Ghrelin O-acyltransferase (GOAT) is essential for growth hormone-mediated survival of calorie-restricted mice. - Test2 - elhamkhani - TriplyDB

Ghrelin O-acyltransferase (GOAT) is essential for growth hormone-mediated survival of calorie-restricted mice.

Ghrelin O-acyltransferase (GOAT) is essential for growth hormone-mediated survival of calorie-restricted mice.

http://www.wikidata.org/entity/Q33841793

about

Ghrelin secretion stimulated by {beta}1-adrenergic receptors in cultured ghrelinoma cells and in fasted mice A healthy gastrointestinal microbiome is dependent on dietary diversity Obesity Impairs the Action of the Neuroendocrine Ghrelin System Ghrelin's second life: from appetite stimulator to glucose regulator Anorexia nervosa and bone metabolism Ghrelin O Acyl Transferase (GOAT) as a Novel Metabolic Regulatory Enzyme Ghrelin.Metabolic Benefit of Chronic Caloric Restriction and Activation of Hypothalamic AGRP/NPY Neurons in Male Mice Is Independent of Ghrelin.Fatty acylation of proteins: The long and the short of it.The role of ghrelin in GH secretion and GH disorders.Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.The ghrelin axis in disease: potential therapeutic indications.Ghrelin and cachexia: will treatment with GHSR-1a agonists make a difference for patients suffering from chronic wasting syndromes?The GOAT-ghrelin system is not essential for hypoglycemia prevention during prolonged calorie restriction.Ghrelin and growth hormone: story in reverse.An age-dependent interaction with leptin unmasks ghrelin's bone-protective effects Therapeutic Potential of Targeting the Ghrelin Pathway.Ghrelin: much more than a hunger hormone.Essential Role of Growth Hormone and IGF-1 in Therapeutic Effect of Ghrelin in the Course of Acetic Acid-Induced Colitis.A small molecule antagonist of ghrelin O-acyltransferase (GOAT).Divergent neuronal circuitries underlying acute orexigenic effects of peripheral or central ghrelin: critical role of brain accessibility.Long-term physiological alterations and recovery in a mouse model of separation associated with time-restricted feeding: a tool to study anorexia nervosa related consequences Apo-Ghrelin Receptor (apo-GHSR1a) Regulates Dopamine Signaling in the Brain.Neuroanatomical characterization of a growth hormone secretagogue receptor-green fluorescent protein reporter mouse.Fasting vs dietary restriction in cellular protection and cancer treatment: from model organisms to patients.Anorexia nervosa: a unified neurological perspective Scientific side trips: six excursions from the beaten path.Altered ghrelin secretion in mice in response to diet-induced obesity and Roux-en-Y gastric bypass.Fibroblast growth factor 21 is not required for the reductions in circulating insulin-like growth factor-1 or global cell proliferation rates in response to moderate calorie restriction in adult mice Assessing interactions between Ghsr and Mc3r reveals a role for AgRP in the expression of food anticipatory activity in male mice.Prolonged restraint stress increases IL-6, reduces IL-10, and causes persistent depressive-like behavior that is reversed by recombinant IL-10.Ablation of ghrelin O-acyltransferase does not improve glucose intolerance or body adiposity in mice on a leptin-deficient ob/ob background.Ghrelin and its potential in the treatment of eating/wasting disorders and cachexia.Glucose and weight control in mice with a designed ghrelin O-acyltransferase inhibitor.Expression of Serum Retinol Binding Protein and Transthyretin within Mouse Gastric Ghrelin Cells.Reduced autophagy in livers of fasted, fat-depleted, ghrelin-deficient mice: reversal by growth hormone.Effect of oral glucose administration on rebound growth hormone release in normal and obese women: the role of adiposity, insulin sensitivity and ghrelin.Ghrelin directly stimulates glucagon secretion from pancreatic alpha-cells.Biology's response to dieting: the impetus for weight regain.Stress-related alterations of acyl and desacyl ghrelin circulating levels: mechanisms and functional implications.

P2860

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P2860

Ghrelin O-acyltransferase (GOAT) is essential for growth hormone-mediated survival of calorie-restricted mice.

http://www.wikidata.org/entity/Q33841793

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

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Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

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type

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Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

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Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

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prefLabel

Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

@ast

Ghrelin O-acyltransferase (GOA ...... al of calorie-restricted mice.

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PNAS.1002271107

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Ghrelin O-acyltransferase (GOA ...... val of calorie-restricted mice

@en

P2093

David M Valenzuela

http://www.w3.org/2001/XMLSchema#string

George D Yancopoulos

http://www.w3.org/2001/XMLSchema#string

Guosheng Liang

http://www.w3.org/2001/XMLSchema#string

Mark W Sleeman

http://www.w3.org/2001/XMLSchema#string

Michael S Brown

http://www.w3.org/2001/XMLSchema#string

Robert Lin Li

http://www.w3.org/2001/XMLSchema#string

Tong-Jin Zhao

http://www.w3.org/2001/XMLSchema#string

Xuefen Xie

http://www.w3.org/2001/XMLSchema#string

P2860

Ghrelin is a growth-hormone-releasing acylated peptide from stomach

Ghrelin octanoylation mediated by an orphan lipid transferase

Mice lacking ghrelin receptors resist the development of diet-induced obesity

Identification of the acyltransferase that octanoylates ghrelin, an appetite-stimulating peptide hormone

Deletion of ghrelin impairs neither growth nor appetite

Decreased lipid synthesis in livers of mice with disrupted Site-1 protease gene

Genetic deletion of ghrelin does not decrease food intake but influences metabolic fuel preference

Novel ghrelin assays provide evidence for independent regulation of ghrelin acylation and secretion in healthy young men.

A preprandial rise in plasma ghrelin levels suggests a role in meal initiation in humans.

Ghrelin: structure and function.

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scholarly article

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10.1073/PNAS.1002271107

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16

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107

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Joseph L. Goldstein

Andrew J Murphy

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2010-03-15T00:00:00Z

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41969121

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20231469

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2867684

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