Cannabinoid-2 receptor limits inflammation, oxidative/nitrosative stress, and cell death in nephropathy.
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Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injuryIs lipid signaling through cannabinoid 2 receptors part of a protective system?Modulation of cellular redox homeostasis by the endocannabinoid systemPlant derived substances with anti-cancer activity: from folklore to practiceSpontaneous Cannabinoid Receptor 2 (CB2) Expression in the Cochlea of Adult Albino Rat and Its Up-Regulation after Cisplatin TreatmentPlant-Derived Agents for Counteracting Cisplatin-Induced NephrotoxicityAn integrated view of cisplatin-induced nephrotoxicity and ototoxicityCitral alleviates an accelerated and severe lupus nephritis model by inhibiting the activation signal of NLRP3 inflammasome and enhancing Nrf2 activationRiboflavin ameliorates cisplatin induced toxicities under photoilluminationTargeting cannabinoid signaling for peritoneal dialysis-induced oxidative stress and fibrosis.The endocannabinoid system: 'NO' longer anonymous in the control of nitrergic signalling?Blocking TRAIL-DR5 signaling with soluble DR5 alleviates acute kidney injury in a severely burned mouse model.CB1 cannabinoid receptors promote oxidative/nitrosative stress, inflammation and cell death in a murine nephropathy model.Cannabinoid-1 receptor activation induces reactive oxygen species-dependent and -independent mitogen-activated protein kinase activation and cell death in human coronary artery endothelial cells.Tempol, a superoxide dismutase mimetic agent, ameliorates cisplatin-induced nephrotoxicity through alleviation of mitochondrial dysfunction in miceActivation of a nuclear factor of activated T-lymphocyte-3 (NFAT3) by oxidative stress in carboplatin-mediated renal apoptosis.Sustained oxidative stress causes late acute renal failure via duplex regulation on p38 MAPK and Akt phosphorylation in severely burned rats.Cannabidiol protects against hepatic ischemia/reperfusion injury by attenuating inflammatory signaling and response, oxidative/nitrative stress, and cell death.Cannabidiol as an emergent therapeutic strategy for lessening the impact of inflammation on oxidative stress.Chronic administration of Δ9-tetrahydrocannabinol induces intestinal anti-inflammatory microRNA expression during acute simian immunodeficiency virus infection of rhesus macaquesClinical significance of serum expression of GROβ in esophageal squamous cell carcinoma.Protective role of cannabinoid receptor type 2 in a mouse model of diabetic nephropathy.Sulforaphane, a natural constituent of broccoli, prevents cell death and inflammation in nephropathyPoly(ADP-ribose) polymerase-1 is a key mediator of cisplatin-induced kidney inflammation and injury.Endocannabinoid signaling at the periphery: 50 years after THC.Endometriosis gene expression heterogeneity and biosignature: a phylogenetic analysis.Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathyCB2R orchestrates fibrogenesis through regulation of inflammatory response during the repair of skeletal muscle contusion.Summary of the 2014 Alcohol and Immunology Research Interest Group (AIRIG) meetingβ-Caryophyllene ameliorates cisplatin-induced nephrotoxicity in a cannabinoid 2 receptor-dependent manner.The endocannabinoid system and plant-derived cannabinoids in diabetes and diabetic complicationsMitochondrial reactive oxygen species generation triggers inflammatory response and tissue injury associated with hepatic ischemia-reperfusion: therapeutic potential of mitochondrially targeted antioxidants.Cisplatin nephrotoxicity involves mitochondrial injury with impaired tubular mitochondrial enzyme activity.Targeting cannabinoid receptor CB(2) in cardiovascular disorders: promises and controversies.The novel, orally available and peripherally restricted selective cannabinoid CB2 receptor agonist LEI-101 prevents cisplatin-induced nephrotoxicity.Prolonged activation of human islet cannabinoid receptors in vitro induces adaptation but not dysfunction.Cannabinoid receptor 2 suppresses leukocyte inflammatory migration by modulating the JNK/c-Jun/Alox5 pathwayModulating the endocannabinoid system in human health and disease--successes and failuresProtective Action of Anandamide and Its COX-2 Metabolite against l-Homocysteine-Induced NLRP3 Inflammasome Activation and Injury in Podocytes.Role of the endocannabinoid system in diabetes and diabetic complications.
P2860
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P2860
Cannabinoid-2 receptor limits inflammation, oxidative/nitrosative stress, and cell death in nephropathy.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@ast
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@en
type
label
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@ast
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@en
prefLabel
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@ast
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@en
P2093
P2860
P50
P921
P1476
Cannabinoid-2 receptor limits ...... and cell death in nephropathy.
@en
P2093
Bani Mukhopadhyay
György Haskó
Mohanraj Rajesh
Sándor Bátkai
Vivek Patel
P2860
P304
P356
10.1016/J.FREERADBIOMED.2009.11.022
P577
2009-12-04T00:00:00Z