Helicobacter pylori perturbs iron trafficking in the epithelium to grow on the cell surface - Test2 - elhamkhani - TriplyDB

Helicobacter pylori perturbs iron trafficking in the epithelium to grow on the cell surface

Helicobacter pylori perturbs iron trafficking in the epithelium to grow on the cell surface

http://www.wikidata.org/entity/Q31010231

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An Overview of Helicobacter pylori VacA Toxin Biology Helicobacter pylori Diversity and Gastric Cancer Risk Metalloregulation of Helicobacter pylori physiology and pathogenesis Helicobacter pylori in gastric carcinogenesis: mechanisms The ferric uptake regulator of Helicobacter pylori: a critical player in the battle for iron and colonization of the stomach Helicobacter and salmonella persistent infection strategies Polymorphism in the Helicobacter pylori CagA and VacA toxins and disease Chemodetection and Destruction of Host Urea Allows Helicobacter pylori to Locate the Epithelium.The adherens junctions control susceptibility to Staphylococcus aureus α-toxin Structure of the Helicobacter pylori CagA oncoprotein bound to the human tumor suppressor ASPP2 Nutrition and Helicobacter pylori: Host Diet and Nutritional Immunity Influence Bacterial Virulence and Disease Outcome Iron deficiency accelerates Helicobacter pylori-induced carcinogenesis in rodents and humans Pathobiology of Helicobacter pylori-Induced Gastric Cancer.Dietary factors modulate Helicobacter-associated gastric cancer in rodent models Gastric Helicobacter infection induces iron deficiency in the INS-GAS mouse Functional plasticity in the type IV secretion system of Helicobacter pylori Sequence divergence and conservation in genomes of Helicobacter cetorum strains from a dolphin and a whale Helicobacter pylori targets cancer-associated apical-junctional constituents in gastroids and gastric epithelial cells Helicobacter pylori: Genomic Insight into the Host-Pathogen Interaction.High resolution electron microscopy of the Helicobacter pylori Cag type IV secretion system pili produced in varying conditions of iron availability.Regulation of Helicobacter pylori Virulence Within the Context of Iron Deficiency.In vivo expression of Helicobacter pylori virulence genes in patients with gastritis, ulcer, and gastric cancer.Molecular mechanisms of gastric epithelial cell adhesion and injection of CagA by Helicobacter pylori.The Human Antimicrobial Protein Calgranulin C Participates in Control of Helicobacter pylori Growth and Regulation of Virulence.Chemorepulsion from the Quorum Signal Autoinducer-2 Promotes Helicobacter pylori Biofilm Dispersal.Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system.Helicobacter pylori Infection Induces Anemia, Depletes Serum Iron Storage, and Alters Local Iron-Related and Adult Brain Gene Expression in Male INS-GAS Mice.Identification of genetic modifiers of CagA-induced epithelial disruption in Drosophila Alterations in Helicobacter pylori triggered by contact with gastric epithelial cells Multiple Acid Sensors Control Helicobacter pylori Colonization of the Stomach Micronutrients: A double-edged sword in microbial-induced gastric carcinogenesis.Helicobacter pylori infection and low dietary iron alter behavior, induce iron deficiency anemia, and modulate hippocampal gene expression in female C57BL/6 mice.The basolateral vesicle sorting machinery and basolateral proteins are recruited to the site of enteropathogenic E. coli microcolony growth at the apical membrane.Structural Insights into Polymorphic ABO Glycan Binding by Helicobacter pylori High dietary salt intake exacerbates Helicobacter pylori-induced gastric carcinogenesis Natural history of Helicobacter pylori infection in Mexican schoolchildren: incidence and spontaneous clearance.Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.Role of energy sensor TlpD of Helicobacter pylori in gerbil colonization and genome analyses after adaptation in the gerbil.Who's really in control: microbial regulation of protein trafficking in the epithelium.Diet, microbial virulence, and Helicobacter pylori-induced gastric cancer.

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P2860

Helicobacter pylori perturbs iron trafficking in the epithelium to grow on the cell surface

http://www.wikidata.org/entity/Q31010231

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Helicobacter pylori perturbs i ...... um to grow on the cell surface

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Helicobacter pylori perturbs i ...... um to grow on the cell surface

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Helicobacter pylori perturbs i ...... um to grow on the cell surface

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JOURNAL.PPAT.1002050

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Helicobacter pylori perturbs i ...... um to grow on the cell surface

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Jennifer M Noto

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Judith Romero-Gallo

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Manuel R Amieva

http://www.w3.org/2001/XMLSchema#string

Richard M Peek

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Shumin Tan

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P2860

The spatial orientation of Helicobacter pylori in the gastric mucus

Helicobacter pylori SabA adhesin in persistent infection and chronic inflammation

Current concepts in the management of Helicobacter pylori infection: the Maastricht III Consensus Report

Listeria monocytogenes invades the epithelial junctions at sites of cell extrusion

Listeria monocytogenes internalin B activates junctional endocytosis to accelerate intestinal invasion

Helicobacter pylori usurps cell polarity to turn the cell surface into a replicative niche

The serine protease TMPRSS6 is required to sense iron deficiency

The Helicobacter pylori VacA toxin is a urea permease that promotes urea diffusion across epithelia

Phosphatidylinositol 3,4,5-trisphosphate localization in recycling endosomes is necessary for AP-1B-dependent sorting in polarized epithelial cells

Helicobacter pylori CagA induces a transition from polarized to invasive phenotypes in MDCK cells.

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21589900

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Helicobacter pylori

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3093365

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