Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients. - Test2 - elhamkhani - TriplyDB

Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.

Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.

http://www.wikidata.org/entity/Q33664124

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Potential Roles of Adropin in Central Nervous System: Review of Current Literature Alzheimer's Disease associated with Psychiatric Comorbidities Lithium and the other mood stabilizers effective in bipolar disorder target the rat brain arachidonic acid cascade Second messenger/signal transduction pathways in major mood disorders: moving from membrane to mechanism of action, part II: bipolar disorder Adverse drug reaction prediction using scores produced by large-scale drug-protein target docking on high-performance computing machines Effect of Bcl-2 rs956572 SNP on regional gray matter volumes and cognitive function in elderly males without dementia.DNA methylation of BDNF as a biomarker of early-life adversity Decreased BDNF and TrkB mRNA expression in multiple cortical areas of patients with schizophrenia and mood disorders Targeting mitochondrially mediated plasticity to develop improved therapeutics for bipolar disorder RETRACTED: Dose-dependent changes in neuroinflammatory and arachidonic acid cascade markers with synaptic marker loss in rat lipopolysaccharide infusion model of neuroinflammation Coordinated gene expression of neuroinflammatory and cell signaling markers in dorsolateral prefrontal cortex during human brain development and aging.State-related alterations of gene expression in bipolar disorder: a systematic review.Upregulated expression of brain enzymatic markers of arachidonic and docosahexaenoic acid metabolism in a rat model of the metabolic syndrome The role of BDNF as a mediator of neuroplasticity in bipolar disorder.The Bcl-2 gene polymorphism rs956572AA increases inositol 1,4,5-trisphosphate receptor-mediated endoplasmic reticulum calcium release in subjects with bipolar disorder.Valproate uncompetitively inhibits arachidonic acid acylation by rat acyl-CoA synthetase 4: relevance to valproate's efficacy against bipolar disorder Transcriptional evidence for the role of chronic venlafaxine treatment in neurotrophic signaling and neuroplasticity including also Glutamatergic [corrected] - and insulin-mediated neuronal processes.Partial rodent genetic models for bipolar disorder.Bax inhibitor 1, a modulator of calcium homeostasis, confers affective resilience.Evidence demonstrating role of microRNAs in the etiopathology of major depression Disturbed choline plasmalogen and phospholipid fatty acid concentrations in Alzheimer's disease prefrontal cortex.Chronic valproate treatment blocks D2-like receptor-mediated brain signaling via arachidonic acid in rats.Lamotrigine blocks NMDA receptor-initiated arachidonic acid signalling in rat brain: implications for its efficacy in bipolar disorder Regulation of rat brain polyunsaturated fatty acid (PUFA) metabolism during graded dietary n-3 PUFA deprivation.Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Genetic association of the EGR2 gene with bipolar disorder in Korea Valnoctamide, a non-teratogenic amide derivative of valproic acid, inhibits arachidonic acid activation in vitro by recombinant acyl-CoA synthetase-4 Epigenetic modifications in frontal cortex from Alzheimer's disease and bipolar disorder patients.Gabapentin's minimal action on markers of rat brain arachidonic acid metabolism agrees with its inefficacy against bipolar disorder.Effects of chronic clozapine administration on markers of arachidonic acid cascade and synaptic integrity in rat brain Chronic lithium feeding reduces upregulated brain arachidonic acid metabolism in HIV-1 transgenic rat.Neuroinflammation and synaptic loss Chronic clozapine reduces rat brain arachidonic acid metabolism by reducing plasma arachidonic acid availability.Bcl-2 rs956572 polymorphism is associated with increased anterior cingulate cortical glutamate in euthymic bipolar I disorder.Altered neuronal markers following treatment with mood stabilizer and antipsychotic drugs indicate an increased likelihood of neurotransmitter release.Propylisopropylacetic acid (PIA), a constitutional isomer of valproic acid, uncompetitively inhibits arachidonic acid acylation by rat acyl-CoA synthetase 4: a potential drug for bipolar disorder Comparing clinical responses and the biomarkers of BDNF and cytokines between subthreshold bipolar disorder and bipolar II disorder Elevated Monoamine Oxidase-A Distribution Volume in Borderline Personality Disorder Is Associated With Severity Across Mood Symptoms, Suicidality, and Cognition Synaptic abnormalities in the infralimbic cortex of a model of congenital depression.Upstream Pathways Controlling Mitochondrial Function in Major Psychosis: A Focus on Bipolar Disorder

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Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.

http://www.wikidata.org/entity/Q33664124

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Altered expression of apoptoti ...... rom bipolar disorder patients.

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Altered expression of apoptoti ...... rom bipolar disorder patients.

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Altered expression of apoptoti ...... rom bipolar disorder patients.

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Altered expression of apoptoti ...... rom bipolar disorder patients.

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Altered expression of apoptoti ...... rom bipolar disorder patients.

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Altered expression of apoptoti ...... rom bipolar disorder patients.

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J.NBD.2009.11.010

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Neurobiology of Disease

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Altered expression of apoptoti ...... rom bipolar disorder patients.

@en

P2093

Hyung-Wook Kim

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Jagadeesh S Rao

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P2860

The genetics of bipolar disorder: genome ‘hot regions,’ genes, new potential candidates and future directions

Bipolar disorder and mechanisms of action of mood stabilizers

A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Disappearance of actin-binding protein, drebrin, from hippocampal synapses in Alzheimer's disease

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

Chronic fluoxetine upregulates activity, protein and mRNA levels of cytosolic phospholipase A2 in rat frontal cortex

Dietary n-3 PUFA deprivation alters expression of enzymes of the arachidonic and docosahexaenoic acid cascades in rat frontal cortex

Increased excitotoxicity and neuroinflammatory markers in postmortem frontal cortex from bipolar disorder patients.

Long term lithium treatment suppresses p53 and Bax expression but increases Bcl-2 expression. A prominent role in neuroprotection against excitotoxicity.

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596-603

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10.1016/J.NBD.2009.11.010

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3

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37

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Stanley I. Rapoport

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2009-11-26T00:00:00Z

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40038671

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19945534

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bipolar disorder

apoptotic process

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2823851

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