Transcriptome analysis of synaptoneurosomes identifies neuroplasticity genes overexpressed in incipient Alzheimer's disease.
about
Early mechanisms of pathobiology are revealed by transcriptional temporal dynamics in hippocampal CA1 neurons of prion infected miceDegradation of high affinity HuD targets releases Kv1.1 mRNA from miR-129 repression by mTORC1EphA4 activation of c-Abl mediates synaptic loss and LTP blockade caused by amyloid-β oligomersUncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.RNA-Seq data mining: downregulation of NeuroD6 serves as a possible biomarker for alzheimer's disease brains.Transcriptome analyses identify key cellular factors associated with HIV-1-associated neuropathogenesis in infected men.Brain gene expression patterns differentiate mild cognitive impairment from normal aged and Alzheimer's disease.Regulation of protein levels in subcellular domains through mRNA transport and localized translation.Proteinase-activated receptor-2 up-regulation by Fcgamma-receptor activation in human neutrophilsAstrocyte-neuron lactate transport is required for long-term memory formation.Altered distribution of the EphA4 kinase in hippocampal brain tissue of patients with Alzheimer's disease correlates with pathologyMembrane progesterone receptors (mPRs) mediate progestin induced antimorbidity in breast cancer cells and are expressed in human breast tumors.Concerted perturbation observed in a hub network in Alzheimer's diseaseExercise modifies α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor expression in striatopallidal neurons in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned mouse.Inverse correlation between Alzheimer's disease and cancer: implication for a strong impact of regenerative propensity on neurodegeneration?Profiling axonal mRNA transport.Parallel age-associated changes in brain and plasma neuronal pentraxin receptor levels in a transgenic APP/PS1 rat model of Alzheimer's disease.AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assemblyAmyloid-β(25-35) Modulates the Expression of GirK and KCNQ Channel Genes in the Hippocampus.Preferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex.Network Comparison of Inflammation in Colorectal Cancer and Alzheimer's Disease.Applying the new genomics to alcohol dependence.Proteogenomics of synaptosomal mitochondrial oxidative stress.Functional Maturation of Human Stem Cell-Derived Neurons in Long-Term CulturesEvidence for a neuroprotective microRNA pathway in amnestic mild cognitive impairment.Genome-wide approaches to dissect the roles of RNA binding proteins in translational control: implications for neurological diseasesTranscriptional alterations related to neuropathology and clinical manifestation of Alzheimer's disease.Glutamate system, amyloid ß peptides and tau protein: functional interrelationships and relevance to Alzheimer disease pathologySynaptic gene dysregulation within hippocampal CA1 pyramidal neurons in mild cognitive impairment.Towards the Molecular Foundations of Glutamatergic-targeted Antidepressants.Eph receptors and ephrins in neuron-astrocyte communication at synapses.Gene expression profiling in human neurodegenerative disease.Pathway analysis of the human brain transcriptome in disease.Rare Variants and Transcriptomics in Alzheimer disease.Reversing synapse loss in Alzheimer's disease: Rho-guanosine triphosphatases and insights from other brain disorders.Dissecting Complex and Multifactorial Nature of Alzheimer's Disease Pathogenesis: a Clinical, Genomic, and Systems Biology Perspective.EphA4 is Involved in Sleep Regulation but Not in the Electrophysiological Response to Sleep Deprivation.DENN/MADD/IG20 alternative splicing changes and cell death in Alzheimer's disease.Integrating the Alzheimer's disease proteome and transcriptome: a comprehensive network model of a complex disease.Transcriptional signatures mediated by acetylation overlap with early-stage Alzheimer's disease.
P2860
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P2860
Transcriptome analysis of synaptoneurosomes identifies neuroplasticity genes overexpressed in incipient Alzheimer's disease.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@ast
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@en
type
label
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@ast
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@en
prefLabel
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@ast
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@en
P2093
P2860
P921
P1433
P1476
Transcriptome analysis of syna ...... incipient Alzheimer's disease.
@en
P2093
Bryan Spann
Carol A Miller
Carol McCleary
Celia Williams
Ruty Mehrian Shai
Traci Sitzer
Ya-Hsuan Hsu
Yongchun Wu
P2860
P356
10.1371/JOURNAL.PONE.0004936
P407
P577
2009-03-19T00:00:00Z