Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism. - Test2 - elhamkhani - TriplyDB

Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism.

Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism.

http://www.wikidata.org/entity/Q33454003

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Epithelial cell pro-inflammatory cytokine response differs across dental plaque bacterial species.The cysteine protease α-clostripain is not essential for the pathogenesis of Clostridium perfringens-mediated myonecrosis.Dichotomy of gingipains action as virulence factors: from cleaving substrates with the precision of a surgeon's knife to a meat chopper-like brutal degradation of proteins.The association between rheumatoid arthritis and periodontal disease Transcriptional profiling of bone marrow stromal cells in response to Porphyromonas gingivalis secreted products Porphyromonas gingivalis virulence in a Drosophila melanogaster model TLR2 promoter hypermethylation creates innate immune dysbiosis.Role of vimA in cell surface biogenesis in Porphyromonas gingivalis Porphyromonas gingivalis HmuY stimulates expression of Bcl-2 and Fas by human CD3+ T cells.P. gingivalis modulates keratinocytes through FOXO transcription factors.Identification of interspecies interactions affecting Porphyromonas gingivalis virulence phenotypes Do oral bacteria alter the regenerative potential of stem cells? A concise review.Porphyromonas gingivalis Fim-A genotype distribution among Colombians.Bacterial invasion of epithelial cells and spreading in periodontal tissue.Oral application of a periodontal pathogen impacts SerpinE1 expression and pancreatic islet architecture in prediabetes.Suppression of inflammatory gene expression in T cells by Porphyromonas gingivalis is mediated by targeting MAPK signaling.Gingipain-dependent degradation of mammalian target of rapamycin pathway proteins by the periodontal pathogen Porphyromonas gingivalis during invasion.Toll-like receptor 9 mediates oral bacteria-induced IL-8 expression in gingival epithelial cells.Porphyromonas gingivalis Differentially Modulates Cell Death Profile in Ox-LDL and TNF-α Pre-Treated Endothelial Cells.NOX1/2 activation in human gingival fibroblasts by Fusobacterium nucleatum facilitates attachment of Porphyromonas gingivalis.The Porphyromonas gingivalis O antigen is required for inhibition of apoptosis in gingival epithelial cells following bacterial infection.Endogenous acid ceramidase protects epithelial cells from Porphyromonas gingivalis-induced inflammation in vitro.A novel peptidic inhibitor derived from Streptococcus cristatus ArcA attenuates virulence potential of Porphyromonas gingivalis.Signal-Regulated Protein Kinases/Protein Kinase B-p53-BH3-Interacting Domain Death Agonist Pathway Regulates Gingipain-Induced Apoptosis in Osteoblasts.The thermolysin-like metalloproteinase and virulence factor LasB from pathogenic Pseudomonas aeruginosa induces anoikis of human vascular cells.Filifactor alocis collagenase can modulate apoptosis of normal oral keratinocytes.Interleukin-8 induces DNA synthesis, migration and down-regulation of cleaved caspase-3 in cultured human gingival epithelial cells.Porphyromonas gingivalis influences actin degradation within epithelial cells during invasion and apoptosis.Porphyromonas gingivalis and its lipopolysaccharide differentially regulate the expression of cathepsin B in endothelial cells.Apoptosis and Its Significance in Oral Diseases: An Update

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P2860

Porphyromonas gingivalis induce apoptosis in human gingival epithelial cells through a gingipain-dependent mechanism.

http://www.wikidata.org/entity/Q33454003

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2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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Porphyromonas gingivalis induc ...... gingipain-dependent mechanism.

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Porphyromonas gingivalis induc ...... gingipain-dependent mechanism.

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Porphyromonas gingivalis induc ...... gingipain-dependent mechanism.

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Porphyromonas gingivalis induc ...... gingipain-dependent mechanism.

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Carlos A Garcia

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Denis F Kinane

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Johnah C Galicia

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Manjunatha R Benakanakere

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Panagiota G Stathopoulou

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P2860

Intrinsic apoptotic pathways of gingival epithelial cells modulated by Porphyromonas gingivalis

Osteoprotegerin protects endothelial cells against apoptotic cell death induced by Porphyromonas gingivalis cysteine proteinases.

Gingipains from Porphyromonas gingivalis W83 induce cell adhesion molecule cleavage and apoptosis in endothelial cells.

In situ detection of apoptosis at sites of chronic bacterially induced inflammation in human gingiva

Lysine- and arginine-specific proteinases from Porphyromonas gingivalis. Isolation, characterization, and evidence for the existence of complexes with hemagglutinins.

Temporal activation of anti- and pro-apoptotic factors in human gingival fibroblasts infected with the periodontal pathogen, Porphyromonas gingivalis: potential role of bacterial proteases in host signalling.

Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalis

Gingipains from Porphyromonas gingivalis W83 synergistically disrupt endothelial cell adhesion and can induce caspase-independent apoptosis.

Porphyromonas gingivalis invasion of gingival epithelial cells.

Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy.

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