Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing. - Test2 - elhamkhani - TriplyDB

Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing.

Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing.

http://www.wikidata.org/entity/Q33738028

about

The hnRNP family: insights into their role in health and disease Aberrant Splicing of Estrogen Receptor, HER2, and CD44 Genes in Breast Cancer Splicing Regulators and Their Roles in Cancer Biology and Therapy CD44 Acts as a Signaling Platform Controlling Tumor Progression and Metastasis Wrecked regulation of intrinsically disordered proteins in diseases: pathogenicity of deregulated regulators Defective control of pre-messenger RNA splicing in human disease Actin cytoskeletal control during epithelial to mesenchymal transition: focus on the pancreas and intestinal tract The role of splicing factors in deregulation of alternative splicing during oncogenesis and tumor progression The splicing regulators Esrp1 and Esrp2 direct an epithelial splicing program essential for mammalian development The clinical and prognostic correlation of HRNPM and SLC1A5 in pathogenesis and prognosis in epithelial ovarian cancer.Restraint of the G2/M transition by the SR/RRM family mRNA shuttling binding protein SNXAHRB1 in Aspergillus nidulans.Telomere-regulating genes and the telomere interactome in familial cancers.The Genome-Wide Analysis of Carcinoembryonic Antigen Signaling by Colorectal Cancer Cells Using RNA Sequencing.The Alternative Splicing of Cytoplasmic Polyadenylation Element Binding Protein 2 Drives Anoikis Resistance and the Metastasis of Triple Negative Breast Cancer.Muscleblind-like 1 suppresses breast cancer metastatic colonization and stabilizes metastasis suppressor transcripts Methods for Characterization of Alternative RNA Splicing Epigenetic regulation of ZEB1-RAB25/ESRP1 axis plays a critical role in phenylbutyrate treatment-resistant breast cancer.Dysregulation of splicing proteins in head and neck squamous cell carcinoma.A splicing switch from ketohexokinase-C to ketohexokinase-A drives hepatocellular carcinoma formation Determination of a Comprehensive Alternative Splicing Regulatory Network and Combinatorial Regulation by Key Factors during the Epithelial-to-Mesenchymal Transition RNA splicing factors as oncoproteins and tumour suppressors.Heterogeneous nuclear ribonucleoprotein M associates with mTORC2 and regulates muscle differentiation.Alternative splicing of DNA damage response genes and gastrointestinal cancers.Competing endogenous RNA interplay in cancer: mechanism, methodology, and perspectives.Functional roles of alternative splicing factors in human disease.Mutant JAK3 phosphoproteomic profiling predicts synergism between JAK3 inhibitors and MEK/BCL2 inhibitors for the treatment of T-cell acute lymphoblastic leukemia.Direct Regulation of Alternative Splicing by SMAD3 through PCBP1 Is Essential to the Tumor-Promoting Role of TGF-β A self-enforcing CD44s/ZEB1 feedback loop maintains EMT and stemness properties in cancer cells.Splicing-factor alterations in cancers.Overexpression of c-fos promotes cell invasion and migration via CD44 pathway in oral squamous cell carcinoma.Metformin treatment reduces temozolomide resistance of glioblastoma cells.The role of RNA alternative splicing in regulating cancer metabolism.Rbfox Proteins Regulate Splicing as Part of a Large Multiprotein Complex LASR.Akt Signaling Is Sustained by a CD44 Splice Isoform-Mediated Positive Feedback Loop.miR18a and miR19a Recruit Specific Proteins for Splicing in Thyroid Cancer Cells.Revealing the Determinants of Widespread Alternative Splicing Perturbation in Cancer.Dual Roles for Epithelial Splicing Regulatory Proteins 1 (ESRP1) and 2 (ESRP2) in Cancer Progression.Alternative splicing of CNOT7 diversifies CCR4-NOT functions.hnRNPM guides an alternative splicing program in response to inhibition of the PI3K/AKT/mTOR pathway in Ewing sarcoma cells.Oncogenic Signalling through Mechanistic Target of Rapamycin (mTOR): A Driver of Metabolic Transformation and Cancer Progression.

P2860

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P2860

Cell type-restricted activity of hnRNPM promotes breast cancer metastasis via regulating alternative splicing.

http://www.wikidata.org/entity/Q33738028

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cell type-restricted activity ...... gulating alternative splicing.

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Cell type-restricted activity ...... gulating alternative splicing.

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type

label

Cell type-restricted activity ...... gulating alternative splicing.

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Cell type-restricted activity ...... gulating alternative splicing.

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prefLabel

Cell type-restricted activity ...... gulating alternative splicing.

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Cell type-restricted activity ...... gulating alternative splicing.

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Genes & Development

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Cell type-restricted activity ...... gulating alternative splicing.

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Chonghui Cheng

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David Gius

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Haiyan Tan

http://www.w3.org/2001/XMLSchema#string

Huilin Huang

http://www.w3.org/2001/XMLSchema#string

Jaegyoon Ahn

http://www.w3.org/2001/XMLSchema#string

Junmin Peng

http://www.w3.org/2001/XMLSchema#string

Kalliopi P Siziopikou

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Lauren M Reinke

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Yilin Xu

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P2860

Nova, the paraneoplastic Ri antigen, is homologous to an RNA-binding protein and is specifically expressed in the developing motor system

ESRP1 and ESRP2 are epithelial cell-type-specific regulators of FGFR2 splicing

Regulation of CD44 alternative splicing by SRm160 and its potential role in tumor cell invasion

Homologues of the Caenorhabditis elegans Fox-1 protein are neuronal splicing regulators in mammals

Cooperative assembly of an hnRNP complex induced by a tissue-specific homolog of polypyrimidine tract binding protein

The hnRNP F protein: unique primary structure, nucleic acid-binding properties, and subcellular localization

The epithelial-mesenchymal transition generates cells with properties of stem cells

Phenotypic characterization of human colorectal cancer stem cells

Prospective identification of tumorigenic breast cancer cells

An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype

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1191-1203

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scholarly article

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10.1101/GAD.241968.114

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11

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28

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Marcus E. Peter

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2014-05-19T00:00:00Z

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262536557

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24840202

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4052765

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