The antiretroviral activity of APOBEC3 is inhibited by the foamy virus accessory Bet protein. - Test2 - elhamkhani - TriplyDB

The antiretroviral activity of APOBEC3 is inhibited by the foamy virus accessory Bet protein.

The antiretroviral activity of APOBEC3 is inhibited by the foamy virus accessory Bet protein.

http://www.wikidata.org/entity/Q33841186

about

Restriction of foamy viruses by APOBEC cytidine deaminases Adaptive evolution and antiviral activity of the conserved mammalian cytidine deaminase APOBEC3H.APOBEC3A and APOBEC3B are potent inhibitors of LTR-retrotransposon function in human cells Role and mechanism of action of the APOBEC3 family of antiretroviral resistance factors Differential requirement for conserved tryptophans in human immunodeficiency virus type 1 Vif for the selective suppression of APOBEC3G and APOBEC3F.Prototype foamy virus Bet impairs the dimerization and cytosolic solubility of human APOBEC3G How Active Are Porcine Endogenous Retroviruses (PERVs)?Multiple APOBEC3 restriction factors for HIV-1 and one Vif to rule them all Evolution of foamy viruses: the most ancient of all retroviruses The APOBEC3 family of retroelement restriction factors Foamy virus biology and its application for vector development Foamy virus budding and release Foamy Virus Protein-Nucleic Acid Interactions during Particle Morphogenesis APOBEC3 Interference during Replication of Viral Genomes Reversed functional organization of mouse and human APOBEC3 cytidine deaminase domains Wide distribution and ancient evolutionary history of simian foamy viruses in New World primates Tenth International Foamy Virus Conference 2014--achievements and perspectives Non-simian foamy viruses: molecular virology, tropism and prevalence and zoonotic/interspecies transmission Intrinsic antiviral immunity An ancient history of gene duplications, fusions and losses in the evolution of APOBEC3 mutators in mammals APOBEC3G: an intracellular centurion Going wild: lessons from naturally occurring T-lymphotropic lentiviruses FIV cross-species transmission: an evolutionary prospective.The restriction of zoonotic PERV transmission by human APOBEC3G.Genetic editing of HBV DNA by monodomain human APOBEC3 cytidine deaminases and the recombinant nature of APOBEC3G.Are viruses inhibited by APOBEC3 molecules from their host species?Restrictions to cross-species transmission of lentiviral infection gleaned from studies of FIV Leveraging APOBEC3 proteins to alter the HIV mutation rate and combat AIDS.N-Myc interactor inhibits prototype foamy virus by sequestering viral Tas protein in the cytoplasm.Vif of feline immunodeficiency virus from domestic cats protects against APOBEC3 restriction factors from many felids Long-term restriction by APOBEC3F selects human immunodeficiency virus type 1 variants with restored Vif function Evolution of the primate APOBEC3A cytidine deaminase gene and identification of related coding regions.Two genetic determinants acquired late in Mus evolution regulate the inclusion of exon 5, which alters mouse APOBEC3 translation efficiency Feline lentivirus evolution in cross-species infection reveals extensive G-to-A mutation and selection on key residues in the viral polymerase APOBEC4 Enhances the Replication of HIV-1.Vif Proteins from Diverse Human Immunodeficiency Virus/Simian Immunodeficiency Virus Lineages Have Distinct Binding Sites in A3C.The role of BST2/tetherin in feline retrovirus infection Tetherin inhibits prototypic foamy virus release.Severe restriction of xenotropic murine leukemia virus-related virus replication and spread in cultured human peripheral blood mononuclear cells.Feline tetherin efficiently restricts release of feline immunodeficiency virus but not spreading of infection.

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P2860

The antiretroviral activity of APOBEC3 is inhibited by the foamy virus accessory Bet protein.

http://www.wikidata.org/entity/Q33841186

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2005 nî lūn-bûn

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2005 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2005年の論文

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P2093

Ali Saib

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Carsten Münk

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Egbert Flory

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Fabian Romen

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Heide Muckenfuss

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Klaus Cichutek

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Marion Battenberg

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Martin Löchelt

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Patrizia Bastone

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P2860

The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA

Human APOBEC3F is another host factor that blocks human immunodeficiency virus type 1 replication

A second human antiretroviral factor, APOBEC3F, is suppressed by the HIV-1 and HIV-2 Vif proteins

A single amino acid substitution in human APOBEC3G antiretroviral enzyme confers resistance to HIV-1 virion infectivity factor-induced depletion

Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcripts

DNA deamination mediates innate immunity to retroviral infection

Species-specific exclusion of APOBEC3G from HIV-1 virions by Vif

HIV-1 Vif blocks the antiviral activity of APOBEC3G by impairing both its translation and intracellular stability

Induction of APOBEC3G ubiquitination and degradation by an HIV-1 Vif-Cul5-SCF complex

APOBEC3F properties and hypermutation preferences indicate activity against HIV-1 in vivo

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7982-7987

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22

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15911774

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