Xanthine oxidase inhibition preserves left ventricular systolic but not diastolic function in cardiac volume overload. - Test2 - elhamkhani - TriplyDB

Xanthine oxidase inhibition preserves left ventricular systolic but not diastolic function in cardiac volume overload.

Xanthine oxidase inhibition preserves left ventricular systolic but not diastolic function in cardiac volume overload.

http://www.wikidata.org/entity/Q33816822

about

NSAIDs and Cardiovascular Diseases: Role of Reactive Oxygen Species Cardiac specific expression of threonine 5 to alanine mutant sarcolipin results in structural remodeling and diastolic dysfunction Increased myocardial stiffness due to cardiac titin isoform switching in a mouse model of volume overload limits eccentric remodeling Moderate hypoxia followed by reoxygenation results in blood-brain barrier breakdown via oxidative stress-dependent tight-junction protein disruption.Cardiomyocyte mitochondrial oxidative stress and cytoskeletal breakdown in the heart with a primary volume overload.Decreased Bioenergetic Health Index in monocytes isolated from the pericardial fluid and blood of post-operative cardiac surgery patients.Cardiovascular Mitochondrial Dysfunction Induced by Cocaine: Biomarkers and Possible Beneficial Effects of Modulators of Oxidative Stress.Xanthine oxidase inhibitors in ischaemic heart disease.Exposure to chronic alcohol accelerates development of wall stress and eccentric remodeling in rats with volume overload.Reducing sarcolipin expression mitigates Duchenne muscular dystrophy and associated cardiomyopathy in mice.Febuxostat Modulates MAPK/NF-κBp65/TNF-α Signaling in Cardiac Ischemia-Reperfusion Injury.Desmin loss and mitochondrial damage precede left ventricular systolic failure in volume overload heart failure.A Comparison of Phenomenologic Growth Laws for Myocardial Hypertrophy.

P2860

Q26784322-CEB3D56F-2B76-4623-9C7A-515DD07DC2B9 Q28543394-1E723B87-6545-4F00-AC86-C0D19A5D4B31 Q34995889-44F4D131-D2AC-4408-9A98-5B610DEE06B0 Q35064316-A8A10395-A2D1-4D1F-A95C-02B25761555C Q35178659-2827A378-6995-4ED2-AB7D-FE0BABB7E2FA Q36185222-7808A55B-6B56-4F99-879E-330C27B40524 Q38737300-028553BA-6518-4F5E-9F16-B21222D4F009 Q39324330-7477348D-D52C-4142-BAD1-CB5546A40377 Q39828115-A5506F83-E53B-44C9-919E-7FB1E3A3F849 Q42380533-B7245D62-D3E4-4226-83EE-9DA07E63E45C Q43375817-27A293DC-78BB-4E08-ADC5-211F2BD6C22A Q47902837-949A7334-8DB5-4448-9EB4-8AAD7D5E709D Q52597723-A5531A40-6F87-4508-99F4-785596005B03

P2860

Xanthine oxidase inhibition preserves left ventricular systolic but not diastolic function in cardiac volume overload.

http://www.wikidata.org/entity/Q33816822

description

2013 nî lūn-bûn

@nan

2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@ast

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@en

type

label

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@ast

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@en

prefLabel

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@ast

Xanthine oxidase inhibition pr ...... on in cardiac volume overload.

