Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies. - Test2 - elhamkhani - TriplyDB

Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies.

Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies.

http://www.wikidata.org/entity/Q33725645

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A leading role for the immune system in the pathophysiology of preeclampsia Early life nutritional programming of obesity: mother-child cohort studies Placental Origins of Chronic Disease.The therapeutic potential of antioxidants, ER chaperones, NO and H2S donors, and statins for treatment of preeclampsia.Abnormal oxidative stress responses in fibroblasts from preeclampsia infants.Fatty acid oxidation changes and the correlation with oxidative stress in different preeclampsia-like mouse models.LIMK1 regulates human trophoblast invasion/differentiation and is down-regulated in preeclampsia.Early onset pre-eclampsia is associated with altered DNA methylation of cortisol-signalling and steroidogenic genes in the placenta Autophagy induced by HIF1α overexpression supports trophoblast invasion by supplying cellular energy.Preeclampsia: multiple approaches for a multifactorial disease Early detection of maternal risk for preeclampsia MIR-210 modulates mitochondrial respiration in placenta with preeclampsia.Impaired A2A adenosine receptor/nitric oxide/VEGF signaling pathway in fetal endothelium during late- and early-onset preeclampsia.Widespread DNA hypomethylation at gene enhancer regions in placentas associated with early-onset pre-eclampsia.Augmented trophoblast cell death in preeclampsia can proceed via ceramide-mediated necroptosis Adipose tissue and fetal programming.Relationships between vascular oxygen sensing mechanisms and hypertensive disease processes.Towards biomarker-based tests that can facilitate decisions about prevention and management of preeclampsia in low-resource settings.Ursolic acid sensitizes radioresistant NSCLC cells expressing HIF-1α through reducing endogenous GSH and inhibiting HIF-1α.Etiopathogenesis, prediction, and prevention of preeclampsia.Oxygen Sensitivity of Placental Trophoblast Connexins 43 and 46: A Role in Preeclampsia?Hypoxia-induced β-catenin downregulation involves p53-dependent activation of Siah-1.A Single Sphingomyelin Species Promotes Exosomal Release of Endoglin into the Maternal Circulation in Preeclampsia.Maternal preeclampsia and childhood asthma in the offspring.VEGF, PIGF and HIF-1α in placentas of early- and late-onset pre-eclamptic patients.Angiogenic imbalance as a contributor to the pathophysiology of preeclampsia among black African women.Human placental renin-angiotensin system in normotensive and pre-eclamptic pregnancies at high altitude and after acute hypoxia-reoxygenation insult.Elevating VEGF-A and PDGF-BB secretion by salidroside enhances neoangiogenesis in diabetic hind-limb ischemia.Factor inhibiting HIF1-A novel target of SUMOylation in the human placenta.Ceramide-induced BOK promotes mitochondrial fission in preeclampsia.Comparing the expression patterns of placental magnesium/phosphorus-transporting channels between healthy and preeclamptic pregnancies.

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P2860

Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies.

http://www.wikidata.org/entity/Q33725645

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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JOURNAL.PONE.0013288

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Abnormalities in oxygen sensin ...... mpsia as distinct pathologies.

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Andrea Tagliaferro

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Antonella Racano

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Ariel Many

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Francesca Ietta

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Jinxia Wang

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Martin Post

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P2860

The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

FIH-1: a novel protein that interacts with HIF-1alpha and VHL to mediate repression of HIF-1 transcriptional activity

FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor

Binding of the von Hippel-Lindau tumor suppressor protein to Elongin B and C

Hypoxia-induced assembly of prolyl hydroxylase PHD3 into complexes: implications for its activity and susceptibility for degradation by the E3 ligase Siah2

HIF prolyl-hydroxylase 2 is the key oxygen sensor setting low steady-state levels of HIF-1alpha in normoxia

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

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47520946

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