GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy. - Test2 - elhamkhani - TriplyDB

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

http://www.wikidata.org/entity/Q33575159

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Treatment paradigms for cataplexy in narcolepsy: past, present, and future Unmet needs of patients with narcolepsy: perspectives on emerging treatment options Conditional ablation of orexin/hypocretin neurons: a new mouse model for the study of narcolepsy and orexin system function.Baclofen for narcolepsy with cataplexy: two cases.An automated sleep-state classification algorithm for quantifying sleep timing and sleep-dependent dynamics of electroencephalographic and cerebral metabolic parameters New developments in the management of narcolepsy.A critical evaluation of the gamma-hydroxybutyrate (GHB) model of absence seizures.Mapping the Hypocretin/Orexin Neuronal System: An Unexpectedly Productive Journey.Investigational therapies for the treatment of narcolepsy.Challenges in the development of therapeutics for narcolepsy.GABAergic Neurons of the Central Amygdala Promote Cataplexy.Current and Future Treatment Options for Narcolepsy: A Review.GABA Receptors on Orexin and Melanin-Concentrating Hormone Neurons Are Differentially Homeostatically Regulated Following Sleep Deprivation.Nonpeptide orexin type-2 receptor agonist ameliorates narcolepsy-cataplexy symptoms in mouse models.The effect of hydro-alcoholic of Phoenix Dactylifera extract on sleep and EEG in rat.GABA Receptors and the Pharmacology of Sleep.GHB for cataplexy: Possible mode of action.Trace Amine-Associated Receptor 1 Agonists as Narcolepsy Therapeutics.

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P2860

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

http://www.wikidata.org/entity/Q33575159

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@ast

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@en

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@nl

type

label

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@ast

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@en

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@nl

prefLabel

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@ast

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@en

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@nl

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JNEUROSCI.0080-14.2014

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Journal of Neuroscience

P1476

GABAB agonism promotes sleep and reduces cataplexy in murine narcolepsy.

@en

P2093

Akihiro Yamanaka

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Andrew K Leung

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Jonathan P Wisor

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Sarah Wurts Black

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Stephen R Morairty

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Thomas S Kilduff

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Tsui-Ming Chen

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P2860

Narcolepsy: neural mechanisms of sleepiness and cataplexy

The sleep disorder canine narcolepsy is caused by a mutation in the hypocretin (orexin) receptor 2 gene

A mutation in a case of early onset narcolepsy and a generalized absence of hypocretin peptides in human narcoleptic brains

Narcolepsy and effectiveness of gamma-hydroxybutyrate (GHB): a systematic review and meta-analysis of randomized controlled trials

α4βδ GABA(A) receptors are high-affinity targets for γ-hydroxybutyric acid (GHB)

Narcolepsy in orexin knockout mice: molecular genetics of sleep regulation

Conditional ablation of orexin/hypocretin neurons: a new mouse model for the study of narcolepsy and orexin system function.

Reduced number of hypocretin neurons in human narcolepsy.

Enhancing slow wave sleep with sodium oxybate reduces the behavioral and physiological impact of sleep loss.

Simultaneous stimulation of slow-wave sleep and growth hormone secretion by gamma-hydroxybutyrate in normal young Men.

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6485-6494

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10.1523/JNEUROSCI.0080-14.2014

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