy in association with a decrease in endoplasmic reticulum stress and an increase in autophagy Soluble Urokinase Receptor and the Kidney Response in Diabetes Mellitus.Podocytes, signaling pathways, and vascular factors in diabetic kidney disease Regulation of blood-testis barrier (BTB) dynamics during spermatogenesis via the "Yin" and "Yang" effects of mammalian target of rapamycin complex 1 (mTORC1) and mTORC2.mTORC1 maintains renal tubular homeostasis and is essential in response to ischemic stress.Tribbles homolog 3 attenuates mammalian target of rapamycin complex-2 signaling and inflammation in the diabetic kidney Expression of a novel stress-inducible protein, sestrin 2, in rat glomerular parietal epithelial cells.Genipin inhibits mitochondrial uncoupling protein 2 expression and ameliorates podocyte injury in diabetic mice.In vivo evidence for mTORC2-mediated actin cytoskeleton rearrangement in neurons Autophagy in diabetic nephropathy Gene-gene and gene-environment interactions in apolipoprotein L1 gene-associated nephropathy.New targets for treatment of diabetic nephropathy: what we have learned from animal models.Mammalian target of rapamycin complex 2 signaling pathway regulates transient receptor potential cation channel 6 in podocytes.Dysregulated autophagy contributes to podocyte damage in Fabry's disease.Mtor-Fanconi Anemia DNA Damage Repair Pathway in Cancer.GIV/girdin links vascular endothelial growth factor signaling to Akt survival signaling in podocytes independent of nephrin.Inhibition of insulin/IGF-1 receptor signaling protects from mitochondria-mediated kidney failure.Defective podocyte insulin signalling through p85-XBP1 promotes ATF6-dependent maladaptive ER-stress response in diabetic nephropathy.Insulin signaling: implications for podocyte biology in diabetic kidney disease.TGFβ-induced deptor suppression recruits mTORC1 and not mTORC2 to enhance collagen I (α2) gene expression.Nutrient sensing, autophagy, and diabetic nephropathy.Signaling mechanisms in the regulation of renal matrix metabolism in diabetes.Recessive nephrocerebellar syndrome on the Galloway-Mowat syndrome spectrum is caused by homozygous protein-truncating mutations of WDR73.Shear stress induces cell apoptosis via a c-Src-phospholipase D-mTOR signaling pathway in cultured podocytes.Progress in pathogenesis of proteinuria.

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P2860

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

http://www.wikidata.org/entity/Q30500731

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

@en

prefLabel

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

@ast

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

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Journal of Clinical Investigation

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Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

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Alessia Fornoni

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Andrea Debreczeni-Mór

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Björn Hartleben

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Clemens D Cohen

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Gerd Walz

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Götz Hartleben

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Ken Inoki

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Maja T Lindenmeyer

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Maria-Pia Rastaldi

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Markus A Rüegg

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P2860

The mTOR/PI3K and MAPK pathways converge on eIF4B to control its phosphorylation and activity

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Disruption of the mouse mTOR gene leads to early postimplantation lethality and prohibits embryonic stem cell development

mTOR is essential for growth and proliferation in early mouse embryos and embryonic stem cells

VEGF inhibition and renal thrombotic microangiopathy

TOR signaling in growth and metabolism

mTOR and S6K1 mediate assembly of the translation preinitiation complex through dynamic protein interchange and ordered phosphorylation events

Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB

The TSC1-2 tumor suppressor controls insulin-PI3K signaling via regulation of IRS proteins

Tuberous sclerosis complex proteins control axon formation

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2197-2209

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10.1172/JCI44774

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6

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121

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Dontscho Kerjaschki

Matthias Kretzler

Hermann Pavenstädt

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2011-05-23T00:00:00Z

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51160049

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21606591

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3104746

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