The effects of aging on pulmonary oxidative damage, protein nitration, and extracellular superoxide dismutase down-regulation during systemic inflammation.
about
An update on the LIM and SH3 domain protein 1 (LASP1): a versatile structural, signaling, and biomarker proteinAging-associated oxidative stress inhibits liver progenitor cell activation in mice.Endothelial dysfunction is a potential contributor to multiple organ failure and mortality in aged mice subjected to septic shock: preclinical studies in a murine model of cecal ligation and punctureA new cecal slurry preparation protocol with improved long-term reproducibility for animal models of sepsisAge and sex dimorphisms contribute to the severity of bleomycin-induced lung injury and fibrosis.Advanced age is associated with worsened outcomes and a unique genomic response in severely injured patients with hemorrhagic shock.Mechanisms of dysfunction in senescent pulmonary endothelium.Increased hyperoxia-induced lung injury in nitric oxide synthase 2 null mice is mediated via angiopoietin 2.Superoxide dismutase 3 controls adaptive immune responses and contributes to the inhibition of ovalbumin-induced allergic airway inflammation in mice.Oxidative stress response and Nrf2 signaling in aging.The future of murine sepsis and trauma research models.Aging exacerbates microvascular endothelial damage induced by circulating factors present in the serum of septic patients.Age-dependent vulnerability to experimental acute pancreatitis is associated with increased systemic inflammation and thrombosis.Aged mice are unable to mount an effective myeloid response to sepsis.Sepsis in old age: review of human and animal studies.Acute kidney injury and inflammatory response of sepsis following cecal ligation and puncture in d-galactose-induced aging rats.Protein Interactions at Endothelial Junctions and Signaling Mechanisms Regulating Endothelial Permeability.Transgelin-2: A potential oncogenic factor.Vitamin C and Microvascular Dysfunction in Systemic Inflammation.Human Myeloid-derived Suppressor Cells are Associated With Chronic Immune Suppression After Severe Sepsis/Septic Shock.Late Therapeutic Intervention with Antibiotics and Fluid Resuscitation Allows for a Prolonged Disease Course with High Survival in a Severe Murine Model of Sepsis.Effect of ageing on systemic inflammatory response in acute pancreatitis.Voluntary running exercise protects against sepsis-induced early inflammatory and pro-coagulant responses in aged mice.Protein Posttranslational Modifications: Roles in Aging and Age-Related Disease.Delayed preconditioning prevents ischemia/reperfusion-induced endothelial injury in rats: role of ROS and eNOS.MicroRNAs are dynamically regulated and play an important role in LPS-induced lung injury.Comparative 1D Blue-Native electrophoresis analysis of Plasmodium falciparum and human proteins associated with cytoadherence
P2860
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P2860
The effects of aging on pulmonary oxidative damage, protein nitration, and extracellular superoxide dismutase down-regulation during systemic inflammation.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The effects of aging on pulmon ...... during systemic inflammation.
@ast
The effects of aging on pulmon ...... during systemic inflammation.
@en
type
label
The effects of aging on pulmon ...... during systemic inflammation.
@ast
The effects of aging on pulmon ...... during systemic inflammation.
@en
prefLabel
The effects of aging on pulmon ...... during systemic inflammation.
@ast
The effects of aging on pulmon ...... during systemic inflammation.
@en
P2093
P2860
P1476
The effects of aging on pulmon ...... during systemic inflammation.
@en
P2093
B Mark Evers
Hiroshi Saito
Junji Ueda
Marlene E Starr
Shoji Yamamoto
P2860
P304
P356
10.1016/J.FREERADBIOMED.2010.11.013
P577
2010-11-17T00:00:00Z