A quantitative study of α-synuclein pathology in fifteen cases of dementia associated with Parkinson disease.
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The Progressive BSSG Rat Model of Parkinson's: Recapitulating Multiple Key Features of the Human DiseaseA quantitative study of tau pathology in eleven cases of chronic traumatic encephalopathyHippocampal α-synuclein and interneurons in Parkinson's disease: Data from human and mouse models.Screening for impaired cognitive domains in a large Parkinson's disease population and its application to the diagnostic procedure for Parkinson's disease dementia.Hippocampal proliferation is increased in presymptomatic Parkinson's disease and due to microglia.Calcineurin determines toxic versus beneficial responses to α-synuclein.Microglial phenotypes and toll-like receptor 2 in the substantia nigra and hippocampus of incidental Lewy body disease cases and Parkinson's disease patients.Correlation between decreased CSF α-synuclein and Aβ₁₋₄₂ in Parkinson disease.Intravenous Bone Marrow Stem Cell Grafts Preferentially Migrate to Spleen and Abrogate Chronic Inflammation in Stroke.Oculo-visual changes and clinical considerations affecting older patients with dementia.Failing as doorman and disc jockey at the same time: Amygdalar dysfunction in Parkinson's disease.Synucleinopathies: common features and hippocampal manifestations.Hippocampal α-Synuclein in Dementia with Lewy Bodies Contributes to Memory Impairment and Is Consistent with Spread of Pathology.The Retina in Multiple System Atrophy: Systematic Review and Meta-Analysis.Comparative quantitative study of 'signature' pathological lesions in the hippocampus and adjacent gyri of 12 neurodegenerative disorders.Alpha-synuclein as a pathological link between chronic traumatic brain injury and Parkinson's disease.The BSSG rat model of Parkinson's disease: progressing towards a valid, predictive model of disease.Critical appraisal of pathology transmission in the α-synuclein fibril model of Lewy body disorders.Laminar degeneration of frontal and temporal cortex in Parkinson disease dementia.Differential α-synuclein expression contributes to selective vulnerability of hippocampal neuron subpopulations to fibril-induced toxicity.
P2860
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P2860
A quantitative study of α-synuclein pathology in fifteen cases of dementia associated with Parkinson disease.
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2013 nî lūn-bûn
@nan
2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
A quantitative study of α-synu ...... ciated with Parkinson disease.
@ast
A quantitative study of α-synu ...... ciated with Parkinson disease.
@en
type
label
A quantitative study of α-synu ...... ciated with Parkinson disease.
@ast
A quantitative study of α-synu ...... ciated with Parkinson disease.
@en
prefLabel
A quantitative study of α-synu ...... ciated with Parkinson disease.
@ast
A quantitative study of α-synu ...... ciated with Parkinson disease.
@en
P2093
P2860
P921
P1476
A quantitative study of α-synu ...... ciated with Parkinson disease.
@en
P2093
Kyle M Hurth
Meghan C Campbell
Nigel J Cairns
Paul T Kotzbauer
Richard A Armstrong
Robert E Schmidt
P2860
P2888
P304
P356
10.1007/S00702-013-1084-Z
P577
2013-08-31T00:00:00Z