M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment. - Test2 - elhamkhani - TriplyDB

M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment.

M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment.

http://www.wikidata.org/entity/Q33746669

about

The pathogenicity of the Streptococcus genus Structural Model for Covalent Adhesion of the Streptococcus pyogenes Pilus through a Thioester Bond Unique Footprint in the scl1.3 Locus Affects Adhesion and Biofilm Formation of the Invasive M3-Type Group A Streptococcus Natural variant of collagen-like protein a in serotype M3 group a Streptococcus increases adherence and decreases invasive potential Distinct single amino acid replacements in the control of virulence regulator protein differentially impact streptococcal pathogenesis ETosis: A Microbicidal Mechanism beyond Cell Death.Epithelial microvilli establish an electrostatic barrier to microbial adhesion Streptococcus agalactiae in adults at Chiang Mai University Hospital: a retrospective study.M protein and hyaluronic acid capsule are essential for in vivo selection of covRS mutations characteristic of invasive serotype M1T1 group A Streptococcus.The small regulatory RNA FasX enhances group A Streptococcus virulence and inhibits pilus expression via serotype-specific targets.The FasX Small Regulatory RNA Negatively Regulates the Expression of Two Fibronectin-Binding Proteins in Group A Streptococcus.Characterization of the effect of the histidine kinase CovS on response regulator phosphorylation in group A Streptococcus.Expression of the type 1 pneumococcal pilus is bistable and negatively regulated by the structural component RrgA Rescuing of deficient killing and phagocytic activities of macrophages derived from non-obese diabetic mice by treatment with geldanamycin or heat shock: potential clinical implications.Complex Population Structure and Virulence Differences among Serotype 2 Streptococcus suis Strains Belonging to Sequence Type 28.Activation of PAD4 in NET formation.Application of Whole-Genome Sequencing to an Unusual Outbreak of Invasive Group A Streptococcal Disease.The expanding world of extracellular traps: not only neutrophils but much more Tissue tropisms in group A Streptococcus: what virulence factors distinguish pharyngitis from impetigo strains?Streptococcus pyogenes adhesion and colonization.Increased Pilus Production Conferred by a Naturally Occurring Mutation Alters Host-Pathogen Interaction in Favor of Carriage in Streptococcus pyogenes.Activation of the stress response in macrophages alters the M1/M2 balance by enhancing bacterial killing and IL-10 expression.Mannheimia haemolytica and its leukotoxin cause neutrophil extracellular trap formation by bovine neutrophils Electronic cigarette inhalation alters innate immunity and airway cytokines while increasing the virulence of colonizing bacteria.Galleria mellonella larvae as an infection model for group A streptococcus.The role of pili in Bacillus cereus intraocular infection.The Group A Streptococcus serotype M2 pilus plays a role in host cell adhesion and immune evasion.Statins enhance formation of phagocyte extracellular traps.The ancillary protein 1 of Streptococcus pyogenes FCT-1 pili mediates cell adhesion and biofilm formation through heterophilic as well as homophilic interactions.NET balancing: a problem in inflammatory lung diseases.Identification and characterization of serotype-specific variation in group A Streptococcus pilus expression.Incremental Contributions of FbaA and Other Impetigo-Associated Surface Proteins to Fitness and Virulence of a Classical Group A Streptococcal Skin Strain.In vitro resistance mechanisms of Neisseria meningitidis against neutrophil extracellular traps.

P2860

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P2860

M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment.

http://www.wikidata.org/entity/Q33746669

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

M1T1 group A streptococcal pil ...... phil extracellular entrapment.

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M1T1 group A streptococcal pil ...... phil extracellular entrapment.

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type

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M1T1 group A streptococcal pil ...... phil extracellular entrapment.

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M1T1 group A streptococcal pil ...... phil extracellular entrapment.

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M1T1 group A streptococcal pil ...... phil extracellular entrapment.

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Anjuli M Timmer

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Heather C Maisey

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Maren von Köckritz-Blickwede

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Suzan H M Rooijakkers

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P2860

Group A Streptococcus produce pilus-like structures containing protective antigens and Lancefield T antigens.

A pneumococcal pilus influences virulence and host inflammatory responses

Genome-wide analysis of group a streptococci reveals a mutation that modulates global phenotype and disease specificity.

An IgG-like Domain in the Minor Pilin GBS52 of Streptococcus agalactiae Mediates Lung Epithelial Cell Adhesion

Neutrophil extracellular traps kill bacteria

Pili in gram-positive pathogens

Induction of neutrophil extracellular DNA lattices by placental microparticles and IL-8 and their presence in preeclampsia

Novel cell death program leads to neutrophil extracellular traps

Pathogenesis of group A streptococcal infections

Extracellular deoxyribonuclease made by group A Streptococcus assists pathogenesis by enhancing evasion of the innate immune response.

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Laura E Crotty Alexander

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2009-12-04T00:00:00Z

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40447175

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19960175

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2843839

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