Rescue of cells from ras oncogene-induced growth arrest by a second, complementing, oncogene.
about
Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signalingAnalysis of RAS oncogene mutations in human lymphoid malignanciesAstrocyte elevated gene-1: recent insights into a novel gene involved in tumor progression, metastasis and neurodegenerationGrowth-inhibitory activity and downregulation of the class II tumor-suppressor gene H-rev107 in tumor cell lines and experimental tumorsMyc signaling via the ARF tumor suppressor regulates p53-dependent apoptosis and immortalizationCrk and CrkL are required for cell transformation by v-fos and v-ras.Transient analysis for antiproliferative gene activity.Gene expression profiling of fibroblasts resistant toward oncogene-mediated transformation reveals preferential transcription of negative growth regulators.p53 mutation and mitotic infidelity.Activation of the cyclin D1 gene by the E1A-associated protein p300 through AP-1 inhibits cellular apoptosis.Enumeration of the simian virus 40 early region elements necessary for human cell transformationRAS Interaction with PI3K: More Than Just Another Effector PathwayOncogene activation in human benign tumors of the skin (keratoacanthomas): is HRAS involved in differentiation as well as proliferation?Pleiotrophin mediates hematopoietic regeneration via activation of RAS.Signal transduction mediated by the Ras/Raf/MEK/ERK pathway from cytokine receptors to transcription factors: potential targeting for therapeutic intervention.JAK/STAT, Raf/MEK/ERK, PI3K/Akt and BCR-ABL in cell cycle progression and leukemogenesis.Design and Characterization of Bioengineered Cancer-Like Stem Cells.Cellular aging is a critical determinant of primary cell resistance to v-src transformation.Cellular senescence and chromatin organisation.Transformation of a continuous rat embryo fibroblast cell line requires three separate domains of simian virus 40 large T antigen.Cell lines established by a temperature-sensitive simian virus 40 large-T-antigen gene are growth restricted at the nonpermissive temperatureMutant p53 tumor suppressor alleles release ras-induced cell cycle growth arrest.Mechanisms of oncogenic cooperation in cancer initiation and metastasis.Simian virus 40 large tumor antigen alone or two cooperating oncogenes convert REF52 cells to a state permissive for gene amplification.Activated Raf-1 causes growth arrest in human small cell lung cancer cellsEfficient immortalization of luminal epithelial cells from human mammary gland by introduction of simian virus 40 large tumor antigen with a recombinant retrovirusUpregulation of miR-27a contributes to the malignant transformation of human bronchial epithelial cells induced by SV40 small T antigen.E7 abolishes raf-induced arrest via mislocalization of p21(Cip1).Synergistic response to oncogenic mutations defines gene class critical to cancer phenotype.Primary rat cells expressing a hybrid polyomavirus-simian virus 40 large T antigen have altered growth properties.Tumor suppressor p53 restricts Ras stimulation of RhoA and cancer cell motility.Simian virus 40 large T antigen's association with the CUL7 SCF complex contributes to cellular transformation.Conditional immortalization of human thyroid epithelial cells: a tool for analysis of oncogene action.Cyclin D2 and Ha-Ras transformed rat embryo fibroblasts exhibit a novel deregulation of cell size control and early S phase arrest in low serum.Epithelial carcinogenesis in the mouse: correlating the genetics and the biologySuppression of anchorage-independent growth of human cancer cell lines by the drs gene.P53-dependent effects of RAS oncogene on chromosome stability and cell cycle checkpoints.Elevated expression of the CD4 receptor and cell cycle arrest are induced in Jurkat cells by treatment with the novel cyclic dinucleotide 3',5'-cyclic diguanylic acid.The molecular pathophysiology of myeloid leukaemias: Ras revisited.Molecular Simulations of Solved Co-crystallized X-Ray Structures Identify Action Mechanisms of PDEδ Inhibitors.
P2860
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P2860
Rescue of cells from ras oncogene-induced growth arrest by a second, complementing, oncogene.
description
1988 nî lūn-bûn
@nan
1988 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի մարտին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@ast
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@en
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@nl
type
label
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@ast
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@en
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@nl
prefLabel
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@ast
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@en
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@nl
P2860
P356
P1476
Rescue of cells from ras oncog ...... cond, complementing, oncogene.
@en
P2093
P2860
P304
P356
10.1073/PNAS.85.5.1519
P407
P577
1988-03-01T00:00:00Z