Deficiency of invariant V alpha 14 natural killer T cells decreases atherosclerosis in LDL receptor null mice
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Missense mutation in APOC3 within the C-terminal lipid binding domain of human ApoC-III results in impaired assembly and secretion of triacylglycerol-rich very low density lipoproteins: evidence that ApoC-III plays a major role in the formation of lCardiovascular changes in atherosclerotic ApoE-deficient mice exposed to Co60 (γ) radiationToll-like receptor 3 ligand dampens liver inflammation by stimulating Valpha 14 invariant natural killer T cells to negatively regulate gammadeltaT cells.Statins impair CD1d-mediated antigen presentation through the inhibition of prenylation.Impact of CD1d deficiency on metabolism.Reciprocal interaction between macrophages and T cells stimulates IFN-γ and MCP-1 production in Ang II-induced cardiac inflammation and fibrosisLymphocytes in atherosclerosis.Lymphocyte subpopulations in myocardial infarction: a comparison between peripheral and intracoronary blood.Natural killer T cells are required for lipopolysaccharide-mediated enhancement of atherosclerosis in apolipoprotein E-deficient mice.ATP-binding cassette transporter G1 intrinsically regulates invariant NKT cell developmentNatural killer T cells and atherosclerosis: form and function meet pathogenesisInflammation and immune system interactions in atherosclerosis.Molecular biology of atherosclerosis.Dendritic cells in atherosclerotic inflammation: the complexity of functions and the peculiarities of pathophysiological effectsAre human iNKT cells keeping tabs on lipidome perturbations triggered by oxidative stress in the blood?Natural killer T cells in atherosclerosis.Cytotoxic lymphocytes and atherosclerosis: significance, mechanisms and therapeutic challenges.A CD1d-dependent lipid antagonist to NKT cells ameliorates atherosclerosis in ApoE-/- mice by reducing lesion necrosis and inflammation.Human Invariant NKT Cells Induce IL-1β Secretion by Peripheral Blood Monocytes via a P2X7-Independent Pathway.Activation of decidual invariant natural killer T cells promotes lipopolysaccharide-induced preterm birth.Deficiency of Invariant Natural Killer T Cells Does Not Protect Against Obesity but Exacerbates Atherosclerosis in Ldlr-/- Mice.Natural killer T cells in lipoprotein metabolism and atherosclerosis.
P2860
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P2860
Deficiency of invariant V alpha 14 natural killer T cells decreases atherosclerosis in LDL receptor null mice
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@ast
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@en
Deficiency of invariant V alph ...... sis in LDL receptor null mice.
@nl
type
label
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@ast
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@en
Deficiency of invariant V alph ...... sis in LDL receptor null mice.
@nl
prefLabel
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@ast
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@en
Deficiency of invariant V alph ...... sis in LDL receptor null mice.
@nl
P2093
P2860
P356
P1476
Deficiency of invariant V alph ...... osis in LDL receptor null mice
@en
P2093
Jessica Gage
Leah Rogers
Lindsay Willcox
Mirela Hasu
Mohamad Thabet
Sarah Burchat
Stewart C Whitman
Tanya A Ramsamy
P2860
P304
P356
10.1093/CVR/CVN005
P577
2008-01-10T00:00:00Z