about
Atypical IκB proteins - nuclear modulators of NF-κB signalingPeptide-induced negative selection of thymocytes activates transcription of an NF-kappa B inhibitorThe role of c-FLIP in modulation of CD95-induced apoptosisSLy2 targets the nuclear SAP30/HDAC1 complexConstitutive expression of murine c-FLIPR causes autoimmunity in aged miceRegulation of death receptor-mediated apoptosis pathways.IκB(NS) protein mediates regulatory T cell development via induction of the Foxp3 transcription factor.Cleavage of roquin and regnase-1 by the paracaspase MALT1 releases their cooperatively repressed targets to promote T(H)17 differentiation.The role of c-FLIP splice variants in urothelial tumours.Thalidomide induces limb anomalies by PTEN stabilization, Akt suppression, and stimulation of caspase-dependent cell deathPhosphorylation of Atg5 by the Gadd45β-MEKK4-p38 pathway inhibits autophagy.Differential sialylation of cell surface glycoconjugates in a human B lymphoma cell line regulates susceptibility for CD95 (APO-1/Fas)-mediated apoptosis and for infection by a lymphotropic virus.Snail regulates cell survival and inhibits cellular senescence in human metastatic prostate cancer cell lines.Fas/CD95-mediated apoptosis of type II cells is blocked by Toxoplasma gondii primarily via interference with the mitochondrial amplification loop.A single nucleotide polymorphism determines protein isoform production of the human c-FLIP protein.c-REL and IκBNS Govern Common and Independent Steps of Regulatory T Cell Development from Novel CD122-Expressing Pre-Precursors.Toxoplasma gondii inhibits Fas/CD95-triggered cell death by inducing aberrant processing and degradation of caspase 8.Loss of caspase-9 reveals its essential role for caspase-2 activation and mitochondrial membrane depolarization.Loss of caspase-9 provides genetic evidence for the type I/II concept of CD95-mediated apoptosis.CD95 ligand mediates T-cell receptor-induced apoptosis of a CD4+ CD8+ double positive thymic lymphoma.The role of CAP3 in CD95 signaling: new insights into the mechanism of procaspase-8 activation.Specificity of anti-human CD95 (APO-1/Fas) antibodies.Glutathione dependence of caspase-8 activation at the death-inducing signaling complex.Differential modulation of apoptosis sensitivity in CD95 type I and type II cells.Inhibition of autophagy through MAPK14-mediated phosphorylation of ATG5.Death receptor-induced signaling pathways are differentially regulated by gamma interferon upstream of caspase 8 processing.Mutational analyses of c-FLIPR, the only murine short FLIP isoform, reveal requirements for DISC recruitment.Different forms of cell death induced by putative BCL2 inhibitors.Alternative splicing of caspase-8 mRNA during differentiation of human leukocytes.Caspase inhibitor zVAD.fmk reduces infarct size after myocardial ischaemia and reperfusion in rats but not in mice.A mathematical model of the impact of insulin secretion dynamics on selective hepatic insulin resistance.IκBζ is a transcriptional key regulator of CCL2/MCP-1.Gene expression analysis of thymocyte selection in vivo.The active caspase-8 heterotetramer is formed at the CD95 DISCImpaired CD95-mediated apoptosis in autoimmunity and occurrence of a p22 Caspase-8 cleavage product in JIAUp-regulation of c-FLIPS+R upon CD40 stimulation is associated with inhibition of CD95-induced apoptosis in primary precursor B-ALLUp-regulation of c-FLIP short by NFAT contributes to apoptosis resistance of short-term activated T cellsc-FLIP is crucial for IL-7/IL-15-dependent NKp46+ ILC development and protection from intestinal inflammation in miceA Hypermorphic Nfkbid Allele Contributes to Impaired Thymic Deletion of Autoreactive Diabetogenic CD8+ T Cells in NOD MiceThe NF-κB transcription factor c-Rel controls host defense against Citrobacter rodentium
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Ingo Schmitz
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P214
P1053
G-2093-2013
P106
P1153
7006151577
P21
P214
P31
P3829
P496
0000-0002-5360-0419
P734
P7859
viaf-8266021