Noninfectious retrovirus particles drive the APOBEC3/Rfv3 dependent neutralizing antibody response - Test2 - elhamkhani - TriplyDB

Noninfectious retrovirus particles drive the APOBEC3/Rfv3 dependent neutralizing antibody response

Noninfectious retrovirus particles drive the APOBEC3/Rfv3 dependent neutralizing antibody response

http://www.wikidata.org/entity/Q31036522

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Viral quasispecies evolution Studies on the restriction of murine leukemia viruses by mouse APOBEC3 Tetherin/BST-2: Restriction Factor or Immunomodulator?Type I interferon signaling is required for the APOBEC3/Rfv3-dependent neutralizing antibody response but not innate retrovirus restriction.Immunoglobulin somatic hypermutation by APOBEC3/Rfv3 during retroviral infection Immunoglobulin VH gene diversity and somatic hypermutation during SIV infection of rhesus macaques.Incorporation of mouse APOBEC3 into murine leukemia virus virions decreases the activity and fidelity of reverse transcriptase.Late-emerging strains of HIV induce T-cell homeostasis failure by promoting bystander cell death and immune exhaustion in naïve CD4 and all CD8 T-cells.Mouse knockout models for HIV-1 restriction factors.Tetherin promotes the innate and adaptive cell-mediated immune response against retrovirus infection in vivo.A single nucleotide polymorphism in tetherin promotes retrovirus restriction in vivo.Fv1 restriction and retrovirus vaccine immunity in Apobec3-deficient 129P2 mice.Friend retrovirus drives cytotoxic effectors through Toll-like receptor 3 Requirement for Fc effector mechanisms in the APOBEC3/Rfv3-dependent neutralizing antibody response Reassessment of murine APOBEC1 as a retrovirus restriction factor in vivo.Interferon-α Subtypes in an Ex Vivo Model of Acute HIV-1 Infection: Expression, Potency and Effector Mechanisms T Cell Production of IFNγ in Response to TLR7/IL-12 Stimulates Optimal B Cell Responses to Viruses.Tetherin/BST-2 promotes dendritic cell activation and function during acute retrovirus infection.Restriction by SAMHD1 Limits cGAS/STING-Dependent Innate and Adaptive Immune Responses to HIV-1.Ribonuclease L is not critical for innate restriction and adaptive immunity against Friend retrovirus infection Humoral immunity in the Friend retrovirus infection model.APOBECs and virus restriction.IFN-α treatment inhibits acute Friend retrovirus replication primarily through the antiviral effector molecule Apobec3.APOBEC3 enzymes restrict marginal zone B cells.[Exosomes are carriers for immunostimulatory viral RNA].

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Noninfectious retrovirus particles drive the APOBEC3/Rfv3 dependent neutralizing antibody response

http://www.wikidata.org/entity/Q31036522

description

2011 nî lūn-bûn

@nan

2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Noninfectious retrovirus parti ...... neutralizing antibody response

@ast

Noninfectious retrovirus parti ...... neutralizing antibody response

@en

type

label

Noninfectious retrovirus parti ...... neutralizing antibody response

@ast

Noninfectious retrovirus parti ...... neutralizing antibody response

@en

prefLabel

Noninfectious retrovirus parti ...... neutralizing antibody response

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Noninfectious retrovirus parti ...... neutralizing antibody response

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Noninfectious retrovirus parti ...... neutralizing antibody response

@en

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Bradley S Barrett

http://www.w3.org/2001/XMLSchema#string

Diana S Smith

http://www.w3.org/2001/XMLSchema#string

Karl J Heilman

http://www.w3.org/2001/XMLSchema#string

Kejun Guo

http://www.w3.org/2001/XMLSchema#string

Kim J Hasenkrug

http://www.w3.org/2001/XMLSchema#string

Leonard H Evans

http://www.w3.org/2001/XMLSchema#string

Warner C Greene

http://www.w3.org/2001/XMLSchema#string

P2860

Human APOBEC3F is another host factor that blocks human immunodeficiency virus type 1 replication

SAMHD1 is the dendritic- and myeloid-cell-specific HIV-1 restriction factor counteracted by Vpx

Induction of APOBEC3 family proteins, a defensive maneuver underlying interferon-induced anti-HIV-1 activity

Human immunodeficiency virus type 1 cDNAs produced in the presence of APOBEC3G exhibit defects in plus-strand DNA transfer and integration

Natural variation in Vif: differential impact on APOBEC3G/3F and a potential role in HIV-1 diversification

Retrovirus envelope domain at 1.7 angstrom resolution

Isolation of a human gene that inhibits HIV-1 infection and is suppressed by the viral Vif protein

Species-specific exclusion of APOBEC3G from HIV-1 virions by Vif

HIV-1 Vif blocks the antiviral activity of APOBEC3G by impairing both its translation and intracellular stability

Apobec3 encodes Rfv3, a gene influencing neutralizing antibody control of retrovirus infection

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e1002284

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scholarly article

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10.1371/JOURNAL.PPAT.1002284

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10

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7

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Mario L. Santiago

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2011-10-06T00:00:00Z

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51716525

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21998583

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3188525

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