Dscam mediates remodeling of glutamate receptors in Aplysia during de novo and learning-related synapse formation. - Test2 - elhamkhani - TriplyDB

Dscam mediates remodeling of glutamate receptors in Aplysia during de novo and learning-related synapse formation.

Dscam mediates remodeling of glutamate receptors in Aplysia during de novo and learning-related synapse formation.

http://www.wikidata.org/entity/Q30524913

about

Synaptic functions of invertebrate varicosities: what molecular mechanisms lie beneath The cell adhesion molecule neuroplastin-65 is a novel interaction partner of γ-aminobutyric acid type A receptors DSCAM and DSCAML1 function in self-avoidance in multiple cell types in the developing mouse retina Modulation of cell adhesion systems by prenatal nicotine exposure in limbic brain regions of adolescent female rats.Aplysia cell adhesion molecule and a novel protein kinase C activity in the postsynaptic neuron are required for presynaptic growth and initial formation of specific synapses.Hippocampal overexpression of Down syndrome cell adhesion molecule in amyloid precursor protein transgenic mice.Branching out: mechanisms of dendritic arborization.Drosophila dscam proteins regulate postsynaptic specificity at multiple-contact synapses.Developmentally dynamic colocalization patterns of DSCAM with adhesion and synaptic proteins in the mouse retina A novel null allele of mouse DSCAM survives to adulthood on an inbred C3H background with reduced phenotypic variability.Long-term habituation of the gill-withdrawal reflex in aplysia requires gene transcription, calcineurin and L-type voltage-gated calcium channels.A presynaptic role for FMRP during protein synthesis-dependent long-term plasticity in Aplysia.Changes in D-aspartate ion currents in the Aplysia nervous system with aging Identification of a serotonin receptor coupled to adenylyl cyclase involved in learning-related heterosynaptic facilitation in Aplysia.Protein kinase M maintains long-term sensitization and long-term facilitation in aplysia Rapid increase in clusters of synaptophysin at onset of homosynaptic potentiation in Aplysia Reconsolidation of long-term memory in Aplysia.Coordinated interaction of Down syndrome cell adhesion molecule and deleted in colorectal cancer with dynamic TUBB3 mediates Netrin-1-induced axon branching.DSCAM promotes refinement in the mouse retina through cell death and restriction of exploring dendrites.The Down syndrome critical region regulates retinogeniculate refinement Cell autonomy of DSCAM function in retinal development Nonassociative learning in invertebrates Structural Components of Synaptic Plasticity and Memory Consolidation Spontaneous transmitter release is critical for the induction of long-term and intermediate-term facilitation in Aplysia Spontaneous transmitter release recruits postsynaptic mechanisms of long-term and intermediate-term facilitation in Aplysia.Pharmacological evidence that D-aspartate activates a current distinct from ionotropic glutamate receptor currents in Aplysia californica Motor hypertonia and lack of locomotor coordination in mutant mice lacking DSCAM Role of DSCAM in the development of the spinal locomotor and sensorimotor circuits.Overexpression of Down syndrome cell adhesion molecule impairs precise synaptic targeting Common mechanisms of synaptic plasticity in vertebrates and invertebrates.Self-avoidance and tiling: Mechanisms of dendrite and axon spacing.Mapping molecular memory: navigating the cellular pathways of learning.Insular Cortex is Critical for the Perception, Modulation, and Chronification of Pain.Whereas short-term facilitation is presynaptic, intermediate-term facilitation involves both presynaptic and postsynaptic protein kinases and protein synthesis.Leveraging genome characteristics to improve gene discovery for putamen subcortical brain structure.A third copy of the Down syndrome cell adhesion molecule (Dscam) causes synaptic and locomotor dysfunction in Drosophila.An increase in synaptic NMDA receptors in the insular cortex contributes to neuropathic pain.Age- and speed-dependent modulation of gaits in DSCAM2J mutant mice.DSCAM differentially modulates pre- and postsynaptic structural and functional central connectivity during visual system wiring

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Dscam mediates remodeling of glutamate receptors in Aplysia during de novo and learning-related synapse formation.

http://www.wikidata.org/entity/Q30524913

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Dscam mediates remodeling of g ...... ing-related synapse formation.

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Dscam mediates remodeling of g ...... ing-related synapse formation.

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Dscam mediates remodeling of g ...... ing-related synapse formation.

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Dscam mediates remodeling of g ...... ing-related synapse formation.

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J.NEURON.2009.01.010

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Dscam mediates remodeling of g ...... ning-related synapse formation

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Craig H Bailey

http://www.w3.org/2001/XMLSchema#string

Eric R Kandel

http://www.w3.org/2001/XMLSchema#string

Harshad Vishwasrao

http://www.w3.org/2001/XMLSchema#string

Hsiu-Ling Li

http://www.w3.org/2001/XMLSchema#string

Iksung Jin

http://www.w3.org/2001/XMLSchema#string

Nadia Sutedja

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Robert D Hawkins

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Wei Chen

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P2860

A striking organization of a large family of human neural cadherin-like cell adhesion genes

DSCAM, a highly conserved gene in mammals, expressed in differentiating mouse brain

Mammalian DSCAMs: roles in the development of the spinal cord, cortex, and cerebellum?

Dscam2 mediates axonal tiling in the Drosophila visual system

The immunoglobulin superfamily protein SYG-1 determines the location of specific synapses in C. elegans

Neurite arborization and mosaic spacing in the mouse retina require DSCAM.

Drosophila Dscam is an axon guidance receptor exhibiting extraordinary molecular diversity

Sidekicks: synaptic adhesion molecules that promote lamina-specific connectivity in the retina.

Dscam diversity is essential for neuronal wiring and self-recognition

A vast repertoire of Dscam binding specificities arises from modular interactions of variable Ig domains.

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