Oral mucosal endotoxin tolerance induction in chronic periodontitis. - Test2 - elhamkhani - TriplyDB

Oral mucosal endotoxin tolerance induction in chronic periodontitis.

Oral mucosal endotoxin tolerance induction in chronic periodontitis.

http://www.wikidata.org/entity/Q33797036

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Inflammatory Mediators of Leprosy Reactional Episodes and Dental Infections: A Systematic Review Immune cells link obesity-associated type 2 diabetes and periodontitis Prolonged exposure to bacterial toxins downregulated expression of toll-like receptors in mesenchymal stromal cell-derived osteoprogenitors.The potential of p38 MAPK inhibitors to modulate periodontal infections.Acute inflammation at a mandibular solitary horizontal incompletely impacted molar.Periodontal innate immune mechanisms relevant to atherosclerosis and obesity.Macrophage subset sensitivity to endotoxin tolerisation by Porphyromonas gingivalis Periodontal innate immune mechanisms relevant to atherosclerosis.TLR signaling that induces weak inflammatory response and SHIP1 enhances osteogenic functions.Dendritic cells: microbial clearance via autophagy and potential immunobiological consequences for periodontal disease Effects of Porphyromonas gingivalis LipopolysaccharideTolerized Monocytes on Inflammatory Responses in Neutrophils.Microbial carriage state of peripheral blood dendritic cells (DCs) in chronic periodontitis influences DC differentiation, atherogenic potential.Trigeminal nociceptors express TLR-4 and CD14: a mechanism for pain due to infection.Dendritic cells at the oral mucosal interface.Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss.Toll-like receptors and their role in periodontal health and disease.A novel class of lipoprotein lipase-sensitive molecules mediates Toll-like receptor 2 activation by Porphyromonas gingivalis.Resistance of MMP9 and TIMP1 to endotoxin tolerance.Human squamous cell carcinomas evade the immune response by down-regulation of vascular E-selectin and recruitment of regulatory T cells Oral mucosal dendritic cells and periodontitis: many sides of the same coin with new twists Porphyromonas gingivalis lipopolysaccharide induces tumor necrosis factor-alpha and interleukin-6 secretion, and CCL25 gene expression, in mouse primary gingival cell lines: interleukin-6-driven activation of CCL2 Rapid myeloid cell transcriptional and proteomic responses to periodontopathogenic Porphyromonas gingivalis Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriae.Toll gates to periodontal host modulation and vaccine therapy Complementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the host.HIV Infection and Compromised Mucosal Immunity: Oral Manifestations and Systemic Inflammation.Members of the Oral Microbiota Are Associated with IL-8 Release by Gingival Epithelial Cells in Healthy Individuals.Macrophage polarization: an opportunity for improved outcomes in biomaterials and regenerative medicine Periodontal innate immune mechanisms relevant to obesity.Bacteria take control of tolls and T cells to destruct jaw bone.The role of adipokines in periodontal infection and healing.Altered Toll-like receptor 2-mediated endotoxin tolerance is related to diminished interferon beta production.The role of Toll-like receptors in periodontitis.Evaluation of TLR2 and 4 in Chronic Periodontitis.The paradox of painless periodontal disease.Signaling mechanism for Aspergillus fumigatus tolerance in corneal fibroblasts induced by LPS pretreatment.Increased nucleic Acid receptor expression in chronic periodontitis.Impaired immune tolerance to Porphyromonas gingivalis lipopolysaccharide promotes neutrophil migration and decreased apoptosis.Targeting of DC-SIGN on human dendritic cells by minor fimbriated Porphyromonas gingivalis strains elicits a distinct effector T cell response.Interleukin-1alpha stimulation in monocytes by periodontal bacteria: antagonistic effects of Porphyromonas gingivalis.

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P2860

Oral mucosal endotoxin tolerance induction in chronic periodontitis.

http://www.wikidata.org/entity/Q33797036

description

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2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2005 թվականի փետրվարին հրատարակված գիտական հոդված

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Oral mucosal endotoxin tolerance induction in chronic periodontitis.

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Oral mucosal endotoxin tolerance induction in chronic periodontitis.

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Oral mucosal endotoxin tolerance induction in chronic periodontitis.

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Oral mucosal endotoxin tolerance induction in chronic periodontitis.

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IAI.73.2.687-694.2005

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Infection and Immunity

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Oral mucosal endotoxin tolerance induction in chronic periodontitis.

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Christopher W Cutler

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Manoj Muthukuru

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Ravi Jotwani

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P2860

Tumor necrosis factor (TNF)-mediated kinase cascades: bifurcation of nuclear factor-kappaB and c-jun N-terminal kinase (JNK/SAPK) pathways at TNF receptor-associated factor 2

Bacterial diversity in human subgingival plaque

Decreased expression of Toll-like receptor-4 and MD-2 correlates with intestinal epithelial cell protection against dysregulated proinflammatory gene expression in response to bacterial lipopolysaccharide

Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

Human intestinal epithelial cells are broadly unresponsive to Toll-like receptor 2-dependent bacterial ligands: implications for host-microbial interactions in the gut.

Multiple dendritic cell (DC) subpopulations in human gingiva and association of mature DCs with CD4+ T-cells in situ.

Clinical benefits of oral irrigation for periodontitis are related to reduction of pro-inflammatory cytokine levels and plaque.

Porphyromonas gingivalis lipopolysaccharide is both agonist and antagonist for p38 mitogen-activated protein kinase activation.

Mucosal immunity: its role in defense and allergy.

Porphyromonas gingivalis lipopolysaccharide antagonizes Escherichia coli lipopolysaccharide at toll-like receptor 4 in human endothelial cells.

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10.1128/IAI.73.2.687-694.2005

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