The scavenger protein apoptosis inhibitor of macrophages (AIM) potentiates the antimicrobial response against Mycobacterium tuberculosis by enhancing autophagy.
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Sensing of Mycobacterium tuberculosis and consequences to both host and bacillusCell death and autophagy in tuberculosisMycobacterium tuberculosis PE_PGRS41 Enhances the Intracellular Survival of M. smegmatis within Macrophages Via Blocking Innate Immunity and Inhibition of Host Defense.Persistent Unresolved Inflammation in the Mecp2-308 Female Mutated Mouse Model of Rett SyndromeIdentification of modules related to programmed cell death in CHD based on EHENDifferential transcriptional response in macrophages infected with cell wall deficient versus normal Mycobacterium Tuberculosis.The human CD5L/AIM-CD36 axis: A novel autophagy inducer in macrophages that modulates inflammatory responses.Measuring Phagosome pH by Ratiometric Fluorescence Microscopy.Interaction of AIM with insulin-like growth factor-binding protein-4.Alu repeats as transcriptional regulatory platforms in macrophage responses to M. tuberculosis infection.AIM/CD5L: a key protein in the control of immune homeostasis and inflammatory disease.Apoptosis inhibitor of macrophage (AIM) reduces cell number in canine histiocytic sarcoma cell lines.Inflammatory protein response in CDKL5-Rett syndrome: evidence of a subclinical smouldering inflammation.Differential macrophage response to slow- and fast-growing pathogenic mycobacteria.The macrophage soluble receptor AIM/Api6/CD5L displays a broad pathogen recognition spectrum and is involved in early response to microbial aggressionTLR4-NOX2 axis regulates the phagocytosis and killing of Mycobacterium tuberculosis by macrophages.Mesenchymal stem cells internalize Mycobacterium tuberculosis through scavenger receptors and restrict bacterial growth through autophagy.Orchestrating Role of Apoptosis Inhibitor of Macrophage in the Resolution of Acute Lung Injury.IL-36/LXR axis modulates cholesterol metabolism and immune defense to Mycobacterium tuberculosis.CD5L Promotes M2 Macrophage Polarization through Autophagy-Mediated Upregulation of ID3.A Beneficial Effect of Low-Dose Aspirin in a Murine Model of Active Tuberculosis.
P2860
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P2860
The scavenger protein apoptosis inhibitor of macrophages (AIM) potentiates the antimicrobial response against Mycobacterium tuberculosis by enhancing autophagy.
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2013 nî lūn-bûn
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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
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2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2013年の論文
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The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@ast
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@en
type
label
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@ast
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@en
prefLabel
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@ast
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@en
P2093
P2860
P50
P921
P1433
P1476
The scavenger protein apoptosi ...... ulosis by enhancing autophagy.
@en
P2093
Elena Marzo
Lucía Sanjurjo
Marta Valeri
Neus Cáceres
Núria Amézaga
P2860
P304
P356
10.1371/JOURNAL.PONE.0079670
P407
P577
2013-11-04T00:00:00Z