about
Pathogenesis of Lassa feverCap binding and immune evasion revealed by Lassa nucleoprotein structureInhibition of Innate Immune Responses Is Key to Pathogenesis by ArenavirusesEarly and strong immune responses are associated with control of viral replication and recovery in lassa virus-infected cynomolgus monkeys.Effective vaccine for lassa fever.Lassa fever: epidemiology, clinical features, and social consequences.Comparative analysis of disease pathogenesis and molecular mechanisms of New World and Old World arenavirus infections.Pathogenesis of arenavirus hemorrhagic fevers.Transcriptome analysis of human peripheral blood mononuclear cells exposed to Lassa virus and to the attenuated Mopeia/Lassa reassortant 29 (ML29), a vaccine candidatePathogenesis of Lassa fever in cynomolgus macaques.Human dendritic cells infected with the nonpathogenic Mopeia virus induce stronger T-cell responses than those infected with Lassa virus.The role of platelets in the pathogenesis of viral hemorrhagic feversGenomic profiling of host responses to Lassa virus: therapeutic potential from primate to manIdentification of cell surface molecules involved in dystroglycan-independent Lassa virus cell entryLassa virus.Chimeric Mice with Competent Hematopoietic Immunity Reproduce Key Features of Severe Lassa Fever.Hemorrhagic fever occurs after intravenous, but not after intragastric, inoculation of rhesus macaques with lymphocytic choriomeningitis virus.Systems Pharmacology Uncovers the Multiple Mechanisms of Xijiao Dihuang Decoction for the Treatment of Viral Hemorrhagic FeverInnate immune response to arenaviral infection: a focus on the highly pathogenic New World hemorrhagic arenaviruses.Lymphocytic choriomeningitis virus (LCMV) infection of macaques: a model for Lassa fever.Immune responses and Lassa virus infection.Early blood profiles of virus infection in a monkey model for Lassa fever.Identification of virulence determinants within the L genomic segment of the pichinde arenavirus.Gene expression in primate liver during viral hemorrhagic fever.A plasma inhibitor of platelet aggregation in patients with Lassa fever.Reverse genetics generation of chimeric infectious Junin/Lassa virus is dependent on interaction of homologous glycoprotein stable signal peptide and G2 cytoplasmic domains.Metabolomics analyses identify platelet activating factors and heme breakdown products as Lassa fever biomarkers.Axl can serve as entry factor for Lassa virus depending on the functional glycosylation of dystroglycan.Lassa fever: another threat from West Africa.
P2860
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P2860
description
1988 nî lūn-bûn
@nan
1988 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Hematologic dysfunction in Lassa fever.
@ast
Hematologic dysfunction in Lassa fever.
@en
type
label
Hematologic dysfunction in Lassa fever.
@ast
Hematologic dysfunction in Lassa fever.
@en
prefLabel
Hematologic dysfunction in Lassa fever.
@ast
Hematologic dysfunction in Lassa fever.
@en
P2093
P2860
P356
P1476
Hematologic dysfunction in Lassa fever.
@en
P2093
Fisher-Hoch S
McCormick JB
P2860
P304
P356
10.1002/JMV.1890260204
P577
1988-10-01T00:00:00Z