Inactivation of a putative flagellar motor switch protein FliG1 prevents Borrelia burgdorferi from swimming in highly viscous media and blocks its infectivity
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Analysis of the Borrelia burgdorferi cyclic-di-GMP-binding protein PlzA reveals a role in motility and virulenceAnalysis of a Spontaneous Non-Motile and Avirulent Mutant Shows That FliM Is Required for Full Endoflagella Assembly in Leptospira interrogansSpirochaete flagella hook proteins self-catalyse a lysinoalanine covalent crosslink for motilityFlaA proteins in Leptospira interrogans are essential for motility and virulence but are not required for formation of the flagellum sheathTwo residues predominantly dictate functional difference in motility between Shewanella oneidensis flagellins FlaA and FlaBAnalysis of a Borrelia burgdorferi phosphodiesterase demonstrates a role for cyclic-di-guanosine monophosphate in motility and virulence.Chemoreceptors and flagellar motors are subterminally located in close proximity at the two cell poles in spirochetes.A novel gene inactivation system reveals altered periplasmic flagellar orientation in a Borrelia burgdorferi fliL mutantTwo CheW coupling proteins are essential in a chemosensory pathway of Borrelia burgdorferi.The unique paradigm of spirochete motility and chemotaxis.Motility and Ultrastructure of Spirochaeta thermophilaCarbon storage regulator A (CsrA(Bb)) is a repressor of Borrelia burgdorferi flagellin protein FlaB.Transposon mutagenesis as an approach to improved understanding of Borrelia pathogenesis and biologyImpact of maintenance immunosuppressive therapy on the fecal microbiome of renal transplant recipients: Comparison between an everolimus- and a standard tacrolimus-based regimen.Molecular architecture of the bacterial flagellar motor in cellsAnalysis of an ordered, comprehensive STM mutant library in infectious Borrelia burgdorferi: insights into the genes required for mouse infectivity.Spirochetal motility and chemotaxis in the natural enzootic cycle and development of Lyme diseasePeriplasmic flagellar export apparatus protein, FliH, is involved in post-transcriptional regulation of FlaB, motility and virulence of the relapsing fever spirochete Borrelia hermsii.CheY3 of Borrelia burgdorferi is the key response regulator essential for chemotaxis and forms a long-lived phosphorylated intermediate.Analysis of the HD-GYP domain cyclic dimeric GMP phosphodiesterase reveals a role in motility and the enzootic life cycle of Borrelia burgdorferi.Motor rotation is essential for the formation of the periplasmic flagellar ribbon, cellular morphology, and Borrelia burgdorferi persistence within Ixodes scapularis tick and murine hosts.Mutations in the Borrelia burgdorferi Flagellar Type III Secretion System Genes fliH and fliI Profoundly Affect Spirochete Flagellar Assembly, Morphology, Motility, Structure, and Cell Division.Study of the response regulator Rrp1 reveals its regulatory role in chitobiose utilization and virulence of Borrelia burgdorferi.Motility is crucial for the infectious life cycle of Borrelia burgdorferi.Borrelia burgdorferi CheD Promotes Various Functions in Chemotaxis and the Pathogenic Life Cycle of the SpirocheteCryoelectron tomography reveals the sequential assembly of bacterial flagella in Borrelia burgdorferi.A single-domain FlgJ contributes to flagellar hook and filament formation in the Lyme disease spirochete Borrelia burgdorferi.Periplasmic flagella in Borrelia burgdoferi function to maintain cellular integrity upon external stress.Borrelia burgdorferi needs chemotaxis to establish infection in mammals and to accomplish its enzootic cycle.Inactivation of bb0184, which encodes carbon storage regulator A, represses the infectivity of Borrelia burgdorferi.The riboswitch regulates a thiamine pyrophosphate ABC transporter of the oral spirochete Treponema denticola.Characterization of Stress and Innate Immunity Resistance of Wild-type and Δp66 Borrelia burgdorferi.Measuring Borrelia burgdorferi Motility and Chemotaxis.The effect of bacterial chemotaxis on host infection and pathogenicity.A novel glycan modifies the flagellar filament proteins of the oral bacterium Treponema denticola.
P2860
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P2860
Inactivation of a putative flagellar motor switch protein FliG1 prevents Borrelia burgdorferi from swimming in highly viscous media and blocks its infectivity
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Inactivation of a putative fla ...... dia and blocks its infectivity
@ast
Inactivation of a putative fla ...... dia and blocks its infectivity
@en
type
label
Inactivation of a putative fla ...... dia and blocks its infectivity
@ast
Inactivation of a putative fla ...... dia and blocks its infectivity
@en
prefLabel
Inactivation of a putative fla ...... dia and blocks its infectivity
@ast
Inactivation of a putative fla ...... dia and blocks its infectivity
@en
P2093
P2860
P1476
Inactivation of a putative fla ...... dia and blocks its infectivity
@en
P2093
Chunhao Li
Fang Ting Liang
Hongbin Xu
P2860
P304
P356
10.1111/J.1365-2958.2010.07078.X
P407
P577
2010-02-18T00:00:00Z