Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation
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Metabolic Dysfunction Underlying Autism Spectrum Disorder and Potential Treatment Approaches.Rigor and Reproducibility in Rodent Behavioral Research.Autism-like behavior caused by deletion of vaccinia-related kinase 3 is improved by TrkB stimulation.Integrated multifactor analysis explores core dysfunctional modules in autism spectrum disorder.
P2860
Increased expression of AT-1/SLC33A1 causes an autistic-like phenotype in mice by affecting dendritic branching and spine formation
description
2016 nî lūn-bûn
@nan
2016 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Increased expression of AT-1/S ...... branching and spine formation
@ast
Increased expression of AT-1/S ...... branching and spine formation
@en
type
label
Increased expression of AT-1/S ...... branching and spine formation
@ast
Increased expression of AT-1/S ...... branching and spine formation
@en
prefLabel
Increased expression of AT-1/S ...... branching and spine formation
@ast
Increased expression of AT-1/S ...... branching and spine formation
@en
P2093
P2860
P50
P356
P1476
Increased expression of AT-1/S ...... branching and spine formation
@en
P2093
Heather A Mitchell
James A Dowell
Jingxin Wang
John M Denu
Lingjun Li
Luigi Puglielli
Rikki Hullinger
Yajing Peng
P2860
P304
P356
10.1084/JEM.20151776
P407
P577
2016-05-30T00:00:00Z