Molecular Mechanisms of Neurodegenerative Diseases Induced by Human Retroviruses: A Review. - Test2 - elhamkhani - TriplyDB

Molecular Mechanisms of Neurodegenerative Diseases Induced by Human Retroviruses: A Review.

Molecular Mechanisms of Neurodegenerative Diseases Induced by Human Retroviruses: A Review.

http://www.wikidata.org/entity/Q33753132

about

Human endogenous retroviruses and the nervous system Novel PI3K/Akt inhibitors screened by the cytoprotective function of human immunodeficiency virus type 1 Tat HIV-1 nef protein structures associated with brain infection and dementia pathogenesis Exosomes in Viral Disease.Crosstalk between HDAC6 and Nox2-based NADPH oxidase mediates HIV-1 Tat-induced pro-inflammatory responses in astrocytes.Variation in the biological properties of HIV-1 R5 envelopes: implications of envelope structure, transmission and pathogenesis.Human immunodeficiency virus-1 (HIV-1)-mediated apoptosis: new therapeutic targets.Intracerebroventricular administration of HIV-1 Tat induces brain cytokine and indoleamine 2,3-dioxygenase expression: a possible mechanism for AIDS comorbid depression.Of mice and monkeys: can animal models be utilized to study neurological consequences of pediatric HIV-1 infection?Molecular and pathologic insights from latent HIV-1 infection in the human brain HIV-1 Tat Regulates Occludin and Aβ Transfer Receptor Expression in Brain Endothelial Cells via Rho/ROCK Signaling Pathway Strategies to Block HIV Transcription: Focus on Small Molecule Tat Inhibitors.Are human polyomaviruses co-factors for cancers induced by other oncoviruses?Tax posttranslational modifications and interaction with calreticulin in MT-2 cells and human peripheral blood mononuclear cells of human T cell lymphotropic virus type-I-associated myelopathy/tropical spastic paraparesis patients.HIV-1 Tat Induces Unfolded Protein Response and Endoplasmic Reticulum Stress in Astrocytes and Causes Neurotoxicity through Glial Fibrillary Acidic Protein (GFAP) Activation and Aggregation.Evaluation of T Regulatory Lymphocytes Transcription Factors in HTLV-1-Associated Myelopathy/Tropical Spastic Paraparesis (HAM/TSP) Patients.Tax secretion from peripheral blood mononuclear cells and Tax detection in plasma of patients with human T-lymphotropic virus-type 1-associated myelopathy/tropical spastic paraparesis and asymptomatic carriers.The human immunodeficiency virus coat protein gp120 promotes forward trafficking and surface clustering of NMDA receptors in membrane microdomains.5,7-Dihydroxy-6-Methoxy-Flavonoids Eliminate HIV-1 D3-transfected Cytoprotective Macrophages by Inhibiting the PI3K/Akt Signaling Pathway.HDAC6 mediates HIV-1 tat-induced proinflammatory responses by regulating MAPK-NF-kappaB/AP-1 pathways in astrocytes.15-deoxy-Δ¹²,¹⁴ -prostaglandin J₂ inhibits human immunodeficiency virus-1 tat-induced monocyte chemoattractant protein-1/CCL2 production by blocking the extracellular signal-regulated kinase-1/2 signaling pathway independently of peroxisome prolifer CNS Infections

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Molecular Mechanisms of Neurodegenerative Diseases Induced by Human Retroviruses: A Review.

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American Journal of Infectious Diseases

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Molecular Mechanisms of Neurod ...... Human Retroviruses: A Review.

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Bryan P Irish

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Roles of CD4 and coreceptors in binding, endocytosis, and proteolysis of gp120 envelope glycoproteins derived from human immunodeficiency virus type 1

Chemokine receptor expression and signaling in macaque and human fetal neurons and astrocytes: implications for the neuropathogenesis of AIDS

Endocytosis of major histocompatibility complex class I molecules is induced by the HIV–1 Nef protein

Chemokine and chemokine-receptor expression in human glial elements: induction by the HIV protein, Tat, and chemokine autoregulation

Inhibition of CXCR4-dependent HIV-1 infection by extracellular HIV-1 Tat

HIV-1 gp120- and gp160-induced apoptosis in cultured endothelial cells is mediated by caspases

Mechanism for down-regulation of CD28 by Nef.

MCP-1 (CCL2) protects human neurons and astrocytes from NMDA or HIV-tat-induced apoptosis

Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

Role of flanking E box motifs in human immunodeficiency virus type 1 TATA element function

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