Role of neutrophils in induction of acute inflammation in T-cell-mediated immune dermatosis, psoriasis: a neutrophil-associated inflammation-boosting loop.
about
Inhibition of immune-complex mediated dermal inflammation in rats following either oral or topical administration of a small molecule C5a receptor antagonistCellular dissection of psoriasis for transcriptome analyses and the post-GWAS eraAssociation between Psoriasis and Cardiovascular Risk Factors in Korean Patients.Modulation of epidermal transcription circuits in psoriasis: new links between inflammation and hyperproliferation.Topical application of delphinidin reduces psoriasiform lesions in the flaky skin mouse model by inducing epidermal differentiation and inhibiting inflammationPathophysiology of generalized pustular psoriasis.Calcitriol inhibits keratinocyte proliferation by upregulating leukocyte elastase inhibitor (serpin B1).Topical alpha-selective p38 MAP kinase inhibition reduces acute skin inflammation in guinea pigSuprabasal overexpression of the hsRPB7 gene in psoriatic epidermis as identified by a reverse transcriptase-polymerase chain reaction differential display model comparing psoriasis plaque tissue with peritonsillar mucosaAcute generalized exanthematous pustulosis: role of cytotoxic T cells in pustule formation.Severity of Psoriasis Associates With Aortic Vascular Inflammation Detected by FDG PET/CT and Neutrophil Activation in a Prospective Observational StudyUpdate on psoriasis immunopathogenesis and targeted immunotherapy.Genome-wide analysis of the genetic regulation of gene expression in human neutrophils.Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways.The IL-23/IL-17 axis in psoriatic arthritis.The roles of cells and cytokines in the pathogenesis of psoriasis.The Inflammatory Response in Psoriasis: a Comprehensive Review.The chemokine receptors CXCR1 and CXCR2 regulate oral cancer cell behaviour.Comparison of stress-induced PRINS gene expression in normal human keratinocytes and HaCaT cells.Hydrogen sulfide inhibits IL-8 expression in human keratinocytes via MAP kinase signaling.Interleukin-18 induces acute biphasic reduction in the levels of circulating leukocytes in mice.The Increased Expression of Matrix Metalloproteinase-9 Messenger RNA in the Non-lesional Skin of Patients with Large Plaque Psoriasis Vulgaris.Tissue and Serum Inflammatory Cytokine Levels in Korean Psoriasis Patients: A Comparison between Plaque and Guttate PsoriasisThe host defense peptide LL-37 selectively permeabilizes apoptotic leukocytes.Granulocyte and Monocyte Adsorption Apheresis for Generalized Pustular Psoriasis: Therapeutic Outcomes in Three Refractory Patients.Metalloelastase (MMP-12) and 92-kDa gelatinase (MMP-9) as well as their inhibitors, TIMP-1 and -3, are expressed in psoriatic lesions.High frequency of cholestasis in generalized pustular psoriasis: Evidence for neutrophilic involvement of the biliary tract.Establishing the dynamics of neutrophil accumulation in vivo by reflectance confocal microscopy.Functional MASP2 single nucleotide polymorphism plays no role in psoriasis.Effect of Efalizumab on neutrophil and monocyte functions in patients with psoriasis.Microbicidal protein psoriasin is a multifunctional modulator of neutrophil activation.Targeting IL-17 in psoriatic arthritis.Treatment of recalcitrant pustular psoriasis with infliximab: effective reduction of chemokine expression.Psoriasis genomics: analysis of proinflammatory (type 1) gene expression in large plaque (Western) and small plaque (Asian) psoriasis vulgaris.Effect of tofacitinib, a Janus kinase inhibitor, on haematological parameters during 12 weeks of psoriasis treatment.Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines.Suppressing IL-36-driven inflammation using peptide pseudosubstrates for neutrophil proteases.Sexy again: the renaissance of neutrophils in psoriasis.Phase III clinical study of maxacalcitol ointment in patients with palmoplantar pustulosis: A randomized, double-blind, placebo-controlled trial.Identification of small-molecule elastase inhibitors as antagonists of IL-36 cytokine activation.
