Critical role of IRF-5 in the development of T helper 1 responses to Leishmania donovani infection.
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The Contribution of Immune Evasive Mechanisms to Parasite Persistence in Visceral LeishmaniasisInterferon-regulatory factors determine macrophage phenotype polarizationLessons from other diseases: granulomatous inflammation in leishmaniasisAn AAAG-Rich Oligodeoxynucleotide Rescues Mice from Bacterial Septic Peritonitis by Interfering Interferon Regulatory Factor 5.Differential Regulation of the Immune Response in the Spleen and Liver of Mice Infected with Leishmania donovani.IRF-5-Mediated Inflammation Limits CD8+ T Cell Expansion by Inducing HIF-1α and Impairing Dendritic Cell Functions during Leishmania InfectionLymph node hypertrophy following Leishmania major infection is dependent on TLR9.The Challenge of Stability in High-Throughput Gene Expression Analysis: Comprehensive Selection and Evaluation of Reference Genes for BALB/c Mice Spleen Samples in the Leishmania infantum Infection Model.Pleiotropic IFN-dependent and -independent effects of IRF5 on the pathogenesis of experimental lupusPhenotype and function of B cells and dendritic cells from interferon regulatory factor 5-deficient mice with and without a mutation in DOCK2.Regulatory actions of Toll-like receptor 2 (TLR2) and TLR4 in Leishmania donovani infection in the liver.Immobilized heavy chain-hyaluronic acid polarizes lipopolysaccharide-activated macrophages toward M2 phenotypeInnate antiviral response: role in HIV-1 infection.Regulation of T helper cell differentiation by interferon regulatory factor family members.Immunopathogenesis of non-healing American cutaneous leishmaniasis and progressive visceral leishmaniasis.Epigenetics and the IRFs: a complex interplay in the control of immunity and autoimmunity.Immunity to visceral leishmaniasis: implications for immunotherapy.Toll-like receptor signaling in parasitic infections.Host resistance to visceral leishmaniasis: prevalence and prevention.HIF-1α is a key regulator in potentiating suppressor activity and limiting the microbicidal capacity of MDSC-like cells during visceral leishmaniasis.Fungal pattern receptors down-regulate the inflammatory response by a cross-inhibitory mechanism independent of interleukin-10 production.IRF5:RelA interaction targets inflammatory genes in macrophages.IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cells.Analysis of the expression of toll-like receptors 2 and 4 and cytokine production during experimental Leishmania chagasi infection.Type I Interferon Signaling Is Required for CpG-Oligodesoxynucleotide-Induced Control of Leishmania major, but Not for Spontaneous Cure of Subcutaneous Primary or Secondary L. major Infection.HIF-1α hampers dendritic cell function and Th1 generation during chronic visceral leishmaniasis.
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Critical role of IRF-5 in the development of T helper 1 responses to Leishmania donovani infection.
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2011 nî lūn-bûn
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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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name
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@ast
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@en
type
label
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@ast
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@en
prefLabel
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@ast
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@en
P2093
P2860
P1433
P1476
Critical role of IRF-5 in the ...... Leishmania donovani infection.
@en
P2093
Andrea Paun
Paula M Pitha
Rashmi Bankoti
Simona Stäger
Trupti Joshi
P2860
P304
P356
10.1371/JOURNAL.PPAT.1001246
P577
2011-01-06T00:00:00Z