Deletion of Crry and DAF on murine platelets stimulates thrombopoiesis and increases factor H-dependent resistance of peripheral platelets to complement attack.
about
Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia-reperfusion injury.Murine systemic thrombophilia and hemolytic uremic syndrome from a factor H point mutation.Complements are involved in alcoholic fatty liver disease, hepatitis and fibrosis
P2860
Deletion of Crry and DAF on murine platelets stimulates thrombopoiesis and increases factor H-dependent resistance of peripheral platelets to complement attack.
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2013 nî lūn-bûn
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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած
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2013 թվականի փետրվարին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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name
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@ast
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@en
type
label
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@ast
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@en
prefLabel
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@ast
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@en
P2093
P2860
P356
P1476
Deletion of Crry and DAF on mu ...... latelets to complement attack.
@en
P2093
Lidia Barata
Sayaka Sato
Takashi Miwa
Wen-Chao Song
P2860
P304
P356
10.4049/JIMMUNOL.1202536
P407
P577
2013-02-06T00:00:00Z