Age-dependent response of CCAAT/enhancer binding proteins following traumatic brain injury in mice
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The Controlled Cortical Impact Model: Applications, Considerations for Researchers, and Future DirectionsEffects of aging on blood brain barrier and matrix metalloproteases following controlled cortical impact in mice.Influence of age on brain edema formation, secondary brain damage and inflammatory response after brain trauma in miceDifferential response of miRNA-21 and its targets after traumatic brain injury in aging mice.CCAAT/enhancer binding protein β expression is increased in the brain during HIV-1-infection and contributes to regulation of astrocyte tissue inhibitor of metalloproteinase-1.Rate of neurodegeneration in the mouse controlled cortical impact model is influenced by impactor tip shape: implications for mechanistic and therapeutic studies.Ablation of CCAAT/Enhancer-Binding Protein Delta (C/EBPD): Increased Plaque Burden in a Murine Alzheimer's Disease ModelCCAAT/enhancer binding protein-δ expression by dendritic cells regulates CNS autoimmune inflammatory disease.Bilateral gene interaction hierarchy analysis of the cell death gene response emphasizes the significance of cell cycle genes following unilateral traumatic brain injuryAge modulates the injury-induced metallomic profile in the brain.Traumatic brain injury in aged animals increases lesion size and chronically alters microglial/macrophage classical and alternative activation statesComplement component 3 (C3) expression in the hippocampus after excitotoxic injury: role of C/EBPβ.The Controlled Cortical Impact Model of Experimental Brain Trauma: Overview, Research Applications, and Protocol.Brain injury results in lower levels of melatonin receptors subtypes MT1 and MT2.Aging of cerebral white matter.The transcription factor CCAAT enhancer-binding protein β protects rat cerebellar granule neurons from apoptosis through its transcription-activating isoforms.Serum amyloid A inhibits dendritic cell differentiation by suppressing GM-CSF receptor expression and signalingTraumatic Brain Injury in the Elderly: Is it as Bad as we Think?CCAAT/Enhancer binding protein β silencing mitigates glial activation and neurodegeneration in a rat model of Parkinson's diseaseMicroRNAs as diagnostic markers and therapeutic targets for traumatic brain injury.Early down regulation of the glial Kir4.1 and GLT-1 expression in pericontusional cortex of the old male mice subjected to traumatic brain injury.Age Dependent Hypothalamic and Pituitary Responses to Novel Environment Stress or Lipopolysaccharide in Rats.C/EBPβ regulates delta-secretase expression and mediates pathogenesis in mouse models of Alzheimer's disease.Transcription factors Tp73, Cebpd, Pax6, and Spi1 rather than DNA methylation regulate chronic transcriptomics changes after experimental traumatic brain injury.
P2860
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P2860
Age-dependent response of CCAAT/enhancer binding proteins following traumatic brain injury in mice
description
2009 nî lūn-bûn
@nan
2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Age-dependent response of CCAA ...... traumatic brain injury in mice
@ast
Age-dependent response of CCAA ...... traumatic brain injury in mice
@en
type
label
Age-dependent response of CCAA ...... traumatic brain injury in mice
@ast
Age-dependent response of CCAA ...... traumatic brain injury in mice
@en
prefLabel
Age-dependent response of CCAA ...... traumatic brain injury in mice
@ast
Age-dependent response of CCAA ...... traumatic brain injury in mice
@en
P2860
P1476
Age-dependent response of CCAA ...... traumatic brain injury in mice
@en
P2093
Nancy E J Berman
Rajat Sandhir
P2860
P304
P356
10.1016/J.NEUINT.2009.10.002
P577
2009-10-13T00:00:00Z