Criteria for empiric treatment of hyperfibrinolysis after trauma.
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TACTIC: Trans-Agency Consortium for Trauma-Induced CoagulopathyEffect of thromboelastography (TEG®) and rotational thromboelastometry (ROTEM®) on diagnosis of coagulopathy, transfusion guidance and mortality in trauma: descriptive systematic reviewFibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.Tranexamic acid and trauma-induced coagulopathy.Fibrinogen and platelet contributions to clot formation: implications for trauma resuscitation and thromboprophylaxisThrombelastographic pattern recognition in renal disease and traumaThrombin generation and fibrin clot formation under hypothermic conditions: an in vitro evaluation of tissue factor initiated whole blood coagulation.Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapyChallenges in definitive fracture management of blast injuriesDesign of the Study of Tranexamic Acid during Air Medical Prehospital Transport (STAAMP) Trial: Addressing the Knowledge Gaps.The "Death Diamond": Rapid thrombelastography identifies lethal hyperfibrinolysis.Shock-induced systemic hyperfibrinolysis is attenuated by plasma-first resuscitation.Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.Management of traumatic haemorrhage--the European perspective.Hyperfibrinolysis in severe isolated traumatic brain injury may occur without tissue hypoperfusion: a retrospective observational multicentre study.Pathophysiology of trauma-induced coagulopathy: disseminated intravascular coagulation with the fibrinolytic phenotype.Tranexamic Acid Use in Prehospital Uncontrolled Hemorrhage.A principal component analysis of coagulation after trauma.[Uncritical use of tranexamic acid in trauma patients : Do no further harm!]Stability of tranexamic acid after 12-week storage at temperatures from -20°c to 50°c.Exposing the bidirectional effects of alcohol on coagulation in trauma: Impaired clot formation and decreased fibrinolysis in rotational thromboelastometry.Fibrinolysis in trauma: a review.Harmful or Physiologic: Diagnosing Fibrinolysis Shutdown in a Trauma Cohort With Rotational Thromboelastometry.The coagulopathy of trauma.7.5% NaCl Resuscitation Leads to Abnormal Clot Fibrinolysis after Severe Hemorrhagic Shock and its Correction with 7.5% NaCl Adenosine, Lidocaine, and Mg2.
P2860
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P2860
Criteria for empiric treatment of hyperfibrinolysis after trauma.
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2012 nî lūn-bûn
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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
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2012 թվականի հուլիսին հրատարակված գիտական հոդված
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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Criteria for empiric treatment of hyperfibrinolysis after trauma.
@ast
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@en
type
label
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@ast
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@en
prefLabel
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@ast
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@en
P2093
P2860
P1476
Criteria for empiric treatment of hyperfibrinolysis after trauma.
@en
P2093
Brittney J Redick
Ian M Crane
Leslie M Cachola
Mary F Nelson
Matthew E Kutcher
Michael W Cripps
Mitchell Jay Cohen
Molly D Greenberg
Ryan C McCreery
P2860
P356
10.1097/TA.0B013E3182598C70
P577
2012-07-01T00:00:00Z