Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes. - Test2 - elhamkhani - TriplyDB

Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.

Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.

http://www.wikidata.org/entity/Q33556077

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GABAergic interneuron origin of schizophrenia pathophysiology.Clinical experience and laboratory investigations in patients with anti-NMDAR encephalitis GABAB-mediated rescue of altered excitatory-inhibitory balance, gamma synchrony and behavioral deficits following constitutive NMDAR-hypofunction.Neurophysiological and computational principles of cortical rhythms in cognition Prefrontal Cortex and Social Cognition in Mouse and Man Autoantibodies to central nervous system neuronal surface antigens: psychiatric symptoms and psychopharmacological implications Auditory dysfunction in schizophrenia: integrating clinical and basic features.Synchrony in schizophrenia: a window into circuit-level pathophysiology Clinical and experimental studies of potentially pathogenic brain-directed autoantibodies: current knowledge and future directions Stress-induced deficits in cognition and emotionality: a role of glutamate Synapse-specific contributions in the cortical pathology of schizophrenia Synaptic plasticity, neural circuits, and the emerging role of altered short-term information processing in schizophrenia.Cerebral energy metabolism and the brain's functional network architecture: an integrative review Impact of ketamine on neuronal network dynamics: translational modeling of schizophrenia-relevant deficits.Following the genes: a framework for animal modeling of psychiatric disorders Insights into cortical oscillations arising from optogenetic studies Transcriptional regulation of GAD1 GABA synthesis gene in the prefrontal cortex of subjects with schizophrenia Windows to the soul: vision science as a tool for studying biological mechanisms of information processing deficits in schizophrenia Ion channels as drug targets in central nervous system disorders The role of glutamatergic inputs onto parvalbumin-positive interneurons: relevance for schizophrenia Dissecting the phenotypes of Dravet syndrome by gene deletion Gamma rhythms link prefrontal interneuron dysfunction with cognitive inflexibility in Dlx5/6(+/-) mice.Collagen-derived matricryptins promote inhibitory nerve terminal formation in the developing neocortex.Glutamate Decarboxylase 67 Deficiency in a Subset of GABAergic Neurons Induces Schizophrenia-Related Phenotypes Altered Corticostriatal Connectivity and Exploration/Exploitation Imbalance Emerge as Intermediate Phenotypes for a Neonatal Dopamine Dysfunction.Spine pruning drives antipsychotic-sensitive locomotion via circuit control of striatal dopamine.Tiagabine improves hippocampal long-term depression in rat pups subjected to prenatal inflammation Neuromagnetic oscillations predict evoked-response latency delays and core language deficits in autism spectrum disorders.Inhibition of parvalbumin-expressing interneurons results in complex behavioral changes.Knockout of NMDA-receptors from parvalbumin interneurons sensitizes to schizophrenia-related deficits induced by MK-801 Duration of culture and sonic hedgehog signaling differentially specify PV versus SST cortical interneuron fates from embryonic stem cells Excitation-inhibition discoordination in rodent models of mental disorders.REM sleep and its Loss-Associated Epigenetic Regulation with Reference to Noradrenaline in Particular The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophrenia Common mechanisms of excitatory and inhibitory imbalance in schizophrenia and autism spectrum disorders Xanthurenic Acid Activates mGlu2/3 Metabotropic Glutamate Receptors and is a Potential Trait Marker for Schizophrenia Disease-relevant autoantibodies in first episode schizophrenia Npas4: a neuronal transcription factor with a key role in social and cognitive functions relevant to developmental disorders.Overexpression of Reelin prevents the manifestation of behavioral phenotypes related to schizophrenia and bipolar disorder.Neocortical excitation/inhibition balance in information processing and social dysfunction

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P2860

Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.

http://www.wikidata.org/entity/Q33556077

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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type

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Nature Neuroscience

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Postnatal NMDA receptor ablati ...... schizophrenia-like phenotypes.

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Elizabeth M Quinlan

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Elyse R Sklar

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Gu Yu

http://www.w3.org/2001/XMLSchema#string

Juan E Belforte

http://www.w3.org/2001/XMLSchema#string

Veronika Zsiros

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Zhihong Jiang

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P2860

Hyperactivity and hyperconnectivity of the default network in schizophrenia and in first-degree relatives of persons with schizophrenia

Human studies of prepulse inhibition of startle: normal subjects, patient groups, and pharmacological studies

The essential role of hippocampal CA1 NMDA receptor-dependent synaptic plasticity in spatial memory

Disrupted motor learning and long-term synaptic plasticity in mice lacking NMDAR1 in the striatum

Cortical inhibitory neurons and schizophrenia

Implications of normal brain development for the pathogenesis of schizophrenia

Subanesthetic effects of the noncompetitive NMDA antagonist, ketamine, in humans. Psychotomimetic, perceptual, cognitive, and neuroendocrine responses

Environmental stress and psychiatric illness.

GABAergic interneurons: implications for understanding schizophrenia and bipolar disorder.

Genetic animal models: focus on schizophrenia.

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10.1038/NN.2447

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1

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13

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Kazutoshi Nakazawa

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2009-11-15T00:00:00Z

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38091481

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19915563

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