Reactive oxygen species as a signal in glucose-stimulated insulin secretion. - Test2 - elhamkhani - TriplyDB

Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

http://www.wikidata.org/entity/Q33280759

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Roles of Oxidative Stress in Polycystic Ovary Syndrome and Cancers Selenium and diabetes--evidence from animal studies Oxidative stress and diabetes: what can we learn about insulin resistance from antioxidant mutant mouse models?Evaluation of the association between arsenic and diabetes: a National Toxicology Program workshop review Physiological effects and therapeutic potential of proinsulin C-peptide Redox homeostasis in pancreatic β cells Chemiluminescence Imaging of Superoxide Anion Detects Beta-Cell Function and Mass At the interface of antioxidant signalling and cellular function: Key polyphenol effects Triclosan in water, implications for human and environmental health Oxidative stress in obesity: a critical component in human diseases CNC-bZIP protein Nrf1-dependent regulation of glucose-stimulated insulin secretion Molecular basis for vulnerability to mitochondrial and oxidative stress in a neuroendocrine CRI-G1 cell line Uncoupling protein-2 attenuates glucose-stimulated insulin secretion in INS-1E insulinoma cells by lowering mitochondrial reactive oxygen species Expression of transient receptor potential ankyrin 1 (TRPA1) and its role in insulin release from rat pancreatic beta cells Reduced antioxidant capacity and diet-induced atherosclerosis in uncoupling protein-2-deficient mice.Persistent oxidative stress due to absence of uncoupling protein 2 associated with impaired pancreatic beta-cell function.Diabetic beta-cells can achieve self-protection against oxidative stress through an adaptive up-regulation of their antioxidant defenses.A gene-environment interaction analysis of plasma selenium with prevalent and incident diabetes: The Hortega study.YES, a Src family kinase, is a proximal glucose-specific activator of cell division cycle control protein 42 (Cdc42) in pancreatic islet β cells A systems biology perspective on Nrf2-mediated antioxidant response ROS signaling, oxidative stress and Nrf2 in pancreatic beta-cell function.Nuclear factor erythroid 2-related factor 2 deletion impairs glucose tolerance and exacerbates hyperglycemia in type 1 diabetic mice.Measurement of DCF fluorescence as a measure of reactive oxygen species in murine islets of Langerhans Mitochondrial transcription factor B2 is essential for mitochondrial and cellular function in pancreatic β-cells.Regulation of insulin secretion: role of mitochondrial signalling.Glucose sensing in the pancreatic beta cell: a computational systems analysis.Sumoylation modulates oxidative stress relevant to the viability and functionality of pancreatic beta cells.Low-level arsenic impairs glucose-stimulated insulin secretion in pancreatic beta cells: involvement of cellular adaptive response to oxidative stress.Control of the intracellular redox state by glucose participates in the insulin secretion mechanism Influence of arsenate and arsenite on signal transduction pathways: an update Tiam1/Rac1 signaling pathway mediates palmitate-induced, ceramide-sensitive generation of superoxides and lipid peroxides and the loss of mitochondrial membrane potential in pancreatic beta-cells.Mitochondrial uncoupling and lifespan.Reactive oxygen species stimulate insulin secretion in rat pancreatic islets: studies using mono-oleoyl-glycerol Reactive oxygen species facilitate translocation of hormone sensitive lipase to the lipid droplet during lipolysis in human differentiated adipocytes Methylglyoxal mediates adipocyte proliferation by increasing phosphorylation of Akt1 Heterozygous SOD2 deletion impairs glucose-stimulated insulin secretion, but not insulin action, in high-fat-fed mice.Recent advances in hydrogen peroxide imaging for biological applications Two tales of antioxidant enzymes on β cells and diabetes.Identification of ATP synthase as a lipid peroxide protein adduct in pancreatic islets from humans with and without type 2 diabetes mellitus.Phagocyte-like NADPH oxidase generates ROS in INS 832/13 cells and rat islets: role of protein prenylation.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

http://www.wikidata.org/entity/Q33280759

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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type

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion.

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Jeffrey M Reece

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Kiefer Daniel

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Lisa M Floering

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Qiang Zhang

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