Tet1 is critical for neuronal activity-regulated gene expression and memory extinction. - Test2 - elhamkhani - TriplyDB

Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

http://www.wikidata.org/entity/Q30661863

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The emerging insights into catalytic or non-catalytic roles of TET proteins in tumors and neural development Where Environment Meets Cognition: A Focus on Two Developmental Intellectual Disability Disorders Modifiers and Readers of DNA Modifications and Their Impact on Genome Structure, Expression, and Stability in Disease Making Sense of Epigenetics Epigenetic Mechanisms in Developmental Alcohol-Induced Neurobehavioral Deficits Epigenetic Alterations in Alzheimer's Disease New Insights on Retrieval-Induced and Ongoing Memory Consolidation: Lessons from Arc Dynamic DNA methylation in the brain: a new epigenetic mark for experience-dependent plasticity Turning over DNA methylation in the mind Unlocking epigenetic codes in neurogenesis Pharmacology of cognitive enhancers for exposure-based therapy of fear, anxiety and trauma-related disorders Role of TET enzymes in DNA methylation, development, and cancer Epigenetics and therapeutic targets mediating neuroprotection Molecular mechanisms of synaptic remodeling in alcoholism Base-resolution profiling of active DNA demethylation using MAB-seq and caMAB-seq Ascorbic acid and the brain: rationale for the use against cognitive decline.Overweight and CpG methylation of the Pomc promoter in offspring of high-fat-diet-fed dams are not "reprogrammed" by regular chow diet in rats.Neocortical Tet3-mediated accumulation of 5-hydroxymethylcytosine promotes rapid behavioral adaptation Whole-genome analysis of 5-hydroxymethylcytosine and 5-methylcytosine at base resolution in the human brain.Impairment in extinction of contextual and cued fear following post-training whole-body irradiation Epigenetic mechanisms during ageing and neurogenesis as novel therapeutic avenues in human brain disorders.Ten-eleven translocation 2 interacts with forkhead box O3 and regulates adult neurogenesis.Methyl-CpG binding domain protein 1 regulates localization and activity of Tet1 in a CXXC3 domain-dependent manner.Enrichment methods provide a feasible approach to comprehensive and adequately powered investigations of the brain methylome An evolving view of epigenetic complexity in the brain.DNA modifications in the mammalian brain Insights into DNA hydroxymethylation in the honeybee from in-depth analyses of TET dioxygenase.Mechanisms of epigenetic memory and addiction.Epigenetic memory: the Lamarckian brain On the resilience of remote traumatic memories against exposure therapy-mediated attenuation.New approaches to manipulating the epigenome.Epigenetic regulation of pluripotency and differentiation Age-associated decrease in global DNA methylation in patients with major depression.Epigenetic mechanisms of memory formation and reconsolidation.5-Hydroxymethylcytosine: a stable or transient DNA modification?Epigenetic mechanisms in fear conditioning: implications for treating post-traumatic stress disorder.'Memory and molecular turnover,' 30 years after inception.Genome-wide antagonism between 5-hydroxymethylcytosine and DNA methylation in the adult mouse brain.The emerging nexus of active DNA demethylation and mitochondrial oxidative metabolism in post-mitotic neurons Energizing Genetics and Epi-genetics: Role in the Regulation of Mitochondrial Function.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

http://www.wikidata.org/entity/Q30661863

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2013 nî lūn-bûn

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2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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J.NEURON.2013.08.003

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Tet1 is critical for neuronal activity-regulated gene expression and memory extinction.

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Albert W Cheng

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Andrii Rudenko

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Jinsoo Seo

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Kym F Faull

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Li-Huei Tsai

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Meelad M Dawlaty

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Thuc Le

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P2860

The nuclear DNA base 5-hydroxymethylcytosine is present in Purkinje neurons and the brain

Epigenetic regulation of gene expression: how the genome integrates intrinsic and environmental signals

Hydroxylation of 5-methylcytosine by TET1 promotes active DNA demethylation in the adult brain

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

An epigenetic blockade of cognitive functions in the neurodegenerating brain

Driving fast-spiking cells induces gamma rhythm and controls sensory responses

Emotional processing in anterior cingulate and medial prefrontal cortex

A hippocampal insulin-growth factor 2 pathway regulates the extinction of fear memories

Tet-mediated formation of 5-carboxylcytosine and its excision by TDG in mammalian DNA

A hippocampal Cdk5 pathway regulates extinction of contextual fear

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10.1016/J.NEURON.2013.08.003

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79

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Rudolf Jaenisch

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