Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation
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Effect of Soluble Epoxide Hydrolase on the Modulation of Coronary Reactive Hyperemia: Role of Oxylipins and PPARγDeletion of soluble epoxide hydrolase enhances coronary reactive hyperemia in isolated mouse heart: role of oxylipins and PPARγVascular Endothelial Over-Expression of Human Soluble Epoxide Hydrolase (Tie2-sEH Tr) Attenuates Coronary Reactive Hyperemia in Mice: Role of Oxylipins and ω-HydroxylasesHeart of the matter: coronary dysfunction in metabolic syndrome.Contribution of electromechanical coupling between Kv and Ca v1.2 channels to coronary dysfunction in obesity.Metabolic syndrome and coronary artery disease in Ossabaw compared with Yucatan swine.Modeling cardiac arrest and resuscitation in the domestic pigCa2+ regulatory mechanisms of exercise protection against coronary artery disease in metabolic syndrome and diabetes.Contribution of voltage-dependent K⁺ channels to metabolic control of coronary blood flow.Impaired coronary collateral growth: miR-shaken neutrophils caught in the act.Large conductance, calcium- and voltage-gated potassium (BK) channels: regulation by cholesterol.Daming capsule restores endothelial dysfunction induced by high-fat dietPenitrem A as a tool for understanding the role of large conductance Ca(2+)/voltage-sensitive K(+) channels in vascular function.Vascular endothelial overexpression of human CYP2J2 (Tie2-CYP2J2 Tr) modulates cardiac oxylipin profiles and enhances coronary reactive hyperemia in mice.Exercise training-enhanced, endothelium-dependent dilation mediated by altered regulation of BK(Ca) channels in collateral-dependent porcine coronary arteriolesA1 adenosine receptor negatively modulates coronary reactive hyperemia via counteracting A2A-mediated H2O2 production and KATP opening in isolated mouse heartsContribution of hydrogen sulfide to the control of coronary blood flow.Role of ion channels in coronary microcirculation: a review of the literature.Sudden cardiac death and obesity.Cardiovascular consequences of metabolic syndrome.Contribution of voltage-dependent K+ and Ca2+ channels to coronary pressure-flow autoregulation.Contribution of adenosine A(2A) and A(2B) receptors to ischemic coronary dilation: role of K(V) and K(ATP) channels.Contribution of IKCa channels to the control of coronary blood flow.Reduced coronary reactive hyperemia in mice was reversed by the soluble epoxide hydrolase inhibitor (t-AUCB): Role of adenosine A2A receptor and plasma oxylipins.Regulation of Coronary Blood Flow.The Coronary Microcirculation in Health and Disease
P2860
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P2860
Metabolic syndrome reduces the contribution of K+ channels to ischemic coronary vasodilation
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@ast
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@en
type
label
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@ast
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@en
prefLabel
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@ast
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@en
P2093
P2860
P1476
Metabolic syndrome reduces the ...... ischemic coronary vasodilation
@en
P2093
Gregory A Payne
Gregory M Dick
Ian N Bratz
Johnathan D Tune
Léna Borbouse
Michael Sturek
Mouhamad Alloosh
Zachary C Berwick
Zachary P Neeb
P2860
P304
P356
10.1152/AJPHEART.00888.2009
P577
2010-01-29T00:00:00Z