@en

P1433

Q33816822-26EF510B-79A7-4A35-85C5-A43B8012E806

P1476

Q33816822-62B7B0D5-0A59-4A21-BF8C-4BFB377D8026

P2093

Q33816822-10276652-1B12-496A-8A73-D4A74CDE9F9C

Q33816822-29D395F1-79EE-4B28-AD68-7368C1D87B11

Q33816822-2C1C0F86-8F6A-483A-BC2B-F8EBCC92D02F

Q33816822-60F23444-7F85-4DB0-9A9A-8A81DB7955D5

Q33816822-6AE4C40D-3E4F-4FBF-B908-623F074A1D73

Q33816822-A9ED9F44-0DA3-46DF-8E45-E98215C9D88F

Q33816822-AEEF2823-4F2F-4D8D-A245-29936811BCC6

Q33816822-B1AD7123-475C-4A7C-9FC3-30CD1787C43C

Q33816822-B90F575C-AEAD-4EBF-80D0-D38D7DBA8A4A

Q33816822-D04B8975-825E-4AAD-9D83-F6E86EC84745

P2860

Q33816822-0B0A82CE-B3DE-4A2E-A7D1-F06C91492BE3

Q33816822-13950056-4CF7-4240-9821-CAC9D62B05CC

Q33816822-1A4D776B-C68D-4A49-B3DE-63C6FEBD4607

Q33816822-1B7EE6AD-C0CD-4B70-AEFE-A0852C1D7F07

Q33816822-27F5346A-E664-494F-A86E-99301FADF0DA

Q33816822-3345C0E5-A2AF-4BDF-88F0-2A08841AD99E

Q33816822-39962663-5002-4C5F-B5B0-C62612B75789

Q33816822-3DC232DC-14D1-4B9E-9E34-2735AFC36497

Q33816822-4B76DD65-7551-40F5-9798-A99D264D4376

Q33816822-505CEF29-96E2-4266-9E7A-508640EF7F11

P304

Q33816822-4A9AD2A3-DC35-477B-A21B-4ECD3B725426

P31

Q33816822-095058DD-44C7-4F76-91DB-5E8DF0797E36

P356

Q33816822-E6B13FBD-475B-4019-9420-AFB01AD64F64

P433

Q33816822-197DBE55-3FB8-4525-966A-5C727031CDD9

P478

Q33816822-470B81D2-6D57-40EC-AFA5-A0AAACE89F9B

P50

Q33816822-59A9B96A-C1B5-42C1-B24F-4AB3E3ED49E8

P577

Q33816822-937B8B5D-F25B-446F-BEFE-478B5CD240FE

P5875

Q33816822-0A46878D-75AA-4EEF-9E7A-E3231D21A71D

P698

Q33816822-E2D11B81-025C-4756-970E-67B80DAC6739

P921

Q33816822-265414EB-42A9-47AD-9052-87EB04A15FE4

Q33816822-9DC5C939-3BE7-4858-942D-87BFB4E88712

P932

Q33816822-051D7B22-C766-4B61-85A0-54818C7C519A

P356

AJPHEART.00007.2013

P1433

American Journal of Physiology - Heart and Circulatory Physiology

P1476

Xanthine oxidase inhibition pr ...... ion in cardiac volume overload

@en

P2093

Blake R Zelickson

http://www.w3.org/2001/XMLSchema#string

Danielle M Yancey

http://www.w3.org/2001/XMLSchema#string

James D Gladden

http://www.w3.org/2001/XMLSchema#string

Jason L Guichard

http://www.w3.org/2001/XMLSchema#string

Louis J Dell'Italia

http://www.w3.org/2001/XMLSchema#string

Mayilvahanan Shanmugam

http://www.w3.org/2001/XMLSchema#string

Michelle S Johnson

http://www.w3.org/2001/XMLSchema#string

Mustafa I Ahmed

http://www.w3.org/2001/XMLSchema#string

Scott Ballinger

http://www.w3.org/2001/XMLSchema#string

Victor Darley-Usmar

http://www.w3.org/2001/XMLSchema#string

P2860

Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinol

Ablation of sarcolipin enhances sarcoplasmic reticulum calcium transport and atrial contractility

Cardiac-specific overexpression of sarcolipin inhibits sarco(endo)plasmic reticulum Ca2+ ATPase (SERCA2a) activity and impairs cardiac function in mice

Allopurinol enhances the contractile response to dobutamine and exercise in dogs with pacing-induced heart failure.

Diastolic dysfunction in volume-overload hypertrophy is associated with abnormal shearing of myolaminar sheets.

Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.

Impaired S-nitrosylation of the ryanodine receptor caused by xanthine oxidase activity contributes to calcium leak in heart failure

Cardiac kallikrein-kinin system is upregulated in chronic volume overload and mediates an inflammatory induced collagen loss.

Allopurinol acutely increases adenosine triphospate energy delivery in failing human hearts.

Cardiac-specific overexpression of sarcolipin in phospholamban null mice impairs myocyte function that is restored by phosphorylation

P304

H1440-50

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1152/AJPHEART.00007.2013

http://www.w3.org/2001/XMLSchema#string

P433

10

http://www.w3.org/2001/XMLSchema#string

P478

305

http://www.w3.org/2001/XMLSchema#string

P50

P577

2013-09-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

256470237

http://www.w3.org/2001/XMLSchema#string

P698

24014679

http://www.w3.org/2001/XMLSchema#string

P921

P932

4073978

http://www.w3.org/2001/XMLSchema#string