Targeting and function of the mitochondrial fission factor GDAP1 are dependent on its tail-anchor.
about
Charcot-Marie-Tooth disease and intracellular trafficLack of GDAP1 induces neuronal calcium and mitochondrial defects in a knockout mouse model of charcot-marie-tooth neuropathyA locus-specific database for mutations in GDAP1 allows analysis of genotype-phenotype correlations in Charcot-Marie-Tooth diseases type 4A and 2K.Development of a prediction system for tail-anchored proteinsCharcot-Marie-Tooth disease-associated mutants of GDAP1 dissociate its roles in peroxisomal and mitochondrial fission.Glutathione-conjugating and membrane-remodeling activity of GDAP1 relies on amphipathic C-terminal domainThe Gdap1 knockout mouse mechanistically links redox control to Charcot-Marie-Tooth disease.Intermediate Charcot-Marie-Tooth disease.CMT-linked loss-of-function mutations in GDAP1 impair store-operated Ca2+ entry-stimulated respiration.Regulation of Mitochondrial Dynamics by Proteolytic Processing and Protein Turnover.Clinical and mutational spectrum of Japanese patients with Charcot-Marie-Tooth disease caused by GDAP1 variants.
P2860
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P2860
Targeting and function of the mitochondrial fission factor GDAP1 are dependent on its tail-anchor.
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
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2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
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name
Targeting and function of the ...... dependent on its tail-anchor.
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Targeting and function of the ...... dependent on its tail-anchor.
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Targeting and function of the ...... dependent on its tail-anchor.
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Targeting and function of the ...... dependent on its tail-anchor.
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P2093
P2860
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Targeting and function of the ...... dependent on its tail-anchor.
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Konstanze M Wagner
Marcel Rüegg
Ueli Suter
P2860
P356
10.1371/JOURNAL.PONE.0005160
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P50
P577
2009-04-02T00:00:00Z