Pressure is proinflammatory in lung venular capillaries.
about
Protein-losing enteropathy in patients with Fontan circulation: is it triggered by infection?Preoperative prediction of pediatric patients with effusions and edema following cardiopulmonary bypass surgery by serological and routine laboratory data.Pediatric Acute Respiratory Distress Syndrome: Fluid Management in the PICUStudies on the cell biology of interendothelial cell gapsHypoxic pulmonary vasoconstriction requires connexin 40-mediated endothelial signal conduction.Stabilized imaging of immune surveillance in the mouse lung.Characterization of pressure-induced calcium response in neuronal cell lines.Resolution of pulmonary edema. Thirty years of progress.Mechanisms regulating endothelial cell barrier function.Actin cytoskeleton regulates stretch-activated Ca2+ influx in human pulmonary microvascular endothelial cells.Plasma angiopoietin-2 in clinical acute lung injury: prognostic and pathogenetic significance.Update on pulmonary edema: the role and regulation of endothelial barrier function.Cytokines and perinatal brain damage.Integrated control of lung fluid balance.Pathophysiology of pulmonary hypertension in acute lung injury.Hypoxia, leukocytes, and the pulmonary circulation.Platelets induce endothelial tissue factor expression in a mouse model of acid-induced lung injuryAirway strain during mechanical ventilation in an intact animal modelITF1697, a stable Lys-Pro-containing peptide, inhibits weibel-palade body exocytosis induced by ischemia/reperfusion and pressure elevation.Acute lung injury and the acute respiratory distress syndrome: four decades of inquiry into pathogenesis and rational management.Real-time lung microscopy.Acute lung injury/acute respiratory distress syndrome (ALI/ARDS): the mechanism, present strategies and future perspectives of therapies.Physiological determinants of the pulmonary filtration coefficientA novel signaling mechanism between gas and blood compartments of the lungParacrine purinergic signaling determines lung endothelial nitric oxide production.High-altitude pulmonary edema.Ventilation-induced lung injury.TRPV4: physiological role and therapeutic potential in respiratory diseases.Mechanisms and mediators of lung injury after acute kidney injury.The pathophysiology of pulmonary hypertension in left heart disease.Fluid-induced lung injury-role of TRPV4 channels.Vascular regulation of type II cell exocytosis.Stretch activates nitric oxide production in pulmonary vascular endothelial cells in situ.Mechano-oxidative coupling by mitochondria induces proinflammatory responses in lung venular capillaries.Hyperosmolarity enhances the lung capillary barrier.Obstructive Sleep Apnea and Subclinical Interstitial Lung Disease in MESA.IDH3 mediates apoptosis of alveolar epithelial cells type 2 due to mitochondrial Ca2+ uptake during hypocapnia.Diabetes worsens pulmonary diffusion in heart failure, and insulin counteracts this effect.Pressure-induced endothelial Ca(2+) oscillations in lung capillaries.High tidal volume ventilation induces proinflammatory signaling in rat lung endothelium.
P2860
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P2860
Pressure is proinflammatory in lung venular capillaries.
description
1999 nî lūn-bûn
@nan
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Pressure is proinflammatory in lung venular capillaries.
@ast
Pressure is proinflammatory in lung venular capillaries.
@en
type
label
Pressure is proinflammatory in lung venular capillaries.
@ast
Pressure is proinflammatory in lung venular capillaries.
@en
prefLabel
Pressure is proinflammatory in lung venular capillaries.
@ast
Pressure is proinflammatory in lung venular capillaries.
@en
P2093
P2860
P356
P1476
Pressure is proinflammatory in lung venular capillaries.
@en
P2093
A C Issekutz
J Bhattacharya
W M Kuebler
P2860
P304
P356
10.1172/JCI6872
P407
P577
1999-08-01T00:00:00Z