Differential expression of forkhead box transcription factors following butylated hydroxytoluene lung injury.
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Genomic and genic deletions of the FOX gene cluster on 16q24.1 and inactivating mutations of FOXF1 cause alveolar capillary dysplasia and other malformationsPulmonary mastocytosis and enhanced lung inflammation in mice heterozygous null for the Foxf1 geneForkhead box F1 is essential for migration of mesenchymal cells and directly induces integrin-beta3 expressionTranscription profiling of lung adenocarcinomas of c-myc-transgenic mice: identification of the c-myc regulatory gene network.Molecular characterization of lung dysplasia induced by c-Raf-1.Pathogenetics of alveolar capillary dysplasia with misalignment of pulmonary veinsExpression of Foxm1 transcription factor in cardiomyocytes is required for myocardial development.Transcription factors in mouse lung development and function.Endothelial cell-specific deletion of transcription factor FoxM1 increases urethane-induced lung carcinogenesis.Differentiated cultures of primary hamster tracheal airway epithelial cellsAlveolar capillary dysplasiaAnti-inflammatory effects of the neurotransmitter agonist Honokiol in a mouse model of allergic asthma.Foxm1 regulates resolution of hyperoxic lung injury in newborns.FOXM1 participates in PLK1-regulated cell cycle progression in renal cell cancer cells.Novel FOXF1 mutations in sporadic and familial cases of alveolar capillary dysplasia with misaligned pulmonary veins imply a role for its DNA binding domain.Lethal lung hypoplasia and vascular defects in mice with conditional Foxf1 overexpression.FOXF1 transcription factor is required for formation of embryonic vasculature by regulating VEGF signaling in endothelial cells.Foxf1 haploinsufficiency reduces Notch-2 signaling during mouse lung development.Postnatal ablation of Foxm1 from cardiomyocytes causes late onset cardiac hypertrophy and fibrosis without exacerbating pressure overload-induced cardiac remodeling.FOXF1 transcription factor promotes lung regeneration after partial pneumonectomy.Genomic and Epigenetic Complexity of the FOXF1 Locus in 16q24.1: Implications for Development and Disease.Conditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Foxm1 transcription factor is required for lung fibrosis and epithelial-to-mesenchymal transition.Wild-type levels of the mouse Forkhead Box f1 gene are essential for lung repair.Fusion of lung lobes and vessels in mouse embryos heterozygous for the forkhead box f1 targeted allele.
P2860
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P2860
Differential expression of forkhead box transcription factors following butylated hydroxytoluene lung injury.
description
2001 nî lūn-bûn
@nan
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Differential expression of for ...... ed hydroxytoluene lung injury.
@ast
Differential expression of for ...... ed hydroxytoluene lung injury.
@en
type
label
Differential expression of for ...... ed hydroxytoluene lung injury.
@ast
Differential expression of for ...... ed hydroxytoluene lung injury.
@en
prefLabel
Differential expression of for ...... ed hydroxytoluene lung injury.
@ast
Differential expression of for ...... ed hydroxytoluene lung injury.
@en
P2093
P2860
P1476
Differential expression of for ...... ed hydroxytoluene lung injury.
@en
P2093
P2860
P304
P356
10.1152/AJPLUNG.2001.280.4.L695
P577
2001-04-01T00:00:00Z