P2860
Q28366075-FCF5F3E7-DF47-42EE-A79E-C851D08FD32FQ33768338-7577C3B4-4D97-4B2A-87DC-F5FD5AB10327Q34050825-CB91F445-FE60-48C9-A377-6A2F2A5AE70DQ35048276-C7B197E1-7708-4E81-A58A-416517D1D85CQ35071781-A3359487-962E-4761-8E9C-ED6B8341DD85Q35099781-5C720ABE-A8C1-4A47-94E4-860885346080Q35153480-A1AB723B-88AC-4A5C-A189-2600C2E7C50CQ35557393-94E3E8B6-0334-46F7-8DB0-4074837C8BCDQ35746233-3F4199CF-9DBB-4A27-B306-BC731AB8E59DQ35748154-04B3244D-CD77-4B27-8FB6-D2896A5CE25DQ36326833-C0C93CAB-2BB7-41BB-B416-FBF491C1FB4BQ36445919-A936912A-409E-4458-A6B4-2BE0ACAD7311Q37031874-33E5F481-F493-4065-A741-50749226CD30Q37298535-C719A5A4-CAAC-45F7-8519-CE3570A32C97Q37717133-CADEC26C-6F23-4EF1-B7CD-3C9A035102BFQ37995658-831DB789-F107-471C-B866-8D3737F7C03CQ38792930-5B2BFC0A-6765-47AE-B431-31A4BC4ED35EQ38981288-5CCA25E5-D1AB-4AD4-945C-01E4E04C8457Q39507704-392DF5F8-B164-4C02-91EF-3619C82A28FFQ39545584-C807F2B6-FE27-4308-9F46-F817BBA91E2DQ39625918-FBD7BBAB-E38F-4B7E-9ABB-8C7679B234EBQ40970646-CF18B2E3-EE0C-4D85-9E1C-12274754A157Q41519398-5552613D-3AEF-49C2-94AF-323A5C2D01E8Q41866861-54AE30C4-4D0B-4211-B3CE-6DB42C45314EQ41952126-6644BCF9-2398-40A8-B7CD-DCF038A9B1A3Q42505053-3A0A83CD-0618-46C8-A64D-D896F7B1C33BQ45059663-0E0A34AC-C610-4130-ABB1-8D2C34B98F36Q45465751-6FABA856-896E-41C9-AAC9-B715A173827FQ46539277-7078CD38-EF02-4D65-AF2D-BEBE2F005346Q46545360-61695B61-44E5-46F2-AE73-80654DD4F5FBQ46808883-0CE41D1B-1085-437C-B88A-D81FE39C7850Q47140713-BF12BBFF-E35B-460F-93D4-8307929D446DQ47588113-EF4879A7-D2B3-4960-9A3F-68A5CF04495DQ47730035-FBEFA2E6-092B-4BEF-9F99-CD4B896A612AQ48005178-A65E5053-1A57-4AD3-95A2-76DD05A22F14Q51072843-6690BB47-1AD9-4EDD-9756-7809FC5D4EEFQ52728096-9992D3AA-13FD-4BA3-A92B-B92A88801430Q53096910-4B111A7E-5F57-4E77-8690-B9C39DA2B8B8Q53827140-BABDEB14-04A8-4992-8D4E-EC35883B9959Q55111930-E6369860-FC94-45BE-BBDB-5D23AD061BE1
P2860
Role of neutrophils in induction of acute inflammation in T-cell-mediated immune dermatosis, psoriasis: a neutrophil-associated inflammation-boosting loop.
description
2000 nî lūn-bûn
@nan
2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@ast
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@en
type
label
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@ast
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@en
prefLabel
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@ast
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@en
P2093
P1476
Role of neutrophils in inducti ...... ed inflammation-boosting loop.
@en
P2093
P356
10.1034/J.1600-0625.2000.009001001.X
P577
2000-02-01T00:00:00Z