A ventral tegmental CRF-glutamate-dopamine interaction in addiction. - Test2 - elhamkhani - TriplyDB

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

http://www.wikidata.org/entity/Q33643410

about

Stress and VTA synapses: implications for addiction and depression Multiple roles for orexin/hypocretin in addiction Ethanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Diversity of Dopaminergic Neural Circuits in Response to Drug Exposure Augmented cocaine seeking in response to stress or CRF delivered into the ventral tegmental area following long-access self-administration is mediated by CRF receptor type 1 but not CRF receptor type 2 The role of early life stress as a predictor for alcohol and drug dependence Hormones and Neuropeptide Receptor Heteromers in the Ventral Tegmental Area. Targets for the Treatment of Loss of Control of Food Intake and Substance Use Disorders CRF-R2 and the heterosynaptic regulation of VTA glutamate during reinstatement of cocaine seeking.Drugs of abuse and stress impair LTP at inhibitory synapses in the ventral tegmental area.Pharmacologically-induced stress: a cross-species probe for translational research in drug addiction and relapse.Kappa opioid receptor activation potentiates the cocaine-induced increase in evoked dopamine release recorded in vivo in the mouse nucleus accumbens.Poststress block of kappa opioid receptors rescues long-term potentiation of inhibitory synapses and prevents reinstatement of cocaine seeking.Previous ethanol experience enhances synaptic plasticity of NMDA receptors in the ventral tegmental area.Disruption of maternal parenting circuitry by addictive process: rewiring of reward and stress systems.Endocannabinoid influence in drug reinforcement, dependence and addiction-related behaviors.Corticotropin releasing factor signaling in the central amygdala is recruited during binge-like ethanol consumption in C57BL/6J mice Addiction and brain reward and antireward pathways.Area-specific analysis of the distribution of hypothalamic neurons projecting to the rat ventral tegmental area, with special reference to the GABAergic and glutamatergic efferents.CRF antagonism within the ventral tegmental area but not the extended amygdala attenuates the anxiogenic effects of cocaine in rats.Stress-related neuropeptides and addictive behaviors: beyond the usual suspects.Antagonism of GABA-B but not GABA-A receptors in the VTA prevents stress- and intra-VTA CRF-induced reinstatement of extinguished cocaine seeking in rats.New insights on neurobiological mechanisms underlying alcohol addiction.Preclinical evidence implicating corticotropin-releasing factor signaling in ethanol consumption and neuroadaptation.Emerging role for corticotropin releasing factor signaling in the bed nucleus of the stria terminalis at the intersection of stress and reward.Unbalanced neuronal circuits in addiction.Sex differences in the neural mechanisms mediating addiction: a new synthesis and hypothesis Metabotropic glutamate 7 (mGlu7) receptor: a target for medication development for the treatment of cocaine dependence.Neurobiological mechanisms that contribute to stress-related cocaine use.Nicotine modulation of adolescent dopamine receptor signaling and hypothalamic peptide response.Binge ethanol-drinking potentiates corticotropin releasing factor R1 receptor activity in the ventral tegmental area.Insights to drug addiction derived from ultrastructural views of the mesocorticolimbic system.Neurobiological mechanisms underlying relapse to cocaine use: contributions of CRF and noradrenergic systems and regulation by glucocorticoids.CRF₂ receptor-deficiency reduces recognition memory deficits and vulnerability to stress induced by cocaine withdrawal.Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake.Stereological analyses of reward system nuclei in maternally deprived/separated alcohol drinking rats Dopamine D2 receptor desensitization by dopamine or corticotropin releasing factor in ventral tegmental area neurons is associated with increased glutamate release.Epigenetic regulation of nociceptin/orphanin FQ and corticotropin-releasing factor system genes in frustration stress-induced binge-like palatable food consumption.Fos activation of selective afferents to ventral tegmental area during cue-induced reinstatement of cocaine seeking in rats.CRF1 receptor antagonists do not reverse pharmacological disruption of prepulse inhibition in rodents.Estradiol increases choice of cocaine over food in male rats.

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A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

http://www.wikidata.org/entity/Q33643410

description

2009 nî lūn-bûn

@nan

2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

@ast

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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type

label

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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prefLabel

A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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J.BRAINRES.2009.09.101

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A ventral tegmental CRF-glutamate-dopamine interaction in addiction.

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Marisela Morales

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Roy A Wise

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P2860

Displacement of corticotropin releasing factor from its binding protein as a possible treatment for Alzheimer's disease

Corticotropin-releasing factor requires CRF binding protein to potentiate NMDA receptors via CRF receptor 2 in dopamine neurons

Corticotropin releasing factor (CRF) binding protein: a novel regulator of CRF and related peptides

A role for the bed nucleus of the stria terminalis, but not the amygdala, in the effects of corticotropin-releasing factor on stress-induced reinstatement of cocaine seeking

Corticotropin-releasing factor binding protein within the ventral tegmental area is expressed in a subset of dopaminergic neurons

Novelty-evoked elevations of nucleus accumbens dopamine: dependence on impulse flow from the ventral subiculum and glutamatergic neurotransmission in the ventral tegmental area.

Single cocaine exposure in vivo induces long-term potentiation in dopamine neurons.

The binding protein of corticotropin-releasing factor: ligand-binding site and subunit structure

Cocaine experience establishes control of midbrain glutamate and dopamine by corticotropin-releasing factor: a role in stress-induced relapse to drug seeking.

Cloning and characterization of a functionally distinct corticotropin-releasing factor receptor subtype from rat brain.

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