Exocrine-to-endocrine differentiation is detectable only prior to birth in the uninjured mouse pancreas. - Test2 - elhamkhani - TriplyDB

Exocrine-to-endocrine differentiation is detectable only prior to birth in the uninjured mouse pancreas.

Exocrine-to-endocrine differentiation is detectable only prior to birth in the uninjured mouse pancreas.

http://www.wikidata.org/entity/Q33551040

about

β-cell replacement sources for type 1 diabetes: a focus on pancreatic ductal cells β-Cell regeneration through the transdifferentiation of pancreatic cells: Pancreatic progenitor cells in the pancreas Pancreatic stem cells remain unresolved Pancreas-specific Cre driver lines and considerations for their prudent use Maintenance of β-cell maturity and plasticity in the adult pancreas: developmental biology concepts in adult physiology MUC1-ARF-A Novel MUC1 Protein That Resides in the Nucleus and Is Expressed by Alternate Reading Frame Translation of MUC1 mRNA.Artificial three-dimensional niches deconstruct pancreas development in vitro.De Novo Prediction of Stem Cell Identity using Single-Cell Transcriptome Data Distinct requirements for beta-catenin in pancreatic epithelial growth and patterning.Transient cytokine treatment induces acinar cell reprogramming and regenerates functional beta cell mass in diabetic mice Stem cell approaches for the treatment of type 1 diabetes mellitus.Tamoxifen-Induced Cre-loxP Recombination Is Prolonged in Pancreatic Islets of Adult Mice Pancreatic beta-cells: from generation to regeneration.Stem cell-based strategies for the treatment of type 1 diabetes mellitus Historical perspective: beginnings of the beta-cell: current perspectives in beta-cell development Sox9: a master regulator of the pancreatic program.Regeneration and repair of the exocrine pancreas.Progenitor cell domains in the developing and adult pancreas.β-Cell Generation: Can Rodent Studies Be Translated to Humans?Beta-cell growth and regeneration: replication is only part of the story.Compensatory Response by Late Embryonic Tubular Epithelium to the Reduction in Pancreatic Progenitors.Developmental programming of neonatal pancreatic β-cells by a maternal low-protein diet in rats involves a switch from proliferation to differentiation.β-catenin is selectively required for the expansion and regeneration of mature pancreatic acinar cells in mice.Centroacinar Cells Are Progenitors That Contribute to Endocrine Pancreas Regeneration.Salivary Glands: Stem Cells, Self-duplication, or Both?Growth factors and medium hyperglycemia induce Sox9+ ductal cell differentiation into β cells in mice with reversal of diabetes Cells with surface expression of CD133highCD71low are enriched for tripotent colony-forming progenitor cells in the adult murine pancreas.ALDH1B1 is a potential stem/progenitor marker for multiple pancreas progenitor pools.Colony-forming cells in the adult mouse pancreas are expandable in Matrigel and form endocrine/acinar colonies in laminin hydrogel Direct Reprogramming for Pancreatic Beta-Cells Using Key Developmental Genes Anonymous sources: where do adult β cells come from?β-Cells are not generated in pancreatic duct ligation-induced injury in adult mice.Autonomous interconversion between adult pancreatic α-cells and β-cells after differential metabolic challenges.Islet neogenesis: a possible pathway for beta-cell replenishment.Pancreatic duct ligation after almost complete β-cell loss: exocrine regeneration but no evidence of β-cell regeneration Stem Cells in the Treatment of Insulin-Dependent Diabetes Mellitus.Pathogenesis of pancreatic cancer: lessons from animal models.The quest for tissue stem cells in the pancreas and other organs, and their application in beta-cell replacement Stem cell-based approaches for the treatment of diabetes.Potential pathways to restore β-cell mass: pluripotent stem cells, reprogramming, and endogenous regeneration.

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Exocrine-to-endocrine differentiation is detectable only prior to birth in the uninjured mouse pancreas.

http://www.wikidata.org/entity/Q33551040

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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Exocrine-to-endocrine differen ...... the uninjured mouse pancreas.

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BMC Developmental Biology

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Exocrine-to-endocrine differen ...... n the uninjured mouse pancreas

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L Charles Murtaugh

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P2860

neurogenin3 is required for the development of the four endocrine cell lineages of the pancreas

Cre reporter strains produced by targeted insertion of EYFP and ECFP into the ROSA26 locus

Generalized lacZ expression with the ROSA26 Cre reporter strain

Loss of Cdk4 expression causes insulin-deficient diabetes and Cdk4 activation results in beta-islet cell hyperplasia

Transgenic MUC1 interacts with epidermal growth factor receptor and correlates with mitogen-activated protein kinase activation in the mouse mammary gland

Perinatal survivin is essential for the establishment of pancreatic beta cell mass in mice

Transgenic knockouts reveal a critical requirement for pancreatic beta cell glucokinase in maintaining glucose homeostasis

The transcription factor hepatocyte nuclear factor-6 controls the development of pancreatic ducts in the mouse

Orpk mouse model of polycystic kidney disease reveals essential role of primary cilia in pancreatic tissue organization

Beta-catenin is essential for pancreatic acinar but not islet development

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